This document discusses various placental abnormalities and complications of pregnancy and childbirth. It defines different types of placental abnormalities such as bilobed placentas, placenta previa, and placental hematomas. It also describes complications related to the umbilical cord such as cord prolapse and vasoconstriction. Finally, it discusses postpartum infections and complications that can occur in the days following delivery.
Observations on Abortion: Containing an account of the manner in which it is accomplished, the causes which produced it, and the method of preventing or treating it
This document discusses various placental abnormalities and complications of pregnancy and childbirth. It defines different types of placental abnormalities such as bilobed placentas, placenta previa, and placental hematomas. It also describes complications related to the umbilical cord such as cord prolapse and vasoconstriction. Finally, it discusses postpartum infections and complications that can occur in the days following delivery.
This document discusses various placental abnormalities and complications of pregnancy and childbirth. It defines different types of placental abnormalities such as bilobed placentas, placenta previa, and placental hematomas. It also describes complications related to the umbilical cord such as cord prolapse and vasoconstriction. Finally, it discusses postpartum infections and complications that can occur in the days following delivery.
This document discusses various placental abnormalities and complications of pregnancy and childbirth. It defines different types of placental abnormalities such as bilobed placentas, placenta previa, and placental hematomas. It also describes complications related to the umbilical cord such as cord prolapse and vasoconstriction. Finally, it discusses postpartum infections and complications that can occur in the days following delivery.
1. cord inserts b/n 2 placental lobes – either deposition into a connecting chorionic bridge or - -layer of fibrin diminishes O2 to villus intervening membranes? 1:350 deliveries - ---syncytiotrophoblast necrosis a. Other names? 11. Types of Hematoma 2. Smaller version of bilobate placenta? a. b/n placenta & its adjacent decidua? - 2fold higher incidence in twin b. b/n chorion & decidua aka pregnancies “subchorionic hemorrhage”? - vascular connections of fetal origin c. along roof of intervillous space & - -if retained in the uterus after delivery beneath chorionic plate? Aka - --- HEMORRHAGE “Breus mole” 3. All or large part of fetal membranes are d. b/n placenta & amnion; of fetal covered by functioning villi? -may give vessel origin? rise to serious hemorrhage because of - acute events during 3rd stage labor when associated placenta previa or accreta cord traction ruptures a vessel near cord 4. Placenta is annular in shape? insertion - variant of placenta membranacea - chronic lesions, asstd. w/ fetomaternal - tissue atrophy in a portion of the ring, a hemorrhage or fetal growth restriction horseshoe shape is more common - confused with chorioangioma - antepartum and postpartum bleeding and 12. Calcium salts may be deposited IUGR throughout placenta but are most 5. Central portion of a discoidal placenta is common on maternal surface in basal missing? plate - actual hole in the placenta, more often - Asstd. w/ nulliparity, higher defect involves only villous tissue with socioeconomic status & greater maternal chorionic plate intact serum calcium level 6. -when the chorionic plate is smaller than - Seen in sonography the basal plate --- placental periphery is 13. Striking enlargement of chorionic uncovered? villi commonly seen in severe 7. -fetal surface presents a central erythroblastosis & fetal hydrops depression surrounded by - maternal diabetes, fetal congestive - a thickened, grayish-white ring - heart failure & maternal-fetal syphilis - -ring is composed of a double fold of 14. Resemblance of their components amnion and chorion, with degenerated to the blood vessels & stroma of the decidua and fibrin in between chorionic villus? 8. -when the ring does not have the central - Only BENIGN tumors of the placenta depression with the fold of membranes? - Well-circumscribed , rounded, - less well defined adverse clinical predominantly hypoechoic lesion near outcomes chorionic surface & protruding into 9. -deposits dense fibrinoid layer on amnionic cavity placental basal plate? - > 5 cm, asstd w/ significant AV - Blockade to normal maternal blood flow shunting w/in placenta leading fetal 10. -small yellow white nodules w/in anemia & hydrops placenta are normal part of placental aging? 