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CHAPTER III Postpartum Complications
CHAPTER III Postpartum Complications
CHAPTER III Postpartum Complications
This chapter focuses on the discussion of nursing assessment and management of postpartal
complications.
Duration: 6 hours
Key Terms:
• Postpartum
• Hemorrhage
• Infection
• Thrombophlebitis
• Psychiatric disorder
➢ refers to excessive blood loss during or after the third stage of labor.
➢ Leading cause of maternal mortality.
Causes: 4 T’s
1. Tone – refers to the failure of the uterine muscles to contract.
2. Tissue – Presence of retained placental tissues prevents full uterine contractions.
3. Trauma – caused by lacerations and episiotomy, hematoma, CS, uterine rupture and
uterine inversion
4. Thrombosis – clot formation and fibrin deposition on the placental site stop the oozing
of blood from the blood vessels of the uterus. Disorders of the coagulation system and
platelets, whether persistent or acquired can result in bleeding.
Causes:
Uterine atony
Lacerations
Disseminated intravascular coagulation
Hematoma
Uterine Atony
➢ Refers to the failure of uterus contract continuously after delivery.
➢ Characterized by boggy or relaxed uterus & profuse bleeding.
➢ Most common cause of PPH
➢ Incidence: 1 for every 20 deliveries
Causes
1. Overstretched uterus (multiple gestation, macrosomia, hydramnios)
2. Complication of labor: precipitous labor, prolonged labor,
3. Uterine relaxing agents: anesthesia, analgesia, terbutaline magnesium sulfate,
nitroglycerine
4. High parity and advanced maternal age
5. Retained placental fragments
6. Infection: amnionitis, chorioamnionitis
7. Oxytocin given during labor
Nursing Management:
a. Massage the uterus
b. Keep the bladder empty
c. Modified trendelenburg
d. IV fast drip/ oxytocin IV drip
e. Breastfeeding – posterior pituitary gland will release oxytocin so uterus will contract.
Lacerations
➢ More common after operative obstetrics, a firm uterus with bright red blood or a steady
stream of unclotted blood
➢ Predisposing factors: Primipara, precipitous labor, macrosomia, forceps or vacuum-
assisted birth, mediolateral episiotomy
➢ Degree of lacerations
1. Cervical
2. Perineal
Classification of Perineal Laceration
1ST DEGREE - Vaginal skin & mucus membrane
2ND DEGREE - 1st degree + muscles of vagina
3RD DEGREE - 2nd degree + external sphincter of rectum
4TH DEGREE - 3rd degree + mucus membrane of rectum
Management: Episiorrhapy
3. Vaginal
Causes
1. Primigravida
2. Operative delivery: forceps vacuum extraction, episiotomy
3. Precipitate delivery
4. Large infant
5. Multiple pregnancy
6. Abnormal fetal presentation and position
Management
1. Repair of laceration
Vulvar hematoma
Types:
1. Genital hematomas
• Vulvar hematomas – first symptom is excruciating pain.
• Vaginal hematomas – manifested by severe perineal pain, feeling of pressure,
inability to void, sudden appearance of movable and sensitive mass adjacent to
the vagina.
• Vulvovaginal hematomas
2. Retroperitoneal hematomas
Risk factors
1. Vulvar varicosities
2. Precipitate labor
3. Inadequate suturing of episiotomy or lacerations
4. Primipara
5. Prolonged 2nd stage of labor
6. Large infant
7. Forceps or vacuum assisted birth
Management:
1. If the hematoma is less than 3 to 5 cm in diameter, the physician usually orders palliative
treatments such as ice to the area for the first 12 hours along with pain medication, and
close observation of the area for extension of the hematoma. After 12 hours, sitz baths
are prescribed to replace the application of ice. Sitz baths are therapeutic in providing
comfort and in facilitating reabsorption of the clot.
2. A hematoma larger than 5 cm may require incision and drainage with the possible
placement of a drain.
3. Blood transfusion if severe bleeding occurs.
Complications
1. Infection
2. May interfere with adequate milk production in breastfeeding mothers as it may
continue to produce hormones like estrogen that inhibit prolactin secretion.
