Professional Documents
Culture Documents
EBOOK Ecgs Made Easy E Book 6Th Edition Ebook PDF Download Full Chapter PDF Docx Kindle
EBOOK Ecgs Made Easy E Book 6Th Edition Ebook PDF Download Full Chapter PDF Docx Kindle
(Ebook PDF)
Visit to download the full and correct content document:
https://ebookmass.com/product/ecgs-made-easy-e-book-6th-edition-ebook-pdf/
https://t.me/MBS_MedicalBooksStore
https://t.me/MBS_MedicalBooksStore
ELSEVIER
3251 lllverport Lane
St. Louis, Mbsouri 63043
No part ofthil publication may be reproduced or transmitted in any form or by any mean1, electronk or mechani-
cal, including photocopying, .recording. or any in!ormali.on stDrap and Rtrieval. systmn. without permilaion in
writing from the publiaher. Details on how to 1eek penniasion. further information about the Publither's permi.l-
si.OIIJ policies and our arrangements with organi2al:ian.t such as the Copyright Clearance Center and the Copyright
Licenalng Agency, can be fuWld at our webalte: www.ellevier.com/permiaaiona.
This book and the lndlvldual contributl01111 contained In It are protected Wlder copyright by the Publilher (other
than u may be noted herein).
Notices
Knowledge and best practice in this field aR constantly changing. N new research and experience broaden
our undemanding, changes in research method.t, profesrional practices, or medical treatment may become
neceuary.
Practitioner~ and researchers must alwaya rely on their own aperlence and knowledge In evaluating and
uaing any Information, methods, compound&, or aperlmentl described herein. In usiDg tuc.h information or
method. they should be mindful of their own takty and the takty of others, including parties fur whom they
have a professional Rtpoll8ibility.
With respect. to any drug or pharmaceutical pmduct& ident:ified. reader• are adviled to check the mo1t
cun~nl information provided (i) on procedURS fealuRd or (ii) by the manufacturer of each product to be
administered. to verify the .recommended dose or formula, the method and duration of adminiltration, and
contnindlcatiOIIJ. It is the responsibility of practitioners, relying on their own experience and knowledge of
their patients, to make diagnose~, to determine doaget and the belt t.reatment for each individual patienl, and
to take all appropriate sarety precautionl.
To the fulli:st ment of the law, neither the Publisher nor the alrthon, contributors, or editor1, UNme
any liability fur any injury and/or damage to peraom or property u a matter of produculiabllity, nesJigence
or otherwile, or from any uae or operation of any method&, product., imtruction1, or ideu contained in the
m:rterial herein.
https://t.me/MBS_MedicalBooksStore
Executive Contel!t Stmteglst: Sandra Clark
Content Developmettt SpeclalUU: Laura SeltlrtiMelissa. Kinsey
Publishing Semcu Manager. Deepthi Unni
SenWr Project MaMpr. Umarani Na1arajan
~ Dirmton: Brian Saliabury
Worktng together
to grow l!b-r.arLC'~ Li1
Printed in Canada
de•n:l oping: cnuntrm(:~
iii
I would like to thank the manuscript reviewers foc their commenb and suggestiom. Areu of this
text were rewritten, reorganized, and clarified because of your effom.
I would also like to thank the following health care professionals, who provided many of the
rhythm strips used in this book: Andrew Baird, CEP; James Bratcher; Joanna Burgan, CEP; Holly
Button, CEP; Gretchen Chalmers, CEP; 'Ihomas Cole, CEP; Brent Haines, CEP; Paul Honeywell,
CEP; Timothy Klatt. RN; Bill Loughran. RN; Andrea Lowrey, RN; Joe Martinez, CEP; St.ephanos
Orphanidis, CEP; Jason Payne, CEP; Steve Ruehs, CEP; Patty Seneski, RN; David Stockton, CEP;
Jason Stodghill, CEP; Dionne Socie, CEP; Kristina Tellez, CEP; and Fran Wojculewicz, RN.
A special thanks to Melissa Kinsey for her humor, guidance, advice, and impeccable attention
to detail throughout this project.
iv
To
Deepak C. Patel, MD
vi
Barbara Aehlert, MSBd, BSPA, RN, has been a registered nurse for more than 40 years. with
clinical experience in medicallsurgical nursing. critical care nursing, prehotpital education, and
nursing education. Barbara i.s an active CPR and Advanced Cardiovascular Life Support (ACLS)
instructor with a special interest in teaching basic dysrhythmia recognition and ACLS to nurses
and paramedics.
vii
1 ANATOMY AND PHYSIOLOGY, 1 3 SINUS MECHANISMS, 78
viii
Contents
KEY TERMS
acute coronary syndrome (ACS): A term used to referto distinct base of the heart: Posterior surface of the heart.
conditions caused by a similar sequence of pathologic events- blood pressure: Force exerted by the blood against the walls of the arter-
a temporary or permanent blockage of a coronar_y artery. These ies as the ventricles of the heart contract and relax.
conditions are characterized by an excessive dem nd or inadequate cardiac output (CO): The amount of blood pumped into the aorta each
supply of oxygen and nutrients to the heart muscle associated minute by the heart; defined as the stroke volume multiplied by the
with plaque disruption, thrombus formation, and vasoconstriction. heart rate.
