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    DIAGNOSIS: Deficient Fluid Volume Related To Anemia As Evidenced by Decreased Hemoglobin and Hematocrit

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    Brandt Cajocon
    The document describes an acute left intertrochanteric femur fracture in an elderly patient. Key details include: - The fracture was caused by a fall and results in severe hip pain. - Risk factors include old age, history of osteoarthritis and stroke. - Left untreated, the fracture could lead to hematoma, ischemia, necrosis and complications from impaired blood supply. - Treatment involves administering pain medications like tramadol and ketorolac, and monitoring for signs of peripheral neurovascular dysfunction. The goal is healing and functional restoration of the hip through callus formation.

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    DIAGNOSIS: Deficient Fluid Volume Related To Anemia As Evidenced by Decreased Hemoglobin and Hematocrit

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    Brandt Cajocon
    6 views3 pages
    The document describes an acute left intertrochanteric femur fracture in an elderly patient. Key details include: - The fracture was caused by a fall and results in severe hip pain. - Risk factors include old age, history of osteoarthritis and stroke. - Left untreated, the fracture could lead to hematoma, ischemia, necrosis and complications from impaired blood supply. - Treatment involves administering pain medications like tramadol and ketorolac, and monitoring for signs of peripheral neurovascular dysfunction. The goal is healing and functional restoration of the hip through callus formation.

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    The document describes an acute left intertrochanteric femur fracture in an elderly patient. Key details include: - The fracture was caused by a fall and results in severe hip pain. - Risk factors include old age, history of osteoarthritis and stroke. - Left untreated, the fracture could lead to hematoma, ischemia, necrosis and complications from impaired blood supply. - Treatment involves administering pain medications like tramadol and ketorolac, and monitoring for signs of peripheral neurovascular dysfunction. The goal is healing and functional restoration of the hip through callus formation.

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    6 views3 pages

    DIAGNOSIS: Deficient Fluid Volume Related To Anemia As Evidenced by Decreased Hemoglobin and Hematocrit

    Uploaded by

    Brandt Cajocon
    The document describes an acute left intertrochanteric femur fracture in an elderly patient. Key details include: - The fracture was caused by a fall and results in severe hip pain. - Risk factors include old age, history of osteoarthritis and stroke. - Left untreated, the fracture could lead to hematoma, ischemia, necrosis and complications from impaired blood supply. - Treatment involves administering pain medications like tramadol and ketorolac, and monitoring for signs of peripheral neurovascular dysfunction. The goal is healing and functional restoration of the hip through callus formation.

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    of 3

    REDUCED MUSCLE

    REDUCED ROM OSTEOARTHRITIS


    STRENGHT

    BREAKDOWN IN THE COMPLEX JOINT INSTABILITY


    IMPAIRED BALANCED AND
    SYSTEMS THAT MAINTAIN
    COORDINATION
    POSTURAL CONTROL
    DIAGNOSIS:Acute pain related to left
    intertrochanteric femur fracture as evidenced
    by facial grimace.
    FALL
    INDEPENDENT:
     Monitor vital signs
     Instruct patient to do deep breathing
    techniques
    BONES BREAK DUE  Assist patient to a comfortable position
    TO EXCESSIVE FORCE  Promote periods of rest for the patient.
    DIAGNOSIS: Deficient Fluid
    Volume related to Anemia as DEPENDENT:
    evidenced by decreased DISRUPTION OF BONE BREAKAGE OF THE FEMUR  Administer prescribed Tramadol 50mg
    BONE BETWEEN ITS GREATER IVTT q8H
    Hemoglobin and Hematocrit. INTEGRITY  Administer prescribed Keterolac 30mg
    AND LESSER TROCHANTERS
    IVTT q8H
    Independent:
     Assess skin turgor and
    moisture of mucous Hgb 11.5 (low)
    INTETROCHANTERIC
    membrane. Hct 35.4 (low) PAIN IN THE HIPS
    FEMUR FRACTURE
     Monitor vital signs
    (especially BP and HR). KETOROLAC 30mg
    PNSS 1lL @20
     Evaluate peripheral pulses BLEEDING DAMAGED BLOOD VESSELS Q8H IVTT
    gtts/min
    and capillary refill.
     Monitor intake and output. TRAMADOL 50MG
    Assess color and amount Q8H IVTT
    VASOCONSTRICTION
    of urine. Report urine HEMATOMA FORM AS
    BLOODS POOL AROUND DISRUPTING IN BLOOD
    output less than 30 ml/hr
    THE FRACTURE SITE SUPPLY
    for two (2) consecutive
    hours. PLATELET RESPONSE
     Review laboratory Results
    IF TREATED: ISCHEMIA
    Dependent:
     Administer prescribed REDUCED PLATELET FORMATION OF A
    PNSS 1lL @20 gtts/min PRODUCTION PLATELET PLUG
    NECROSIS
    COAGULATION INITIATING AN
    IF NOT TREATED: THROMBOCYTOPENIA INFLAMMATION
    CASCADE INFLAMMATORY
    RESPONSE

    FIBRIN KETOROLAC
    FORMATION 30mg Q8H
    MIGRATE TO THE
    IVTT
    SITE TO CLEAR
    DEBRIS, TRAMADOL
    CLOT BACTERIA AND 50MG Q8H
    RETRACTION DAMAGED CELLS IVTT
    AND REPAIR

    RELEASE
    FIBRINOLYSIS CYTOKINES AND
    GROWTH FACTORS

    STIMULATE CELL
    SOFT CALLUS PROLIFERATION
    Good Prognosis FORMATION AND MIGRATION

    EXCESS BONE BONE


    MATERIAL IS REMODELING
    RESORBED TISSUE ATTRACTS
    REGENERATION FIBROBLASTS
    Bad Prognosis HARD CALLUS
    FORMATION
    BONE IS
    RESHAPED TO MALUNION DIAGNOSIS: Risk for Peripheral Neurovascular Dysfunction as
    ITS ORIGINAL NONUNION evidenced by swelling of left thigh
    FORM UNION
    ndependent:
     Monitor vital signs.
    FUNCTIONAL  Perform neurovascular assessments.
    RESTORATION
     Assess the strength to perform ROM to all joints.

     Maintain elevation of the injured area unless


    contraindicated.
    Dependent

     Administer prescribed Tramadol 50mg IVTT q8H


     Administer prescribed Keterolac 30mg IVTT q8H
    RISK FACTORS MECHANISM COMPLICATIONS S/S DX MGT.
    MEDS LABS PREDISPOSING FACTORS
    PRECIPITATING FACTORS
     OLD AGE (77 yrs.old)
     ENVIRONMENTAL HAZARDS  HISTORY OF
     TRAUMA OSTHEOARTHOSIS
     HISTORY OF STROKE

    MECHANICAL STRESS

    DEVELOPS SURFACE
    ARTICULAR CARTILAGE
    FIBRILLATION,
    DEGRADATION
    IRREGULARITY AND
    FOCAL EROSION

    EXTENDS DOWN TO
    COLLAGEN MATRIX IMPROPERLY
    THE BONE AND
    DAMAGED MINERALIZED
    CONTINUALLY EXPAND
    COLLAGEN

    CHONDROCYTE
    CHONDROCYTES TO FORM CLUSTERS
    UNDERGO SUBCHONDRAL
    PROLIFERATE
    APOPTOSIS BONE
    THICKENING
    CARTILAGE OUT PHENOTYPIC CHANGE
    GROWTHS

    BONE CYSTS
    OSSIFY AND
    FORM
    OSTEOPHYTES
    SYNOVIAL
    INFLAMMATION AND
    HYPERTROPHY

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