PREDISPOSING PRECIPITATING

AGE(58) PRECANCEROUS LESIONS (BENIGN

SEX(WOMEN) POLYPS)
GENETICS RESIDENCE/ENVIRONMENT(URBAN)
HORMONES OCCUPATION (FARMER)
OBESITY
STRESS

CANCER CELLS

SINGLE INITIAL PROLIFERATIVE

PROLIFERATIVE CELL POPULATION
CELL IN EPITHELIAL CELL

FAILURE OF IMMUNE
SYSTEM RESPONSE

PROLIFERATIVE
CELL POPULATION
IN EPITHELIAL CELL

SMALL BENIGN
NEOPLASM

FUNCTION OF CELL OF IMMUNE

ROUNDS OF CONAL SYSTEM IN RESPONSE TO
SELECTION SPECIFIC ANTIGEN INVADING THE
BODY

GROWTH OF
ADINOMAS(BENIGN
TUMORS)

INVASION OF BASAL LAMINA SUPPORTING

TUMOR CELLS IN STRUCTURE AND SCAFOLDING
BASAL LAMINA THE EPITHELIAL TISSUE
 MALIGNANT NEOPLASM
MULTIPLE CHANGES IN
ABNORMAL/DAMAGED CELL
CHROMOSOME
GROWTH AND
(DUPLICATION/
MULTIPLICATION
DELETION)

PROLIFERATION AND SPREAD

OF CANCER CELLS THROUGH
THE CONNECTIVE TISSUES IN
THE COLON

PENETRATION OF CANCER
CELLS IN THE COLON WALLS

INVASION OF TUMOR TO ABDOMINAL

ORGANS (INTESTINE)

BOWEL
OBSTRUCTION

FUNCTIONAL PHYSICAL
(DISRUPT MECHANICAL BLOCKAGE OF
PERISTALSIS) BOWEL LUMEN

PARTIAL
DECREASE COMPLETE
(GAS, STOOL AND
FREQUENCY OF (NOTHING CAN
LIQUID CAN PASS OBSTIPATION
BOWEL MOVEMENT PASS) ABSENT OF BOWEL
STILL PASS)
MOVEMENT
 ileus: "blockage" No
peristalsis

Bloating
DUE CONTINUOUS cramping
ACCUMULATION OF GI anorexia
IRRITATION OF AUTONOMIC diffuse visceral
CONTENTS (CONTAINS SALT
NERVES IN VISCERAL abdominal pain
AND OTHER ACTIVE
PERITONEUM
SOLUTES)

T1, T2,T3,T4

DUE TO INCREASE IN DUE TO BLOOD VESSEL

SIZE OF NEOPLASTIC SUPPLY OF
INCREASE SIZE AND/OR
GROWTH LOCAL EXTENT OF PRIMARY
OXYGEN
OBSTRUCTION OF TUMOR
NUTRIENTS
CLOSEST ORGAN/VESSEL
(RIGHT URETER)

SEVERE ABDOMINAL PAIN LEADS TO ANEMIA:

PERITONITIS
GUARDING CONTINUED INCREASE BOWEL HEMOGLOBIN: 105 g/L-89 g/L
RIGIDITY DISTENTION AND LUMINAL
HYPERACTIVE BOWEL PRESSURE
HEMATOCRIT: 0.30%-0.26%
SOUND
ABSENT BOWEL SOUND
(ILEUS) (Patient's lab result)
HIGHER LUMINAL PRESSURE
MAY FORCE REGURGITATION

NAUSEA/VOMITING
 INCREASE PRESSURE SQUEEZES SHUT
INTESTINAL BLOOD VESSEL BOWEL ISCHEMA
DECREASE BOWEL AND INFARCTION
PERFUSION TISSUE NECROSIS
MELENA
POSSIBLE
PERFORATION
BACTERIAL INVASION
MEDICAL (INCREASE WBC
TREATMENT COUNT-----> SEE LAB
RESULT)

PHARMACOLOGICAL
SURGICAL MANAGEMENT
MANAGEMENT

COMPLETE REMOVAL OF TUMOR

RELIEVE OBSTRUCTION IN THE GI AND GU TRACT
-DIAGNOSTIC SURGERY PREVENT HEMORRHAGE
-PREVENTIVE SURGERY DRAIN ABSCESSES
-CURATIVE SURGERY
-RECONSTRUCTIVE
SURGERY
-PALLIATIVE SURGERY

NURSING MANAGEMENT BASED ON

IDENTIFIED NURSING PROBLEM
(POST-OPERATIVE CARE)
PROGNOSIS
NCP No. 1

Acute pain r/t post-surgical incision

NCP No. 2

BAD PROGNOSIS GOOD PROGNOSIS Activity intolerance r/t presence of post-

operative surgical incision
Continuous blood loss Removal of tumor can relieve mass
May lead to Fluid and effect, relieves symptoms immediately NCP No. 3
electrolyte imbalance Removes cancer cells
Decrease in appetite, may lead Relieve nerve pain or pressure Impaired Skin Integrity r/t mechanical
to malnutrition Remove a block in digestive area or interruption of skin tissues secondary to
Deprivation of nutrients in other area surgical procedure as evidenced by
normal cell due to malignant Prevents the spread of cancer cells to presence of post-operative wound
neoplasm other body parts
Metastasis improve quality of life
DEATH