Management
1. Confirmation is done by:
• Manual uterine exploration
• Ultrasound can show residual trophoblastic tissue, blood clots, and decidua in the
uterus.
2. D and C to remove adherent placenta.
3. Hysterectomy if manual removal/ D and C is not successful or will result in grave
complications such as severe hemorrhage, DIC and perforation of the uterus.
C. Subinvolution
• incomplete return of the uterus to its prepregnant size, shape and function.
Causes
1. Retained placental fragments
2. Infection – endometritis
3. Uterine tumors
Management :
• Dilatation and Curettage (D&C) except placenta increta, percreta
Acreta – attached placenta to myometrium.
Increta – deeper attachment of placenta to myometrium
Percreta – invasion of placenta to perimetrium
• manual extraction of fragments
• massaging of uterus.
• Treat the cause: antibiotics for infection; removal of uterine tumors, evacuation of
retained placental fragments by D and C
Puerperal Sepsis
➢ Any infection of the reproductive organs that occurs within the first 6 weeks after childbirth
➢ Affects 2-8% of postpartum women.
➢ Defined by two documented temperatures greater than 380C(100.4F) on any two successive
days after the first 24 hours following delivery in the absence of any other apparent fever
source
Risk Factors:
Operative delivery (CS-20x)
Prolonged labor (>18 hours) /rupture of membranes (>24 hours)
Multiple pelvic examinations
Lower socioeconomic status
Intrapartum fever or diagnosis of chorioamnionitis
Obesity
History of UTI and STDs
Internal fetal monitoring
Anemia
Malnutrition
Assessment Findings:
Fever, chills and tachycardia
Change in the color, amount, odor (foul) and consistency of lochia
Painful and tender uterine fundus, delayed uterine involution
Body malaise, anorexia, headache
Burning sensation on urination, dysuria
Prognosis
Improved with early detection and appropriate management
Prevention
prevention and early treatment of anemia in pregnancy
strict asepsis
prevention of lacerations – warm compress, sitz bath, perineal heat lamp
good management of the 3rd stage
perineal cleanliness
adequate treatment of dystocia and PROM with antibiotics
hygiene and proper attire of personnel
Management
Start with ordered antibiotics
Maintain bedrest
Observe standard precautions
Monitor v/s, I and O
Teach perineal hygiene
Provide nutritious, high caloric, high protein, high iron diet
A. Endometritis
➢ Infection of endometrial lining
➢ Chorioamnionitis and cesarean birth
➢ Tubal scarring and interference with future fertility
Causative Agents:
Microorganisms of the vaginal flora:E. coli, klebsiella pneumoniae and proteus species
Group A streptococci – postpartum day 1 or 2
E. coli, anaerobic bacteria – day 3 and 4
Chlamydia trachomatis – more than 7 days
Anaerobic gram-negative bacilli – following CS
Manifestations:
Fever (3rd or 4th day)-greater than 100.4 degree-Farenheit
Tachycardia
Malaise, chills, backache, headache
Profuse foul smelling lochia-remains rubra longer
Excess fundal tenderness
Delayed involution (lochia doesn’t follow RSA)
Boggy and enlarged uterus
Management
Administration of antibiotic, oxytocic and analgesic
Increase fluid intake
Bedrest with BRPs
Assess pain and involution
Sitting in fowler’s/walking
Good perineal hygiene
B. Wound infection
Nursing Assessment
1. REEDA
Redness
Edema
Ecchymosis
Discharge
Approximation of skin edges
2. Generalized fever, localized tissue warmth
3. Tenderness
Perineal Infections
Usually occur on the 3rd – 4th day after giving birth
Common causes:
a. Infected lochia (endometritis)
b. Fecal contamination of the wound
c. Poor hygiene
Causative agents: microorganisms of the vaginal flora
Abdominal wound infections
Often results from contamination of CS incision by m.o of the vaginal flora
Most common risk factors:
a. DM
b. Hypertension
c. Obesity
d. Treatment with corticosteroids
e. Immunosuppression
f. f. Chorioamnionitis
g. Prolonged labor
h. Prolonged rupture of membrane
i. Prolonged operating time
j. Excessive blood loss
k. Abdominal twin delivery
Management
Antibiotic therapy
Good perineal hygiene
Semi-fowler’s position to promote drainage
Analgesic for pain
Increase fluid intake
C. UTI
➢ Bacterial invasion of the kidneys or bladder (CYSTITIS) usually caused by Escherichia coli
➢ Often appears 2-3 days after birth
➢ Common during puerperium because of trauma to the bladder after delivery, urinary
retention and overdistention of the bladder due to anesthesia, catheterization
Risk Factors
CS
Forceps and vacuum delivery
Tocolysis
Induction of labor
Maternal renal disease
Pre-eclampsia, eclampsia
Previous UTI during pregnancy
Assessment Findings
Low-grade fever
suprapubic pain, flank pain
hematuria
Pain/burning on urination
Urinary frequency
Urgency of urination
Management
Administer antibiotics as ordered: 3-7 days, ciprofloxacin, amoxicillin, norfloxacin
Provide warm baths and allow to void in water to alleviate painful voiding.