ACSs consist of three major syndromes: unstable angina, non- chordae tendineae (tendinous cords): Thin strands of fibrous connec-
ST-elevation myocardial infarction, and ST elevation myocardial tive tissue that extend from the AV valves to the papillary muscles that
infarction. prevent the AV valves from bulging back into the atria during ventricular
afterload: The pressure or resistance against which the ventricles must systole (contraction).
pump to eject blood. chronotropy: A change in (heart) rate.
angina pectoris: Chest discomfort or other related symptoms of sudden diastole: Phase of the cardiac cycle in which the atria and ventricles relax
onset that may occur because the increased oxygen demand of the between contractions and blood enters these chambers. When the term
heart temporarily exceeds the blood supply. is used without reference to a specific chamber of the heart, ventricular
apex of the heart: Lower portion of the heart that is formed by the tip of diastole is implied.
the left ventricle. dromotropy: Refers to the speed of conduction through the AV junction.
atria: Two upper chambers of the heart (singular, atrium). dysrhythmia: Any disturbance or abnormality in a normal rhythmic pat-
atrial kick: Blood pushed into the ventricles because of atrial contraction. tern; any cardiac rhythm other than a sinus rhythm.
atrioventricular (AV) valve: The valve located between each atrium ejection fraction: The percentage of blood pumped out of a heart cham-
and ventricle; the tricuspid separates the right atrium from the right ber with each contraction.
ventricle, and the mitral (bicuspid) separates the left atrium from the endocardium: Innermost layer of the heart that lines the inside of the
left ventricle. myocardium and covers the heart valves.
atypical presentation: Uncharacteristic signs and symptoms perceived epicardium: Also known as the visceral pericardium; the external layer of
by some patients experiencing a medical condition, such as an ACS. the heart wall that covers the heart muscle.
1
Chapter 1 Anatomy and Physiology
hBart failure: Acondition In whlctl the heart Is unable tD pump enough pi'Oldmal: Location nearer to the midline of the body or the point of
blood to meet the metabolic needs of the body; It may result from any attachment than something else Is.
cond~ion that impairs preload, afterload, cardiac contractility, or heart sarcolemma: Membrane that covers smooth, striated, and cardiac
rare. muscle fibers.
inDtropy: Refers to a change in myocardial contractility. sarcomere: Smallest functional un~ of a myofibril.
ischemia: Decreased supply of oxygena1ed blood tn a body part or organ. sarcoplasm: SemWiuid cytnplasm of muscle cells.
mediastinum: Middle area of the thoracic cavity; contains the heart, great sarcoplasmic reticulum: Network of tubules and sacs that plays an
vessels, trachea, and esophagus, among other structures; extends from important role in muscle contraction and relalration by releasing and
the sternum to the vertebral column. storing calcium Ions.
mltochondrta: The energy-producing parts of a cell. semilunar (SL) valves: Valves shaped like half-moons that separate the
mvocardlallnfarctlon (M~: Death of some mass of the heart muscle ventricles from the aorta and pulmonary artery.
caused by an Inadequate blood supply. septum: An lntBmal wall of connective tissue.
mvocardlum: Middle and thickest layer of the heart; contains the cardiac stroke volume {SV): The amount of blood e]eclBd from a ventricle with
muscle fibers that cause contraction of the heart and contal ns the each heartbeat
conduction system and blood supply. sulcus: Groove.
myofibril: Slender striated strand of muscle tissue. systole: Contraction of the heart (usually refarri ng to ventricular contrac-
papillary muscles: Muscles attached to the chordae mndineae of the AV tion), during which blood is propelled intn the pulmonary artery and
valves and the ventricular muscle of the heart that help prevent the AV aorta; when the tenn is used without reference to a specific chamber of
valves from bulging too far intn the abia. the heart, ventricular systole is implied.
pericardium: A double-walled sac thai erdoses the heart and helps tone: A term that may be used when referring to the normal state of bal-
protect It from trauma and Infection. anced tension In body tissues.
peripheral resistance: Resistance to the flow of blood determined by venous return: Amount of blood flowing lntn the right atrium each minute
blood vessel diameter and the tone of the vascular musculature. from the syslemlc c1rculatlon.
preload: Force exerted by the blood on the walls of the venb1cles at the venb1cles: The two lower chambers of the heart
end of diastole.
LOCATION, SIZE, AND SHAPE formed by portions of the right atrium and the left and right
vmtrides (Fig. 1.4). However, because the heart is tilted
OFTHEHEART slightly toward the left in the chest, the right ventricle is the
[Oblectlve 1] area of the heart that lies most directly behind the sternum.
The heart is a hollow muscular organ that lies in the space The apa, or lower portion, of the heart is formed by the tip
between the lungs (i.e., the mediastinum) in the middle of of the left ventricle. The apex lies just above the diaphragm
the chest (Pig. 1.1). It sits behind the stemwn and just above at about the level of the fifth intercostal space in the midcla-
the diaphragm. About two thirds of the heart lies to the left vicular line.
of the midline of the stemwn. The remaining third lies to the The heart's left side (i.e., left lateral surface) faces the
right of the sternum. left lung and is made up mostly of the left ventricle and a
The adult heart is about 5 inches ( 12 an) long, 3.5 inches portion of the left atrium. The right lateral surface faces
(9 em) wide, and 2.5 inches (6 em) thick (Fig_ 1.2). It typically the right lung and consists of the right atrium. The heart's
weighs between 250 and 350 g (about 11 oz) and is about bottom (i.e., inferior) surface is formed primarily by the
the size of its owner's fist The weight of the heart is about left ventricle, with small portions of the right ventricle
0.4596 ofa man's body weight and about 0.40% ofa woman's, and right atrium. The right and left ventricles are sepa-
A person's heart size and weight are influenced by his or her rated by a groove containing the posterior interventricu-
age, body weight and build. frequency of physical exercise, lar vessels. Because the inferior surface of the heart rests
and heart disease. on the diaphragm, it is also called the diaphragmatic sur-
face (Fig. 1.5).
Mldclavlcular
line
Fig. 1.1 Antar1or v1aw of tha chest wall of a man lhM!rg skslalal structullls and
the surface projactlon of the heart (From Draka R, Vogl AW, Mlb:hall AWM: Gtay's
8II8JDmy for studsnts, ed 3, New York, 2015, Churchill LMngstooe.) Fig. 1.2 Appean~nca of 1h& heart. This pho!Dgraph shows a living human heart
p111pered for transplan1al!on Into a paUent. NoiB liB slza llllaUveiD 1he hands that Rill
hading 1t. (From PaiiDn KT, Thlbolil&u GA: Anatomy& physiology. &d 9, St. Louis.
