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Renal System PDF
Renal System PDF
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①
Acute nephritis/ Nephritic syndrome ✓
③ Anasarca④ D
⑨✓Nephritic syndrome ① ✓
u
Nephrotic syndrome
iepweotie orange proteinuria
D ✓
✓
①
Lab
diagnosis of Acute
glomerulonephritis
✓
Managementof Nephrotic syndrome
.
É eatmeat of BTI
/
Acute
pyelonephritis ②
☒chronic Renal fatinne
✓
Proteinuria ②A
Hematuria ② it
☒ edema ② VD
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?⃝
Acute
-
GDomerutonephritisfntepwu.tt Syndromes - -
-
@
-
•
①
/ ÷nI÷m÷÷nµ/
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Haematweia
Definition-_→
④ ⑦ Nephritic syndrome
.
Oliguria
⑦ Etiology
312
Cchngh)
-
fluid retention
G
,
÷
Proteinuria ]
(17-1)
Pathogenesis Das
-
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a-EE.int
capillaries
t
swollen , infiltrated by
endothelial
vhf
and
proliferation of
cell
me¥d ¥
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Tub# flat-Faced
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a
belomeuulau capsular epithelium
Progress -
→ obstruct
ProI-E-e-ere-scent-sf.se
Degenareatiothnofees .
⑦ clinical
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Management
-
/→
→ signs
Cough -7313-3193
Symptoms
.
Antistreptolysino
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Investigations Esri
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✓
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form hump
in sub endothelial
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when
layer)
deposited
complication -
N④ Non cardiogenic
Acute renal
faliwee
④ Hypertensive encephalopathy
.
,*
Pulinonany oedema
④Ui
secondary infection ;
⑨ bronchitis ,
✓
peritonitis .
✓④ Oedemagioltisa
④ Differential
D-iagnosis-oon-ngioneuroticoedema.aateaaaipy.am
• Embolicnephritis from
Subacute bacterial
endocarditis .
and
⑧ Course
Prognosis -
319 (cnngn)
informant nephritis →
② Acute Post -
Streptococcal
315-316 faugh) -
③
Haematwua -
② Proteinuria -
②
-
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ive -1>-0.581244
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- - -
- -
=_
I
=_= ECOII
.-
③ Oede_ma→ =
✓
F- =
%
°
= ✓ ✓
=
Trimethoprimesufhamethonoitole
-
21 days
For
-
12h
( 6018 omg
) every
-
n '
I ✓
I -
-
Uday .
ofloseacin
300mg / 12 -21dg
-
-
-
-
=
✓
E-
=-
-
=
-
"
mid pioonolact
+ Metolazoe
⑦
Nephrotic syndrome -
③
-
* ChronicRenalFaliwie_
s-stagesof-e.ie#
of nephron
Pathogenesis progressivet
loss > 90m ' / min
-
OEFR
.
→
normal / nigh
→ GFR ) 60 89mi / mid
hypertrophy ( wFR=
-
→ mild CKD
compensatory CKD (crFR=
45 -
59hr11min
I → 3. A -
lnqorderate
( be FR=
30-44 /
ml min)
creatinine @ ) though it is
313
serum
-
nephron
loss due to hyper filtration 15-29 ml / )
min
→ Severe GKD ( oeFR=
I → End
Stage Renal 215m11 ) mi
I
End Renal Disease
Stage
1
Glomerular
sclerosis .
Fibrosis
hyperfiltration
,
.
t reduce
ACE inhibitor, ARBS
reno
protective
Investigations
-
Normoehromic anemia [ erythropoietin d)
Blood Test -
Normocytic
Hb TLC , DLC
,
"
-0 K+ C) ca ①
' '
Biochemistry -
Bicarbonate ④
P
(d) ,
→ Lead to
urine
gpoeravity 10W
-
hyaline ,
broad cell casts in CRF
Ecce → 211A
✗
Ray →
cardiomegaly , pleural effusion
.
size reduced
Us be -5
Kidney .
↳ FR → Low
accumulation of fluid in intenser space
Anasarca -7 generalized
.
ummm
body produced by enp-E-in-mstit.at
palpable swelling on
the
-
9-hud#e → Edema
Edeena-mass-ielgener-Eanasauca.ir
farinose malignancy river
✓
- burns
Fatima
-
Etiology-
-
heart
problem
in
+ venous Trauma ,
renal Fatima =
obstruction
-
lymphatics
- .
✓Elevated
capillary hydraulic pressure
eantfaeinne kidney Pregnant
Pathogenesis g.
,
]
permtÉ
-
trauma ,
ascites)
✓ capillary malignant
syndrome )
,
✓
Hypo albumin
@ mia
(Nephrotic
disease Malnutrition
gg.in#ng,prwdtiSr-highor1-owB.P
.
,
liver
sign.Éns
r-di-pii-tup-ezfi.gr .
heart rate
fast
.
slow or
÷÷÷==
-
and kidneys
.
liver
'
me hike
in limb
← immobility
impÉ
on
of face ,
Diagnosis
-
"
blood Test
-
,
-
-
CT scan
- Chest cavity .
- Eco cardiogram
test
-
allergy
Treatment
-
condition
- Treat
underlying
- Diuretics ( Furosemide 40mg )
salt intake
-
Reduce
reduce swelling .
← Massage
✓ keep moving to prevent
spiroidaeetone
- .
.de +
→ Furoseem_ sometime]
Liver cirrhosis combination
f- [ Trippe
hypokalemia /+ 1Metolazone→ ulcer
.
Treat the
.
g.FI#iuIry-svenousu9cer
→
:
Proteinuria
-