Force fluids. You may give 3 liters of fluid per day
Encourage measures to acidify urine (acid-ash diet).
Analgesics for pain
Regular emptying of the bladder
Provide client teaching and discharge planning concerning
a. Avoidance of tub baths
b. Avoidance of bubble baths that might irritate urethra
c. Importance for women to wipe perineum from front to back
d. Increase in foods/fluids that acidify urine.
Predisposing Factors
• The fibrinogen level is still elevated from pregnancy
• Dilatation of lower extremity veins is still present
• The relative inactivity of the period or a prolonged time spent in delivery or birthing room
stirrups
Risk Factors
• Obesity
• Varicose veins
• Previous thrombophlebitis
• Older than 30 y/o with ↑ parity
• High familial incidence
• Smoking
• Use of estrogen supplement
• Anesthesia, surgery
• DM
Causes
1. Injury to blood vessels usually occur during delivery, indwelling catheterization and
infection.
2. Increased blood clotting that normally occurs during pregnancy and after delivery and
with the use of oral contraceptives.
3. Blood stasis that occurs as a result of varicose veins, bed rest after CS and prolonged
inactivity.
Management
1. Prevention
a. Early ambulation after delivery
b. Use of support stockings in women with varicosities
c. Avoid trauma on extremities: Pad stirrups well, limit the time a woman remains in OB
stirrups, avoid pressure on popliteal vessels
d. Avoid activities that contribute to venous stasis such as prolonged bed rest, standing
and sitting.
2. Superficial venous thrombosis involving small clots in the absence of infection usually
resolves without anticoagulant treatment. The management is directed towards relief of
pain and resolution of clot:
• Application of heat to relieve pain
• Aspirin and ibuprofen. Aspirin should never be given to woman who is receiving
heparin.
• Avoid massaging the area
3. Deep vein thrombosis requires intensive management to prevent serious complications
like pulmonary embolism.
• Hospitalization during the acute phase
• Bedrest
• Leg elevation
• Anticoagulant therapy
-Heparin – keep antagonist, protamine sulfate, available
- mother may breastfeed infant as it is not passed to breastmilk.
- Dicumarol – keep antagonist, vitamin K, available.
- passed on breastmilk
• Monitor:
1. partial thromboplastin time (PTT) – should be done before the first dose of
heparin is given, whenever the dosage level is changed, when heparin has
reached a constant level in the blood.
- Normal: between 60-70 seconds
- Low levels in a patient on heparin indicate underdosage (increase dosage);
high levels indicate too much heparin is present (reduce dosage)
2. activated partial thromboplastin time (APTT) – normal: between 25-40 secs.
- An APTT longer than 100 secs indicates spontaneous bleeding.
Educate patient on home coagulant therapy:
- Report abnormal signs: fever, bleeding gums, nose bleeds, headache,
hemoptysis, hematemesis, petechiae, spontaneous bruising, heavy
bleeding, dizziness, sudden abdominal pain, blood in urine and stool,
muscle and joint pain.
