Report 1702735488541

Mrs.
M SRILAKSHMI Lab ID : 31201802395

DOB : Collected : 16-12-2023 06:35
Age : 37 Years Received : 16-12-2023 11:26
Gender : Female Reported : 16-12-2023 13:30
CRM : 223001827162 Status : Interim
Location : HYDERABAD Client : Novocura Tech Health Services Pvt Ltd Hyderabad -
BS9110
Ref DOC : SELF
Sample Quality : Adequate
Parameter Result Unit Biological Ref. Interval Method

COMPLETE BLOOD COUNT (CBC), Whole Blood EDTA
Hemoglobin 11.1 g/dL 12.0 - 15.0 Colorimetric method

Red Blood Cells 4.01 10^6 Cells/µL 3.8 - 4.8 Electrical Impedance method
PCV (Hematocrit) 34.30 % 36 - 46 Calculated
MCV(Mean Corpuscular Volume) 85.6 fL 83 - 101 Calculated
MCH (Mean Corpuscular Hb) 27.6 Pg 27 - 32 Calculated
MCHC (Mean Corpuscular Hb Concentration) 32.2 g/dL 31.5 - 34.5 Calculated
31201802395-Mrs. M SRILAKSHMI-37 Years-Female
Red Cell Distribution Width CV 14.70 % 11.6 - 14.6 Calculated

Red Cell Distribution Width SD 46.10 fL 39 -46 Calculated
WBC -Total Leucocytes Count 6.15 10^3 Cells/µL 4- 10 Flowcytometry
Neutrophils 73.6 % 40 - 80 Flowcytometry
Lymphocytes 22.2 % 20 - 40 Flowcytometry
Monocytes 3.80 % 2-10 Flowcytometry
Eosinophils 0.4 % 1-6 Flowcytometry
Basophils 0.00 % 0-2 Flowcytometry
Neutrophils (Abs) 4.53 10^3 Cells/µL 1.5 - 8.0 Flowcytometry
Lymphocytes (Abs) 1.37 10^3 Cells/µL 1.0 - 4.8 Flowcytometry
Monocytes (Abs) 0.23 10^3 Cells/µL 0.5 - 0.9 Flowcytometry
Eosinophils (Abs) 0.02 10^3 Cells/µL 0.2 - 0.5 Flowcytometry
Basophils (Abs) 0.00 10^3 Cells/µL 0.0 - 0.3 Flowcytometry
Platelet Count 241.00 10^3/µL 150-410 Electrical Impedance method
MPV 10.1 fL 9 - 13 Calculated
PDW 15.4 fL 10.0 - 17.9 Calculated
PlateletCrit 0.24 % 0.22 - 0.44 Calculated
PLCR (Platelet-Large Cell Ratio) 27.80 % 15.0 - 35.0 Calculated
Clinical significance:
CBC is used as a screening tool in the diagnosis or monitoring of many diseases. RBCs, WBCs, and platelets are produced in the bone marrow and released into the peripheral blood.
The primary function of the RBC is to deliver oxygen to tissues. WBCs are key components of the immune system. Platelets play a vital role in blood clotting.Abnormal cell counter
results are confirmed by peripheral blood smear examination by trained pathologist.
Processed At: LifeCell International Pvt Ltd.1-8-303, Sardar Patel Road, Sindhi Colony, Begumpet, Hyderabad, Telangana 500003
This is an Electronically Authenticated Report.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI
DR.JYOTHI MD, Pathology MC-5148

Page 1 of 14 Lab Director
Mrs. M SRILAKSHMI Lab ID : 31201802395
DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF
ESR (Erythrocyte Sedimentation Rate), EDTA 07 mm/hr <=12 Westergren(Manual)