2016, Mosby.)
Anlartor
lntervent~wer
branch of left
coronary artery
Greal canlac vein
Obtuee mergln
Fig. 1.3 The base af the heart. (Ffm1 Drake R, 'ql A.W, Milcrell A."WM: &ay's
anaiDmy for stJJdents, ed 3, New York, 2015, Churchill Uvingslune~
Fig. 1.4 Th& ant:&r1or surface of the haart. (From Drake R, Vogl AW, Mitchell
The right and left phrenic nerves, which innervate the dia- AWM: Gray's anatomy frJr stJJd6nts, &d 3, Naw York, 2015, Churchill L.Mrgstona.)
phragm, pass through the fibrous pericardium as they
descend to the diaphragm. Because these nerves sup- The inner layer of the pericardium, the serous pericar-
ply sensory fibers to the fibrous pericardium, the parietal dium, consists of two layers: parietal and visceral (Fig. 1.7).
serous pericardium, and the mediastinal pleura, discomfort the parietal. layer lines the inside of the fibrous pericardium.
related to conditions affecting the pericardium may be felt
The visceral layer attaches to the large vessels that enter and
In the areas above the shoulders or lateral neck.
exit the heart and covers the outer surface ofthe heart muscle
(ie., the epicardium).
of the structures around it. such as the sternum and dia- Between the visceral and parietal layers is a space (the
phragm, by means of ligaments. This helps prevent exces- pericardia! space) that normally contains about 20 mL of
sive movement of the heart in the chest with changes in serous (pale yellow and transparent) fluid. This fluid acts as a
body position. lubricant, preventing friction as the heart beats.
If the pericardium becomes Inflamed (pericarditis), excess Heart surgery or trauma to the heart, such as a stab wound,
pericardia! fluid can be quickly generated in response to the can cause a rapid buildup of blood in the pericardia! space. The
inflammation. Pericarditis can result from a bacterial or viral buildup of excess blood or fluid in the pericardia! space com-
infection, rheumatoid arthritis, tumors, destruction of the presses the heart. This can affect the heart's abiily to relax and
heart muscle in a heart attack, among other causes. fill with blood between heartbeats. Ifthe heart cannot adequately
Chapter 1 Anatomy and Physiology
fill with blood, the amount of blood the ventricles can pump out shock. Conversely, 1000 mL of fluid may build up over a lon-
to the body (cardi~ output) will be decreased. As a result, the ger period without any significant effect on the heart's ability
amount of blood returning to the heart is also decreased. These to fill. This is because the pericardium accommodates the
changes can result in a life-threatening cond~ion called C8ldiac increased fluid by stretching over time.
temponade. The amount of blood or fluid in the pericardia! The symptoms of cardiac tamponade can be relieved
space needed to impair the heart's ability to fill depends on the by removing the excess fluid from the pericardia! sac.
rate at which the buildup of blood or fluid occurs and the ability Pericardiocentesis is a procedure in which a needle is
of the pericardium to stretch and accommodate the increased inserted into the pericardia! space and the excess fluid
volume of fluid. is sucked out (aspirated) through the needle. If scarring is
The rapid buildup of as little as 100 to 150 ml of fluid or the cause of the tamponade, surgery may be necessary to
blood can be enough to result in signs and symptoms of remove the affected area of the pericardium.
Right Rbrous
ventricle pericardium
{cut;)
Postertor
lnt8r-
ventrlcu lar
art8ly Left verrtricla
and vein
Right Coronary
sulcus
atrium
Inferior
vena cava
Fig. 1.15 The Inferior surface ot the heart The lnfe~or part ot the fibrous pe~card urn has been removed v.tlh the dla-
pluagm. (From Gosling JA: Human anaJDmy: color atlas and text. ad 4, L..ordcn, 2002, Mosby.)
Lung
roots
Left
phrenic
nerve
Fig. 1 .& The fibrous pericanium and phrenic nerves revealed after reiTlCJ\Iill of the lungs. {From Gosling J&.: Human
anafDmy: color afias and text. ed 4, Lllndon, 2002, Mosby.)
Chapter 1 Anatomy and Physiology
Pulmonary
trunk
Fibrous
pert-
cardium
(cut)
V-.1
~--+-:ft-.,_;.;..- MI'OUS
pert-
cardium
Fig. 1.7 The fbrous pericardium has been opened to expose the visceral pericardium ~ring lhe anterior surface of the
heart. (From Gosling JA: HumaiJ anatomy: color atlas and tert; eel 4, London, 2002, Mosby.)
They feed this area first before entering the myocardium and potassium (potassium channels), and calcium (calcium
supplying the heart's inner layers with oxygenated blood. channels). When the muscle is relaxed, the calcium chan-
Ischemia is a decreased supply of oxygenated blood to a nels are closed. As a result, calcium cannot pass through
body part or organ. The heart's subendocardial area is at the the membrane of the SR. This results in a high concen-
greatest risk ofischemia because this area has a high demand tration of calcium in the SR and a low concentration in
for oxygen and it is fed by the most distal branches of the the sarcoplasm, where the muscle cells (sarcomeres) are
coronary arteries. found. If the muscle cells do not have calcium available to
them, contraction is inhibited (the muscle stays relaxed).
CARDIAC MUSCLE The force of cardiac muscle contraction depends largely
Cardiac muscle fibers make up the walls of the heart. on the concentration of calcium ions in the extracellular
These fibers have striations, or stripes, similar to that of fluid.
skeletal muscle. Each muscle fiber is made up of many
muscle cells (Fig. 1.9). Each muscle cell is enclosed in
a membrane called a sarcolemma. Within each cell (as
0 ECG Pear1 _ _ _ _ _ _ _ __
The heart consists of two syncytia: atrial and ventricular.
with all cells) are mitocho.odria, the energy-producing The atrial syncytium consists of the walls of the right and
parts of a cell, and hundreds of long, tube-like structures left atria. The ventricular syncytium consists of the walls of
called myoflbrlls. Myofibrils are made up of many sar~o the right and left ventricles. Normally, impulses can be con-
merea, the basic protein units responsible for contraction. ducted from the atrial syncytium into the ventricular syncy-
The process of contraction requires adenosine triphos- tium only by means of the atrioventricular (AV) junction. The
phate (ATP) for energy. The mitochondria that are inter- AV junction is a part of the heart's electrical system. This
spersed between the myofibrils are important sites of ATP allows the atria to contract a short time before ventricular
production. contraction.