15.4 most common metastatic cancers in the placenta? M,L,L,Bc 16. What type of cancer can metastasize to the 28. –marked focal dilatation that develop w/in fetus? intra-amnionic part of the umb. vein w/in its fetal intra-abd’l portion? 17. Inflammation of the fetal membranes? 29. rare congt’l thinning of vessel wall w/ a. -leukocytes in amnionic fluid? diminished support fr Wharton jelly? - most form at or near cord insertion into the b. -umbilical cord? Placenta 18. a condition in which the umbilical cord is asstd w/ single umb. artery, trisomy 18, fetal inserted at or near the placental margin rather growth restriction & stillbirth than in the center? 30. Good contractions, frequency? 31. After the expulsion of the baby and 19. insertion is normal but umbilical vessels placenta and membranes, what stage of labor? lose their protective Wharton jelly shortly 32. How many cotyledons of the placenta? before insertion? 33. Causes uterine contraction and peripheral - covered only by amnion & prone to vasodilation? --> hypotension and tachy compression, twisting & thrombosis 34. Not given to women with a history of 20. umbilical vessels spread w/ in membranes bronchial asthma? at a distance from the placental margin, which 35. Cautiously given to women with they reach surrounded only by a fold of amnion hypertension? –- vessels vulnerable to compression –- fetal 36. Keeps the fundus low? anoxia? 37. A physiologic pain experienced during 21. placental vessels overlie the cervix, lie b/n postpartum in the uterus (lower abdomen) cervix & presenting fetal part, & supported only when the baby suckles? by membranes? 1:5200 pregnancies 38. From expulsion of membranes up to 1 or 2 22. knobs protruding from cord surface hours post-partum, what stage? -focal redundancies of a vessel or Wharton jelly 39. From delivery of the membrane up to 6 -w/ no clinical significance weeks post-partum? 23. active fetal movements, -high in 40. Rarely exceeds 39celsius and usually lasts monoamnionic twins, risk of stillbirth increased <24 hours? 5-10fold, in live fetuses, increased FHR 41. What are the three complications of uterine abnormalities but cord blood acid base values and pelvic infections? usually normal? 42. A common cause of persisitent fever in 24. UC may be the presenting part in labor & women treated for metritis? asstd w/ fetal malpresentation; cord prolapse or a) Begins at what day? FHR abnormalities is an asstd labor finding? 43. A serious complication, bowel evisceration 25. focal narrowing of cord diameter that can be a morbid, surgical emergency, presents develops in area of fetal umbilical insertion ? w/in 7-10 post-op days, high mortality risk, 26. -Found along the course of the cord; usually caused by infections? a. epithelium –lined remnants of the allantois; 44. Uncommon severe wound infection co-exist w/ persistently patent urachus? a) Risk factors? b. form fr local degeneration of Wharton jelly; b) Symptoms noted until 3-5 days after asstd w/ structural & chromosomal defects delivery (trisomy 18 & 13 )? 45. Organs outside the uterus such as fallopian c. found in1st tri tend to resolve completely? tube and ovary located bilaterally? d. may portrend miscarriage & aneuploidy? 46. Intense parametrial cellulitis forming a 27. have lower perinatal morbidity & phlegmon within the leaves of the broad mortality than those in the artery? ligament, fever persist longer than 72 hours despite IV antimicrobial therapy, fever resolves in 5-7 days? 47. Frequent complication in the pre-antibiotic era, septic embolization was common, ovarian veins may become involved? a) Chills, occasional lower quadrant pain, usually asymptomatic 48. An acute febrile illness w/ severe multisystem derangement, 10-15% case- fatality rate? a) Caused maternal mortality 49. Post-partum incidence of mastitis? a) Suppurative mastitis seldom appear before the end of the 1st week postpartum seen usually at 3rd or 4th week, unilateral b) Engorgement first followed by inflammation c) Sonography (diagnostics) 50. Immediate source of mastitis-causing organisms is almost always caused by? a) Resolves within 48hours b) Dicloxacillin 500mg 4x a day c) Erythromycin d) Vigorous milk expression maybe be a sufficient treatment alone e) Continue breastfeeding
Observations on Abortion: Containing an account of the manner in which it is accomplished, the causes which produced it, and the method of preventing or treating it