Blood
Clinical significance :-
ESR is the measurement of sedimentation of red cells in diluted blood after standing for 1 hour. It is dependent on various physiologic and pathologic factors including hemoglobin
concentration, ratio of plasma proteins, serum lipid concentration etc. Although ESR is a non-specific phenomenon, its measurement is useful in disorders associated with increased
production of acute phase proteins. In RA & TB it provides an index of progess of the disease and it has considerable value in diagnosis of temporal arteritis & polymyalgia
rheumatica. ESR can be low ( 0-1 mm) especially in polycythemia, hypofibrinogenaemia and in abdnormalities of red cells like sickle cells or speherocytosis etc.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF
Test Result Unit Biological Ref Intervals Method
Glucose (Fasting) Plasma 101.60 mg/dL Normal: <100 GOD-POD

Pre-Diabetic: 100-124
Diabetic =>125
HBA1C by HPLC
HbA1c By HPLC,EDTA Blood 4.80 % NORMAL: 4.5-5.6 HPLC

AT RISK : 5.7-6.5
DIABETIC: 6.6-7.0
UNCONTROLLED: 7.1-
8.9
Critically high: >= 9.0
Estimated Average Glucose(eAG) 90.76 mg/dL 70-126 Calculated
Clinical significance :
Hemoglobin A1c (HbA1c) is a result of the nonenzymatic attachment of a hexose molecule to the N-terminal amino acid of the hemoglobin molecule. HbA1c estimation is useful in
evaluating the long-term control of blood glucose concentrations in patients with diabetes, for diagnosing diabetes and to identify patients at increased risk for diabetes (prediabetes).
The ADA recommends measurement of periodic HbA1c measurements to keep the same within the target range.The presence of hemoglobin variants can interfere with the
measurement of hemoglobin A1c (HbA1c).
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF
Amylase, Serum 38.00 U/L 28 - 100 CNP-G3
Clinical Significance
Amylase is produced primarily by the exocrine pancreas and salivary glands, small intestine mucosa, ovaries, placenta, liver, and fallopian tubes. Pancreatic and salivary
isoenzymes are found in serum. In acute pancreatitis, a transient rise in serum amylase activity occurs within 2 to 12 hours of onset; levels return to normal by the third or fourth
day. In acute pancreatitis associated with hyperlipidemia, serum amylase activity may be spuriously normal. Elevated levels also may be seen in a number of nonpancreatic
disease processes including mumps, salivary duct obstruction, ectopic pregnancy, and intestinal obstruction/infarction.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF
High Sensitive CRP (hs-CRP), Serum 17.6 mg/L Risk Stratification for CVD CLIA
Low risk: less than 1.0 mg/L
Average risk: 1.0 to 3.0 mg/L
High risk: above 3.0 mg/L
Clinical significance :
The hs-CRP test accurately measures low levels of CRP to identify low but persistent levels of inflammation and thus helps
predict a person’s risk of developing CVD. Relatively high levels of hs-CRP in otherwise healthy individuals have been found
to be predictive of an increased risk of a future heart attack, stroke, sudden cardiac death, and/or peripheral arterial disease,
even when cholesterol levels are within an acceptable range.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI
DR.JYOTHI MD, Pathology

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF
Phosphorous, Serum 3.22 mg/dL 2.5 - 4.5 Ammonium Molybdate UV
Clinical significance:-
Phosphorus occurs in blood in the form of inorganic phosphate and organically bound phosphoric acid. Serum phosphate concentrations are dependent on meals and variation in
the secretion of hormones such as parathyroid hormone (PTH) and may vary widely. Hyperphosphatemia is usually secondary to an inability of the kidneys to excrete phosphate.
Hypophosphatemia is relatively common in hospitalized patients. Levels below 1.5 mg/dL may result in muscle weakness, hemolysis of red cells, coma, and bone deformity and
impaired growth.
Magnesium, Serum 1.89 mg/dL 1.5-2.5 Xylidyl Blue