The sarcolemma has holes in it that lead into tubes called
T (transverse) tubules. T tubules are extensions of the cell
membrane. Another system of tubules, the sarcoplasmic:
reticulum (SR), stores calcium. Muscle cells need calcium
Heart Chambers
in order to contract. Calcium is moved from the sarco- The heart has four chambers, two atria and two ventri-
plasm of the muscle cell into the SR by means of "'pumps" cles. The outside surface of the heart has grooves called
in the SR. sulci. The coronary arteries and their major branches lie
There are certain places in the cell membrane where in these grooves. The coronary sulcus (groove) encircles
sodium (Na+), potassium (K+), and calcium (Ca++) can the outside of the heart and separates the atria from the
pass. These openings are called pores or channels. There ventricles. It contains the coronary blood vessels and
are specific channels for sodium (sodium channels}, epicardial fat.
Chapter 1 Anatomy and Physiology
F1g. 1.10 lntartor of the heart. This Illustration shows the heart as It would appear If It were a.Jt along a lronllll plane and
opened Ilks a book. The fnlnt portion of the heart lies ID 1hll reader's ~ght; the back portion of the heart lias ID the reader's
Iaft. ThB four chambers Ill 1hll heart-two a~a and two van~des--an~ easily seen. A~ Abtlvant~cular; st.. semilunar. [From
Patton KT, Thllodeau GA: Anatomy & physiology, ad 9, St. llluls, 2016, Mosby.)
ATRIA VENTRICLES
[Obiactive 5] (Obiactive 5]
The two upper chambers of the heart are the right and The heart's two lower chambers are the right and left ven-
left atria (singular, atrium) (Fig. 1.10). An earlike flap tricles. Their purpose is to pump blood. The right ventricle
called an auricle (meaning "little ear·) protrudes from pumps blood to the lungs. The left ventricle pumps blood
each atrium. out to the body. Because the ventricles must pump blood
The purpose of the atria is to receive blood. The right either to the lungs (the right ventricle) or to the rest of the
atrium receives blood low in oxygen from the superior vena body (the left ventricle), the ventricles have a much thicker
cava (which carries blood from the head and upper extremi- myocardial layer than the atria. Because the right ventricle
ties), the inferior vena cava (which carries blood from the moves blood only through the blood vessels of the lungs and
lower body), and the coronary sinus (which is the largest then into the left atrium, it has one sixth of the muscle mass
vein that drains the heart). The left atrium receives freshly and one third of the wall thickness of the left ventricle, which
oxygenated blood from the lungs via the right and left pul- must propel blood to most vessels of the body (Hutchison &:
monary veins. Rudakewich, 2009) (Fig. 1.11).
1he four chambers of the heart vary in muscular wall
thickness, reflecting the degree of pressure each chamber
must generate to pump blood. For example, the atria encoun-
ter little resistance when pumping blood to the ventricles. As
a result, the atria have a thin myocardial layer. The wall of When the left ventricle contracts, it normally produces an
the right atrium is about 2 mm thick. and the wall of the left impulse that can be felt at the apex of the heart (apical
atrium is about 3 m.m thick. Blood is pumped from the atria impulse). This occurs because as the left ventricle con·
through an atrioventricular (AV) valve and into the ventri- tracts, it rotates forward. In a normal heart, this causes the
cles. The valves ofthe heart are discussed later in this chapter. apex of the left ventricle to hit the chest wall. You may be
able to sea the apical impulse in thin individuals. The api-
Q ECG Pearl _ _ _ _ _ _ _ _ __ cal impulse is also called the point of maximal impulse
because it is the site where the left ventricular contraction
Think of the atria as holding tanks or reservoirs for blood. is most strongly felt.
Chapter 1 Anatomy and Physiology
/
Left Papillary Intel'" Trabeculae Marginal
ventricular mUICia Y811lrtcular camaae arl8ry
-11 saptum
Fig. 1.11 Section through the heart shi7Mng 1he &Peal porUn of the left and ~ghl venll1clas. (From Gosling JA: Human
anatumy: oo1or atlas and t8Xt, ed 4, London, 2002, Mosby.)
Heart Valves
The heart has a skeleton, which is made up of four rings
of thick connective tissue. This tissue surrounds the bases
of the pulmonary trunk, the aorta, and the heart valves.
The inside of the rings provides secure attachments for
the heart valves. The outside of the rings provides for the
attachment of the cardiac muscle of the myocardium (Fig.
1.12). The heart's skeleton also helps form the partitions
(septa) that separate the atria from the ventricles.
There are four one-way valves in the heart: two sets of AV
valves and two sets of&emilUIW' (SL) valves. The valves open
and close in a specific sequence and assist in producing the
pressure gradient needed between the chambers to ensure
a smooth :flow of blood through the heart and prevent the
bacldl.ow of blood.
Fig. 1.12 Skeleton of the heart. This IX)Stel1or view shows part of the venll1cular
ATRIOVENTRICULAR VALVES myooardlum with 1he heart valves 81111 attached. The rim of each heart valve Is sup-
ported by a fibrous structure, called the sk8/stonofth6 h6art, which encircles all four
[Oblectlves 6, 7] valves. AV. Atrlovenll1cular. (From PatiDn KT, Thibodeau GA: Anatmny&ph~
Atrioventricular valves separate the atria from the ventricles. ed 9, St Louis, 201 6, Mosby.)