- Avoid aspirins and NSAIDS while taking anticoagulants
• Apply warm wet compress dressings to promote circulation and for comfort.
• Administer prescribed antibiotic to combat infection and analgesic to relieve pain.
• Surgery may be used if the affected vein is likely to present a long term threat of
producing blood clots that will release emboli.
Risk factors
1. Primiparity
2. Ambivalence about maintaining the pregnancy
3. History of postpartum depression or bipolar illness
4. History of mood disorder or premenstrual dysphoric disorder (not necessarily the milder
and less-specific menstrual syndrome.
5. Lack of social support
6. Family history of depression, bipolar illness, and/or anxiety
7. Marital dissatisfaction
8. Anxiety/depression during pregnancy
9. Infant-related stressors such as problematic temperament in the baby
10. Adverse life-events or stressors
11. Inadequate support from family or friends
12. Body image and eating disorders
Treatment
1. Medication – selective serotonin reuptake inhibitors such as sertraline (Zoloft), paroxetine
(Paxil) and fluoxetine (Prozac)
2. Individual/group psychotherapy
3. Practical assistance with child care and other demands of daily life
3. Postpartum psychosis
➢ The most severe and the rarest postpartum psychiatric disorder
➢ Has an incidence rate of 1-2 per 1,000
➢ Evident within the first 3 months postpartum
Risk Factors
1. Previous puerperal psychosis
2. History of manic depressive disorder
3. Obsessive personality
4. Family history of mood disorder
5. Prenatal stressors
Symptoms
1. Agitation
2. Hyperactivity
3. Insomnia
4. Lability
5. Confusion
6. Irrationality
7. Difficulty remembering or concentrating
8. Poor judgment
9. Delusions
10. hallucinations
Management
1. Hospitalization especially when woman exhibits signs of postpartum psychosis such as
hallucinations and delusions. This is because psychotic women are at risk of committing
suicide and in very rare cases, of harming their unborn child or infants.
2. Removal of infant from the mother for safety considerations.
3. Medications: antipsychotic medications, sedatives
4. Electroconvulsive therapy is the last resort if other treatment fails,
5. Psychotherapy (long term)
4. Postpartum onset panic disorder – characterized by frightening panic attacks that include
acute onset of anxiety, fear, rapid breathing, palpitation, and a sense of doom.
Assessment
1. History of previous psychological problems
2. Adequacy of coping skills
3. Degree of self-esteem
4. Presence of mood swings, emotional distress, restlessness, irritability, guilt, extreme anxiety
about the baby, anorexia, inability to complete activities of daily living
Planning/goal-setting
1. Client and family will recognize common postpartum psychological changes.
2. Client will be free from psychological maladaptation.
3. Client and family will recognize signs of psychological impairment and will contact
appropriate resources at time of discharge;
4. Client will function adequately as a parent.
Implementation
1. Observe client with baby, by herself, and with family and friends
2. Recognize early signs of problems
3. Seek client referral to psychiatrist for evaluation of psychological status
4. Support positive parenting behaviors
5. Discuss client’s plans for her baby and herself
6. Refer client to social services, if indicated
Evaluation
1. Client uses appropriate coping strategies to care for self and baby
2. Client has realistic expectations for self and baby.
3. Client perceives that she is receiving the support she needs.
4. Client has support for depressive episodes.
5. Client and family share feelings and concerns openly.
6. Appropriate bonding is observed and baby is safe.
Teacher’s Insight:
Most women do not experience a complication during the postpartum period, but when they
do it can be life threatening and disruptive to the family unit. Hemorrhage, coagulation
disorders, and infections are the primary physiological complications. Women may also
experience problems later in life due to tissue and reproductive organ trauma related to the
childbirth. Postpartum depression and postpartum psychosis are the main psychological
complications.
A focus of postpartum nursing care is to reduce women’s risks for complications related to
childbirth and to identify complications early for prompt interventions. The woman needs to
be evaluated by her health care provider when a complication is suspected.