Magnesium, along with potassium, is a major intracellular cation. Hypermagnesemia is found in acute and chronic renal failure, magnesium overload, and magnesium release from
the intracellular space. Mild-to-moderate hypermagnesemia may prolong atrioventricular conduction time. Magnesium toxicity may result in central nervous system (CNS)
depression, cardiac arrest, and respiratory arrest. Conditions that have been associated with hypomagnesemia include chronic alcoholism, childhood malnutrition, lactation,
malabsorption, acute pancreatitis, hypothyroidism, chronic glomerulonephritis, aldosteronism, and prolonged intravenous feeding.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

Lipid Profile
Total Cholesterol, Serum 140.00 mg/dL Desirable: <200 CHOD-PAP
Borderline: 200 - 239
High: >=240
Triglycerides, Serum 70.20 mg/dL Normal: <150 GPO
High:150-199
Hypertriglyceridemia: 200-499
Very high: >499
HDL Cholesterol, Serum 52.30 mg/dL Low : < 40 Precipitation Method with
High : > 60 PVS and PEGME
Low Density Lipoprotein-Cholesterol (LDL) 73.66 mg/dL Optimal: <100 Calculated

Near Optimal: 100-129
Borderline High: 130-159
High: 160-189
Very High: >189
VLDL 14.04 mg/dL 6-40 Calculated
Total Cholesterol/HDL Ratio 2.68 Optimal: <3.5 Calculated
Near Optimal: 3.5 - 5.0
High: >5
LDL / HDL Ratio 1.41 % Optimal: <2.5 Calculated
Near optimal: 2.5 - 3.5
High: >3.5
Non HDL Cholesterol, Serum 87.70 mg/dL Desirable < 130 Calculated
Borderline High 130-159
High 160-189
Very High: >=190
A complete cholesterol test — also called a lipid panel or lipid profile — is a blood test that can measure the amount of cholesterol and triglycerides in your blood.
A cholesterol test can help determine your risk of the buildup of fatty deposits (plaques) in your arteries that can lead to narrowed or blocked arteries throughout your
body (atherosclerosis).A cholesterol test is an important tool. High levels of lipids (fats) in the blood, including cholesterol and triglycerides, is also called
"hyperlipidemia." Hyperlipidemia can significantly increase a person's risk of heart attacks, strokes, and other serious problems due to vessel wall narrowing or
obstruction.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

RENAL PROFILE
Creatinine, Serum 0.64 mg/dL 0.6 - 1.1 ENZYMATIC
eGFR 127 ml/min/1.73m^2 Normal > 90 Calculated
Mild decrease in GFR : 60-90
Moderate decrease in GFR :
30-59
Severe decrease in GFR : 15-
29
Kidney Failure: < 15
Urea, Serum 14.50 mg/dL 15-48 UREASE-GLDH
Blood Urea Nitrogen (BUN), Serum 6.78 mg/dL 6 -20 Calculated

BUN/Creatinine Ratio, Serum 10.59 % 5.0 - 23.5 Calculated method
Uric Acid, Serum 3.10 mg/dL 2.3-6.6 URICASE-POD
Calcium, Serum 9.20 mg/dL 8.6 - 10.2 Arsenazo Method
Kidney function tests are a reliable way of testing the kidneys, but it is important to remember that they can also change dramatically with illness or dehydration. This panel could be
ordered when a patient has risk factors for kidney dysfunction such as high blood pressure (hypertension), diabetes, cardiovascular disease, obesity, elevated cholesterol, or a family
history of kidney disease. This panel may also be ordered when someone has signs and symptoms of kidney disease, though early kidney disease often does not cause any noticeable
symptoms. It may be initially detected through routine blood or urine testing.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