The two AV valves consist of tough. fibrous rings (annuli
:6.brosi); :flaps (lea11.ets or cusps) of endocardium; chordae
tendineae; and papillary muscles. left atrium and left ventricle (Fig. 1.14). The mitral valve is
1he tricuspid valve is the AV valve that lies between the so named because of its resemblance to a miter, which is a
right atrium and right ventricle. It consists of three separate double-cusp bishop's hat, when open.
cusps or flaps (Fig. 1.13). It is larger in diameter and thinner The AV valves open when a forward pressure gradi-
than the mitral valve. The mitral valve, which is also called ent forces blood in a forward direction. They close when
the bicuspid valve, has only two cusps and lies between the a ba.ck.ward pressure gradient pushes blood backward. The
Chapter 1 Anatomy and Physiology
Superior vena
Right
Fig. 1.13 Internal view of 1he right venll1cle. (From Drake R, Vogl AW, Mitchell AWM: Gmy~ snatrHny frJr si1Jdenls. ed 3,
New Yorll, 2015, Churchill Uvlngstone.)
Pulmonary arteries
Pulmonary veins
Coronary sinus
Fig. 1.14 Internal view of 1he left ventriCle. (From Drake R, Vcgl AW, MitChell AWM: !#a~ anatomy for students, ed 3, New
Ya'k, 2015, Chu I'Ch ill Livingstone.)
Chapter 1 Anatomy and Physiology
AV valves require almost no backflow to cause closure ends and the pressure in the pulmonary artery and aorta
(Hall, 2016). exceeds that of the ventricles.
The flow of blood from the superior and inferior venae
cavae into the atria is normally continuous. About 70%
of this blood flows directly through the atria and into the
ventricles before the atria contract; this is called passi'o'e
filling. & the atria fill with blood, the pressure within the Improper valve function can hamper blood flow through the
atrial chamber rises. This pressure forces the tricuspid heart. Valvular heart disease is the term used to describe
and mitral valves open, and the ventricles begin to fill, a malfunctioning heart valve. Types of valvular heart dis-
gradually increasing the pressure within the ventricles. ease include the following:
When the atria contract, an additionallO% to 30% of the • vaJvular prolapse. If a valve flap inverts, it is said to have
returning blood is added to filling of the ventricles. This prolapsed. Prolapse can occur if one valve flap is larger
additional contribution of blood resulting from atrial than the other. It can also occur if the chordae tendin-
contraction is called atrial kick. On the right side of the eae stretch markedly or rupture.
heart, blood low in oxygen empties into the right ventri- • vaJvutar regurgitation. Blood can flow backward, or
cle. On the left side of the heart, freshly oxygenated blood regurgitate, if one or more of the heart's valves does
not close properly. Valvular regurgitation Is also known
empties into the left ventricle. When the ventricles then
as valvular incompetence or valvular insufficiency.
contract (i.e., systole), the pressure within the ventricles
• Valvular stenosis. If a valve narrows, stiffens, or thick-
rises sharply. The tricuspid and mitral valves completely ens, it is said to be stenosed. The heart must work
close when the pressure within the ventricles exceeds that harder to pump blood through a stenosed valve.
of the atria. Papillary muscles receive their blood supply from the
Chordae tendineae (tendlnoua cords) are thin strands coronary arteries. If a papillary muscle ruptures because
of connective tissue. On one end, they are attached to the of an inadequate blood supply (as in myocardial infarc-
underside of the AV valves. On the other end, they are tion), the attached valve cusps will not completely
attached to small mounds of myocardium called papillary close and may result in a murmur. If a papillary muscle
maades. Papillary muscles project inward from the lower in the left ventricle ruptures, the leaflets of the mitral
portion ofthe ventricular walls. When the ventricles contract valve may invert Q.e., prolapse). This may result in blood
leaking from the left ventricle into the left atrium (e.g.,
and relax, so do the papillary muscles. The papillary muscles
regurgitation} during ventricular contraction. Blood flow
adjust their tension on the chordae tendineae, preventing
to the body o.e., cardiac output) could decrease as a
them from bulging too far into the atria. For example, when result.
the right ventricle contracts, the papillary muscles of the
right ventricle pull on the chordae tendineae. 1he chordae
tendineae prevent the flaps of the tricuspid valve from bulg-
ing too far into the right atrium. 1hus, the chordae tendineae
and papillary muscles serve as anchors. Because the chordae HEART SOUNDS
tendineae are thin and string-like, they are sometimes called Heart sounds occur because of vibrations in the tissues
"heart strings." of the heart caused by the closing of the heart's valves.
Vibrations are created as blood flow is suddenly increased
SEMILUNAR VALVES or slowed with the contraction and relaxation of the
[OIJiactlve B] heart chambers and with the opening and closing of the
The pulmonic and aortic valves are SL valves. 1he SL valves pre- valves.
vent the bacldlow ofblood from the aorta and pulmonary arter- Normal heart sounds are called Sl and S2. 1he first heart
ies into the ventricles. 1he SL valves have three cusps shaped sound ("lubb,.) occurs during ventricular contraction when
like half-moons. 1he openings of the SL valves are smaller than the tricuspid and mitral (AV) valves are closing. The second
the openings of the AV valves, and the flaps of the SL valves are heart sound ("dupp) occurs during ventricular relaxation
smaller and thicker than the AV valves. Unlike the AV valves, as the pulmonic and aortic (SL) valves close. A third heart
the SL valves are not attached to chordae tendineae. sound is produced by ventricular filling. In those younger
When the ventricles contract, the SL valves open, allow- than 40 years ofage, the left ventricle normally permits rapid
ing blood to flow out of the ventricles. When the right filling. The more rapid the ventricular filling, the greater
ventricle contracts, blood low in oxygen flows through the likelihood of hearing a third heart sound. A third heart
the pulmonic valve into the pulmonary trunk. which sound (S3) heard in people older than 40 years ofage is con-
divides into the right and left pulmonary arteries. When sidered abnormal An abnormal third heart sound is fre-
the left ventricle contracts, freshly oxygenated blood flows quently associated with heart failure. An Sl-S2-S3 sequence
through the aortic valve into the aorta and out to the body is called a ventricular gallop or gallop rhythm. It sounds like
(Fig. 1.15). The SL valves close as ventricular contraction "Kentucky"-Ken (Sl) -tuck (S2) -y (S3). The location of the
Chapter 1 Anatomy and Physiology
Pulmonary velr1s..,::-----~...4
Superior vena Aortic valve
cusps
Left atrium7'9~L~
Aorta --r--ollil.l~ Right
venlr1cle
lntervemrtcular
septum
Tricuspid
valve
Mitral valv&-
posterior cusp
Right ventricle
Fig. 1.1& Drawing of a heart split perpendicular to the interventriCular septum to illustrate the anatomic relationships of
the leaflets of the atroventricular and aortiC valveS. (From Koeppen BM, Stanton BA: Beme & LevyJJ/1YSiOlOgY. ed 6, St. L.Duis,
2010, Mosby.)
