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Chronic Kidney Disease Sec To Pulmonary Edema, Sec To HKD
Chronic Kidney Disease Sec To Pulmonary Edema, Sec To HKD
Precipitating Factors
1. Genetics
1. Hypertension
2. Age
2. Obesity
3. Race
3. Diabetes
4. Family History
INJURY TO KIDNEY
INCREASED SERUM
FIBROSIS AND CANNOT REGULATE
POTASSIUM LEVELS
LEAD TO
SCARRING TO THE SERUM POTASSIUM
INCREASED
ALDOSTERONE
KIDNEYS PRODUCTION
S/S
1. SODIUM WASTE PRODUCTS INCREASE
CANNOT FILTER WASTE REDUCE KIDNEY
EDEMA RETURN TO THE ALDOSTERONE
RETENTION IN THE BODY FUNCTION
2. INCREASE CIRCULATION SECRETION
POTASSIUM
3. INCREASE
PHOSPHORUS
PULMONARY EDEMA/
4. DECREASE
PITTING EDEMA
CALCIUM
ACIDOSIS INCREASE SODIUM
DECLINE IN GFR ABSORPTION AND
INCREASE WATER
RETENTION
CONVERTS ANGIOTENSIN
TO ANGIOTENSIN 1 IN THE
DECREASE PERMANENT DAMAGE DEPRESS RENIN LIVER AND ANGIOTENSIN 2
INCREASE BLOOD
PRESSURE AND
INCREASE FLUID
VOLUME IN THE BODY
NO STIMULATION OF CHRONIC KIDNEY
PRODUCTION OF RBC DISEASE
IN THE BONE MARROW
HYPERTENSION
DECREASE
HEMOGLOBIN
ANEMIA
CARDIOGENIC SHOCK
DEATH
POOR PROGNOSIS