LIVER FUNCTION TEST
Bilirubin - Total, Serum 0.24 mg/dL 0.1 - 1.3 Modified TAB Method
Bilirubin - Direct, Serum 0.12 mg/dL < 0.3 DIAZO
Bilirubin - Indirect, Serum 0.12 mg/dL 0.2-1 Calculated
SGOT, Serum 14.60 U/L <31 IFCC without PLP
SGPT,Serum 14.00 U/L <35 IFCC without PLP
Alkaline Phosphatase, Serum 74.0 U/L 42 - 98 AMP
GGT (Gamma Glutamyl Transferase), 24.70 U/L <38 G-glutamyl-p-nitroanilide
Serum
Total Protein, Serum 6.06 gm/dL 6.4-8.8 BIURET
Albumin, Serum 3.61 gm/dL 3.5 - 5.2 BCG
Globulin, Serum 2.45 gm/dL 1.9-3.9 Calculated
A:G ratio 1.47 1.1 - 2.5 Calculated
Liver function tests measure how well the liver is performing its normal functions of producing protein and clearing bilirubin, a blood waste product. Other liver function tests
measure enzymes that liver cells release in response to damage or disease.The hepatic function panel may be used to help diagnose liver disease if a person has signs and symptoms
that indicate possible liver dysfunction. If a person has a known condition or liver disease, testing may be performed at intervals to monitor the health of the liver and to evaluate the
effectiveness of any treatments. Abnormal tests.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

THYROID FUNCTION TEST
Tri Iodo Thyronine (T3 Total), Serum 0.95 ng/mL Non Pregnant: 0.7-2.04 CLIA
Pregnancy:
1st trimester: 0.81-1.9
2nd trimester: 1.0-2.60
3rd trimester: 1.0-2.60
Triiodothyronine (T3) values above 200 ng/dL in adults or over age related cutoffs in children are consistent with hyperthyroidism or increased thyroid hormone-binding proteins.
Abnormal levels (high or low) of thyroid hormone-binding proteins (primarily albumin and thyroid-binding globulin) may cause abnormal T3 concentrations in euthyroid patients.
Please note that Triiodothyronine (T3) is not a reliable marker for hypothyroidism. Therapy with amiodarone can lead to depressed T3 values.
Thyroxine (T4), Serum 8.66 µg/dL 5.5-11.0 CLIA
Thyroxine (T4) is synthesized in the thyroid gland. High T4 are seen in hyperthyroidism and in patients with acute thyroiditis. Low T4 are seen in hypothyroidism, myxedema,
cretinism, chronic thyroiditis, and occasionally, subacute thyroiditis. Increased total thyroxine (T4) is seen in pregnancy and patients who are on estrogen medication. These patients
have increased total T4 levels due to increased thyroxine-binding globulin (TBG) levels. Decreased total T4 is seen in patients on treatment with anabolic steroids or nephrosis
(decreased TBG levels).
Thyroid - Thyroid Stimulating Hormone (TSH), 2.321 µIU/mL Nonpregnant: 0.4-5.5 CLIA
Serum Pregnancy:
First Trimester: 0.3-4.5
Second Trimester: 0.5-4.6
Third trimester: 0.8-5.2
In primary hypothyroidism, TSH (thyroid-stimulating hormone) levels will be elevated. In primary hyperthyroidism, TSH levels will be low. TSH estimation is especially useful in the
differential diagnosis of primary (thyroid) from secondary (pituitary) and tertiary (hypothalamus) hypothyroidism. In primary hypothyroidism, TSH levels are significantly elevated,
while in secondary and tertiary hypothyroidism, TSH levels are low or normal. Elevated or low TSH in the context of normal free thyroxine is often referred to as subclinical hypo- or
hyperthyroidism, respectively.
Pregnancy American Thyroid American European Thyroid society
Association Endocrine Association
1st trimester < 2.5 < 2.5 < 2.5
2nd trimester < 3.0 < 3.0 < 3.0
3rd trimester < 3.5 < 3.0 < 3.0
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