heart's AV and SL valves for auscultation is shown in Fig. the heart are called epicardial coronary arteries. They branch
1.16. A summary ofthe heart's valves and auscultation points into progressively smaller vessels, eventually becoming arte-
for heart sounds appears in Table 1.2. rioles, and then capillaries. Thus, the epicardium has a rich
blood supply to draw from. Branches of the main coronary
arteries penetrate into the heart's muscle mass and supply the
subendocardium with blood. lhe diameter of these "feeder
branches" (i.e., collateral circulation) is much narrower. The
In people younger than 40 years of age, the left ven-
tissues supplied by these branches get enough blood and
tricle normally permits rapid filling. The more rapid the
oxygen to survive, but they do not have much extra blood
ventricular filling, the greater the likelihood of hearing a
third heart sound. A third heart sound (S3) heard in those flow.
older than 40 years of age is considered abnormal. An lhe work of the heart is important To ensure that it has
abnormal third heart sound is frequently associated with an adequate blood supply, the heart makes sure to provide
heart failure. An S 1-32-33 sequence Is called a ventricu- itself with a fresh supply of oxygenated blood before supply-
lar gallop or gallop rhythm. It sounds like Ken (S1) -tuck ing the rest of the body. This freshly oxygenated blood is sup-
(S2) -y (S3). plied mainly by the branches of two vessels: the right and left
Turbulent blood flow within the cardiac chambers and coronary arteries.
vessels can produce heart murmurs. An inflamed pericar- lhe right and left coronary arteries are the very first
dium can produce a peric8rdial friction rub, which sounds branches off the base of the aorta. The openings to these ves-
like rough sandpaper.
sels lie just beyond the cusps of the aortic SL valve. When
the left ventricle contracts (systole), the force of the pres-
sure within the left ventricle pushes blood into the arteries
The Heart's Blood Supply that branch from the aorta. 'Ihis causes the arteries to fill
lhe coronary circulation consists of coronary arteries and However, the heart's blood ~ssels (ie., the coronary arter-
veins. The right and left coronary arteries encircle the myo- ies) are compressed during ventricular contraction, reduc-
cardium like a crown. or corona. ing blood fl.ow to the tissues of the heart. Thus, the coronary
arteries fill when the aortic valve is closed and the left ven-
CORONARY ARTERIES tricle is relaxed (i.e., diastole).
[Oblectlve 8] lhe three major epicardial coronary arteries include the left
lhe main coronary arteries lie on the outer (epicardial) sur- anterior descending (LAD) artery, circumflex (Cx) artery, and
face ofthe heart. Coronary arteries that run on the surface of right coronary artery (RCA). A person is said to have coronary
Chapter 1 Anatomy and Physiology
Pulmonary valve
Fig. 1.18 An!Brlor view ot 1he chest r.owlng the heart, the looatlon of the heart's valves, and where to listen to heart
sounds. (From Drake R, Vogl AW, Mitchell AWM: Glay'unatomyfurstWents, ad 3, New York. 2015, Churchill LMngstone.)
flrachiDC&PMic
..,.,k
A B
Fig. 1.17 Coronary artar18s s'-"~ylng the h881t. 1hEI ~ght ccronary artBry sup~les tha ~ght att1urn, vantJtde, BOO
postarlor aspect of tha left ventricle In most lndMiilals. The left coronary artery dlvldas IIIIo lha left antsr1or descending BOO
ciraJmllex artelies, which perfuse the left venllide. A, Anterior view. B, Posterior view. (Frnm Copstead-Ki ll<hom I., Banasik JL.:
Pathophysiology, ed 5, Philadelphia, 2013, Elsevier.)
Left Coronary Artery A summary of the areas of the heart supplied by the three
lhe left coronary artery (LCA) originates from the left side major coronary arteries is shown in Table 1.3.
of the aorta (see Fig. 1.17). 1he 1irst segment of the LCA is
called the left main coronary artery. It is about the diameter Coronary Artery Dominance
of a soda straw and less than 1 inch (2.5 em) long. ffioc.kage In about 8596 ofpeople the RCA forms the posterior descend-
of the proximal LAD coronary artery has been referred to as ing artery, and in about 1096 of people the circumflex artery
the "widow maker" because of its association with sudden forms the posterior descending artery (Lohr & Benjamin.
cardiac arrest when it is blocked. 2016). The coronary artery that fonn.s the posterior descend-
The left main coronary artery supplies oxygenated blood ing artery is considered the dominant coronary artery. If a
to its two primary branches: the LAD, which is also called the branch of the RCA becomes the posterior descending artery,
anterior interventricular artery, and the Cx.. 1hese vessels are the coronary artery arrangement is described as a right-
slightly smaller than the left main coronary artery. dominant system. If the Cx branches and ends at the poste-
1he LAD is on the outer (ie., epicardial) surface on the front rior descending artery, the coronary artery arrangement is
of the heart It travels along the groove that lies between the described as a left-dominant system. In some people, neither
right and left ventricles (i.e., the anterior interventricular sul- coronary artery is dominant. If damage to the posterior wall
cus) toward the heart's apex. In most patients. the LAD travels of the left ventricle is suspected, a cardiac catheterization
around the apex ofthe left ventricle and ends along the left ven- usually is necessary to determine which coronary artery is
tricle's inferior surface. In the remaining patients, the LAD does involved.
not reach the inferior surface. Instead. it stops at or before the
heart's apex. 1he major branches of the LAD are the septal and ACUTE CORONARY SYNDROMES
diagonal arteries. 1he LAD supplies blood to the fullowing: [Obiactivas 9,10]
• The anterior surface of the left ventricle Aarte coronary syndrome (ACS) is a term that refers to dis-
• Part of the lateral surface of the left ventricle tinct conditions caused by a similar sequence of pathologic
• The anterior two thirds of the interventricular septum events involving abruptly reduced coronary artery blood flow.
1he Cx coronary artery circles around the left side of the This sequence of events results in conditions that range from
heart in a groove on the back of the heart that separates the myocardial ischemia or injury to death (ie., necrosis) of the
left atrium from the left ventricle called the coronary sulcus heart muscle. 1he usual cause of an ACS is the rupture of an
(see Fig. 1.17). 1he Cx supplies blood to the following: atherosclerotic plaque. Arleriosderosis is a cb.ronic disease ofthe
• The left atrium arterial system characterized by abnormal thickening and hard-
• Part of the lateral surface of the left ventricle ening of the vessel walls. Atherosclerosis is a form of arterioscle-
• 1he inferior surface of the left ventricle in about 1596 of rosis in which the thickening and hardening of the vessel walls
individuals are caused by a buildup of fat-like deposits (e.g., plaque) in the
• The posterior surface of the left ventricle in 15% inner lining oflarge and middle-sized muscular arteries. As the
• 1he SA node in about 40% fatty deposits build up, the opening ofthe artery slowly narrows,
• TheAVbundle in 10% to 15% and blood flow to the muscle decreases (Fig. 1.18).
Another random document with
no related content on Scribd:
DANCE ON STILTS AT THE GIRLS’ UNYAGO, NIUCHI
I see increasing reason to believe that the view formed some time
back as to the origin of the Makonde bush is the correct one. I have
no doubt that it is not a natural product, but the result of human
occupation. Those parts of the high country where man—as a very
slight amount of practice enables the eye to perceive at once—has not
yet penetrated with axe and hoe, are still occupied by a splendid
timber forest quite able to sustain a comparison with our mixed
forests in Germany. But wherever man has once built his hut or tilled
his field, this horrible bush springs up. Every phase of this process
may be seen in the course of a couple of hours’ walk along the main
road. From the bush to right or left, one hears the sound of the axe—
not from one spot only, but from several directions at once. A few
steps further on, we can see what is taking place. The brush has been
cut down and piled up in heaps to the height of a yard or more,
between which the trunks of the large trees stand up like the last
pillars of a magnificent ruined building. These, too, present a
melancholy spectacle: the destructive Makonde have ringed them—
cut a broad strip of bark all round to ensure their dying off—and also
piled up pyramids of brush round them. Father and son, mother and
son-in-law, are chopping away perseveringly in the background—too
busy, almost, to look round at the white stranger, who usually excites
so much interest. If you pass by the same place a week later, the piles
of brushwood have disappeared and a thick layer of ashes has taken
the place of the green forest. The large trees stretch their
smouldering trunks and branches in dumb accusation to heaven—if
they have not already fallen and been more or less reduced to ashes,
perhaps only showing as a white stripe on the dark ground.
This work of destruction is carried out by the Makonde alike on the
virgin forest and on the bush which has sprung up on sites already
cultivated and deserted. In the second case they are saved the trouble
of burning the large trees, these being entirely absent in the
secondary bush.
After burning this piece of forest ground and loosening it with the
hoe, the native sows his corn and plants his vegetables. All over the
country, he goes in for bed-culture, which requires, and, in fact,
receives, the most careful attention. Weeds are nowhere tolerated in
the south of German East Africa. The crops may fail on the plains,
where droughts are frequent, but never on the plateau with its
abundant rains and heavy dews. Its fortunate inhabitants even have
the satisfaction of seeing the proud Wayao and Wamakua working
for them as labourers, driven by hunger to serve where they were
accustomed to rule.
But the light, sandy soil is soon exhausted, and would yield no
harvest the second year if cultivated twice running. This fact has
been familiar to the native for ages; consequently he provides in
time, and, while his crop is growing, prepares the next plot with axe
and firebrand. Next year he plants this with his various crops and
lets the first piece lie fallow. For a short time it remains waste and
desolate; then nature steps in to repair the destruction wrought by
man; a thousand new growths spring out of the exhausted soil, and
even the old stumps put forth fresh shoots. Next year the new growth
is up to one’s knees, and in a few years more it is that terrible,
impenetrable bush, which maintains its position till the black
occupier of the land has made the round of all the available sites and
come back to his starting point.
The Makonde are, body and soul, so to speak, one with this bush.
According to my Yao informants, indeed, their name means nothing
else but “bush people.” Their own tradition says that they have been
settled up here for a very long time, but to my surprise they laid great
stress on an original immigration. Their old homes were in the
south-east, near Mikindani and the mouth of the Rovuma, whence
their peaceful forefathers were driven by the continual raids of the
Sakalavas from Madagascar and the warlike Shirazis[47] of the coast,
to take refuge on the almost inaccessible plateau. I have studied
African ethnology for twenty years, but the fact that changes of
population in this apparently quiet and peaceable corner of the earth
could have been occasioned by outside enterprises taking place on
the high seas, was completely new to me. It is, no doubt, however,
correct.
The charming tribal legend of the Makonde—besides informing us
of other interesting matters—explains why they have to live in the
thickest of the bush and a long way from the edge of the plateau,
instead of making their permanent homes beside the purling brooks
and springs of the low country.