ELECTROLYTES
Sodium (Na+), Serum 141.10 mmol/L 136-145 Direct ISE
Sodium is the primary extracellular cation. Hypernatremia (high sodium) is often attributable to excessive loss of sodium-poor body fluids. Hypernatremia is often associated with
hypercalcemia and hypokalemia and is seen in liver disease, cardiac failure, pregnancy, burns, and osmotic diuresis. Hypernatremia occurs in dehydration, increased renal sodium
conservation in hyperaldosteronism, Cushing syndrome, and diabetic acidosis. Severe hypernatremia may be associated with volume contraction, lactic acidosis, and increased
hematocrit.
Potassium (K+), Serum 4.14 mmol/L 3.5 - 5.1 Direct ISE
Potassium is the major cation of the intracellular fluid. Disturbance of potassium homeostasis has serious consequences. Decreases in extracellular potassium are characterized by
muscle weakness, irritability, and eventual paralysis. Hypokalemia (low potassium) is common in vomiting, diarrhea, alcoholism, and folic acid deficiency. Hyperkalemia may be seen
in end-stage renal failure, hemolysis, trauma, Addison disease, metabolic acidosis, acute starvation, dehydration, and with rapid potassium infusion.
Chloride, Serum 105.70 mmol/L 96-106 Direct ISE

Clinical Significance:
Chloride is the major anion in the extracellular water space. Chloride is increased in dehydration, renal tubular acidosis (hyperchloremia metabolic acidosis), acute renal
failure, metabolic acidosis associated with prolonged diarrhea and loss of sodium bicarbonate, diabetes insipidus, adrenocortical hyperfunction, salicylate intoxication,
and with excessive infusion of isotonic saline or extremely high dietary intake of salt. Hyperchloremia acidosis may be a sign of severe renal tubular pathology.Chloride is
decreased in overhydration, chronic respiratory acidosis, salt-losing nephritis, metabolic alkalosis, congestive heart failure.
Bicarbonate, Serum 27.21 mmol/L 22-26 Direct ISE
Bicarbonate is the second largest fraction of the anions in plasma. The bicarbonate content of serum or plasma is a significant indicator of electrolyte
dispersion and anion deficit. Together with pH determination, bicarbonate measurements are used in the diagnosis and treatment of numerous potentially
serious disorders associated with acid-base imbalance in the respiratory and metabolic systems. Some of these conditions are diarrhea, renal tubular
acidosis, carbonic anhydrase inhibitors, hyperkalemic acidosis, renal failure, and ketoacidosis.Alterations of bicarbonate (HCO3) and carbon dioxide (CO2)
dissolved in plasma are characteristic of acid-base imbalance.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

IRON STUDIES
Iron, Serum 16.80 µg/dL 35-145 Chromogen ferrozine
UIBC, Serum 207.20 ug/dL 112 - 346 Ferrozine
Total Iron Binding Capacity (TIBC), Serum 224 ug/dL 250-400 Calculated
% OF IRON SATURATION 8 % Calculated
Clinical Significance: -
Serum iron can be decreased in conditions like iron deficiency anemia and in inflammatory disorders (acute infection,
immunization, and myocardial infarction), Hemorrhage etc.

Increased serum iron can be seen in conditions like hemochromatosis, hemolytic anemia, hepatitis, Iron poisoning and
Frequent blood transfusions.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF
Vitamin D - 25-Hydroxy, Serum 17.39 ng/mL <10: Severe deficiency CLIA

10-19: Mild to moderate
deficiency
20-50: Optimum level
51-80: Increased risk of
hypercalciuria
>80: Toxicity possible
A low blood level of 25-hydroxyvitamin D may mean that a person is not getting enough exposure to sunlight or enough dietary vitamin D to meet his or her body’s demand or that
there is a problem with its absorption from the intestines. Occasionally, drugs used to treat seizures, particularly phenytoin (Dilantin), can interfere with the production of 25-
hydroxyvitamin D in the liver. There is some evidence that vitamin D deficiency may increase the risk of some cancers, immune diseases, and cardiovascular disease. A high level of
25-hydroxyvitamin D usually reflects excess supplementation from vitamin pills or other nutritional supplements.
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI

DOB : Collected : 16-12-2023 06:35
BS9110
Ref DOC : SELF

URINE ROUTINE EXAMINATION
PHYSICAL EXAMINATION
Colour Pale Yellow Pale Yellow Visual
Volume 15 cc ml Visual
Specific Gravity 1.005 1.015 - 1.025 Dip Stick (Bromthymol blue)
Appearance Clear Clear Visual
pH 6.5 5.0 -8.0 Dip Stick (Double Indicators)
BIOCHEMICAL EXAMINATION
Protein, Urine Negative Negative Dip Stick (Protein Error of
Indicators)
Glucose Negative Negative Dip Stick (GOP-POD)
Ketones Absent Negative Dip Stick (Sodium
nitroprusside)
Urobilinogen Normal Normal Dip Stick (Ehrlich)
Bilirubin Negative Negative Dip Stick (Azo-coupling
reaction)
Nitrite Negative Negative Dip Stick (Diazotization)
Blood Negative Negative Dip Stick (Peroxidase)
MICROSCOPIC EXAMINATION
Pus cells 3-4 /hpf 0-5 Microscopy
Epithelial Cells 2-3 /hpf 0-2 Microscopy
RBCs Nil /hpf Nil Microscopy
Casts Nil Nil Microscopy
Crystals Nil Nil Microscopy
Yeast cells Absent Absent Microscopy
Bacteria Absent Absent Microscopy
A urinalysis alone usually doesn't provide a definite diagnosis. Depending on the reason your provider recommended this test, you might need follow-up for unusual results.
Evaluation of the urinalysis results with other tests can help your provider determine next steps.
Getting standard test results from a urinalysis doesn't guarantee that you're not ill. It might be too early to detect disease or your urine could be too diluted.
------------------ End Of Report ------------------
Mrs. M SRILAKSHMI
223001827162
Mrs. M SRILAKSHMI


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Mrs.

M SRILAKSHMI Lab ID : 31201802395

Parameter Result Unit Biological Ref. Interval Method

Hemoglobin 11.1 g/dL 12.0 - 15.0 Colorimetric method

Red Cell Distribution Width CV 14.70 % 11.6 - 14.6 Calculated

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

ESR (Erythrocyte Sedimentation Rate), EDTA 07 mm/hr <=12 Westergren(Manual)

DR.JYOTHI MD, Pathology MC-5148

Test Result Unit Biological Ref Intervals Method

Glucose (Fasting) Plasma 101.60 mg/dL Normal: <100 GOD-POD

HbA1c By HPLC,EDTA Blood 4.80 % NORMAL: 4.5-5.6 HPLC

Estimated Average Glucose(eAG) 90.76 mg/dL 70-126 Calculated

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

Amylase, Serum 38.00 U/L 28 - 100 CNP-G3

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

DR.JYOTHI MD, Pathology

Parameter Result Unit Biological Ref. Interval Method

Phosphorous, Serum 3.22 mg/dL 2.5 - 4.5 Ammonium Molybdate UV

Magnesium, Serum 1.89 mg/dL 1.5-2.5 Xylidyl Blue

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

Low Density Lipoprotein-Cholesterol (LDL) 73.66 mg/dL Optimal: <100 Calculated

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

Blood Urea Nitrogen (BUN), Serum 6.78 mg/dL 6 -20 Calculated

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

Thyroxine (T4), Serum 8.66 µg/dL 5.5-11.0 CLIA

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

Potassium (K+), Serum 4.14 mmol/L 3.5 - 5.1 Direct ISE

Chloride, Serum 105.70 mmol/L 96-106 Direct ISE

Bicarbonate, Serum 27.21 mmol/L 22-26 Direct ISE

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

immunization, and myocardial infarction), Hemorrhage etc.

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

Vitamin D - 25-Hydroxy, Serum 17.39 ng/mL <10: Severe deficiency CLIA

DR.JYOTHI MD, Pathology MC-5148

Parameter Result Unit Biological Ref. Interval Method

------------------ End Of Report ------------------

DR.JYOTHI MD, Pathology MC-5148

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