“The place where the tribe originated is Mahuta, on the southern
side of the plateau towards the Rovuma, where of old time there was
nothing but thick bush. Out of this bush came a man who never
washed himself or shaved his head, and who ate and drank but little.
He went out and made a human figure from the wood of a tree
growing in the open country, which he took home to his abode in the
bush and there set it upright. In the night this image came to life and
was a woman. The man and woman went down together to the
Rovuma to wash themselves. Here the woman gave birth to a still-
born child. They left that place and passed over the high land into the
valley of the Mbemkuru, where the woman had another child, which
was also born dead. Then they returned to the high bush country of
Mahuta, where the third child was born, which lived and grew up. In
course of time, the couple had many more children, and called
themselves Wamatanda. These were the ancestral stock of the
Makonde, also called Wamakonde,[48] i.e., aborigines. Their
forefather, the man from the bush, gave his children the command to
bury their dead upright, in memory of the mother of their race who
was cut out of wood and awoke to life when standing upright. He also
warned them against settling in the valleys and near large streams,
for sickness and death dwelt there. They were to make it a rule to
have their huts at least an hour’s walk from the nearest watering-
place; then their children would thrive and escape illness.”
The explanation of the name Makonde given by my informants is
somewhat different from that contained in the above legend, which I
extract from a little book (small, but packed with information), by
Pater Adams, entitled Lindi und sein Hinterland. Otherwise, my
results agree exactly with the statements of the legend. Washing?
Hapana—there is no such thing. Why should they do so? As it is, the
supply of water scarcely suffices for cooking and drinking; other
people do not wash, so why should the Makonde distinguish himself
by such needless eccentricity? As for shaving the head, the short,
woolly crop scarcely needs it,[49] so the second ancestral precept is
likewise easy enough to follow. Beyond this, however, there is
nothing ridiculous in the ancestor’s advice. I have obtained from
various local artists a fairly large number of figures carved in wood,
ranging from fifteen to twenty-three inches in height, and
representing women belonging to the great group of the Mavia,
Makonde, and Matambwe tribes. The carving is remarkably well
done and renders the female type with great accuracy, especially the
keloid ornamentation, to be described later on. As to the object and
meaning of their works the sculptors either could or (more probably)
would tell me nothing, and I was forced to content myself with the
scanty information vouchsafed by one man, who said that the figures
were merely intended to represent the nembo—the artificial
deformations of pelele, ear-discs, and keloids. The legend recorded
by Pater Adams places these figures in a new light. They must surely
be more than mere dolls; and we may even venture to assume that
they are—though the majority of present-day Makonde are probably
unaware of the fact—representations of the tribal ancestress.
The references in the legend to the descent from Mahuta to the
Rovuma, and to a journey across the highlands into the Mbekuru
valley, undoubtedly indicate the previous history of the tribe, the
travels of the ancestral pair typifying the migrations of their
descendants. The descent to the neighbouring Rovuma valley, with
its extraordinary fertility and great abundance of game, is intelligible
at a glance—but the crossing of the Lukuledi depression, the ascent
to the Rondo Plateau and the descent to the Mbemkuru, also lie
within the bounds of probability, for all these districts have exactly
the same character as the extreme south. Now, however, comes a
point of especial interest for our bacteriological age. The primitive
Makonde did not enjoy their lives in the marshy river-valleys.
Disease raged among them, and many died. It was only after they
had returned to their original home near Mahuta, that the health
conditions of these people improved. We are very apt to think of the
African as a stupid person whose ignorance of nature is only equalled
by his fear of it, and who looks on all mishaps as caused by evil
spirits and malignant natural powers. It is much more correct to
assume in this case that the people very early learnt to distinguish
districts infested with malaria from those where it is absent.
This knowledge is crystallized in the
ancestral warning against settling in the
valleys and near the great waters, the
dwelling-places of disease and death. At the
same time, for security against the hostile
Mavia south of the Rovuma, it was enacted
that every settlement must be not less than a
certain distance from the southern edge of the
plateau. Such in fact is their mode of life at the
present day. It is not such a bad one, and
certainly they are both safer and more
comfortable than the Makua, the recent
intruders from the south, who have made USUAL METHOD OF
good their footing on the western edge of the CLOSING HUT-DOOR
plateau, extending over a fairly wide belt of
country. Neither Makua nor Makonde show in their dwellings
anything of the size and comeliness of the Yao houses in the plain,
especially at Masasi, Chingulungulu and Zuza’s. Jumbe Chauro, a
Makonde hamlet not far from Newala, on the road to Mahuta, is the
most important settlement of the tribe I have yet seen, and has fairly
spacious huts. But how slovenly is their construction compared with
the palatial residences of the elephant-hunters living in the plain.
The roofs are still more untidy than in the general run of huts during
the dry season, the walls show here and there the scanty beginnings
or the lamentable remains of the mud plastering, and the interior is a
veritable dog-kennel; dirt, dust and disorder everywhere. A few huts
only show any attempt at division into rooms, and this consists
merely of very roughly-made bamboo partitions. In one point alone
have I noticed any indication of progress—in the method of fastening
the door. Houses all over the south are secured in a simple but
ingenious manner. The door consists of a set of stout pieces of wood
or bamboo, tied with bark-string to two cross-pieces, and moving in
two grooves round one of the door-posts, so as to open inwards. If
the owner wishes to leave home, he takes two logs as thick as a man’s
upper arm and about a yard long. One of these is placed obliquely
against the middle of the door from the inside, so as to form an angle
of from 60° to 75° with the ground. He then places the second piece
horizontally across the first, pressing it downward with all his might.
It is kept in place by two strong posts planted in the ground a few
inches inside the door. This fastening is absolutely safe, but of course
cannot be applied to both doors at once, otherwise how could the
owner leave or enter his house? I have not yet succeeded in finding
out how the back door is fastened.