The document provides information on how to use an electrocardiogram (ECG/EKG) including how to prepare the patient, place the electrodes, and connect the device. It discusses nursing responsibilities before, during, and after the procedure. Key aspects of interpreting the ECG are summarized such as normal electrical conduction, identifying waves and intervals, and determining heart rate. Common cardiac rhythms like normal sinus rhythm, bradycardia, tachycardia, and premature atrial complexes are outlined.
The document provides information on how to use an electrocardiogram (ECG/EKG) including how to prepare the patient, place the electrodes, and connect the device. It discusses nursing responsibilities before, during, and after the procedure. Key aspects of interpreting the ECG are summarized such as normal electrical conduction, identifying waves and intervals, and determining heart rate. Common cardiac rhythms like normal sinus rhythm, bradycardia, tachycardia, and premature atrial complexes are outlined.
The document provides information on how to use an electrocardiogram (ECG/EKG) including how to prepare the patient, place the electrodes, and connect the device. It discusses nursing responsibilities before, during, and after the procedure. Key aspects of interpreting the ECG are summarized such as normal electrical conduction, identifying waves and intervals, and determining heart rate. Common cardiac rhythms like normal sinus rhythm, bradycardia, tachycardia, and premature atrial complexes are outlined.
• Detects the heart's electrical activity during myocardial
contraction and relaxation, usually recorded by electrodes on the skin. • ECG/EKG signals are recorded to examine heart rate, heart rate variability, analysis of the waveform morphology, arrhythmia, and other similar functions. NURSING RESPONSIBILITIES BEFORE THE PROCEDURE • Remove any metallic objects such as jewelry • Clip chest hair • Avoid drinking cold water right before test • No exercising, or increasing your heart rate, before the test • Keep the room at a moderate temperature to avoid shivering NURSING RESPONSIBILITIES DURING THE PROCEDURE • During the test, ask patient to lie still on the table and breathe normally. • Instruct patient to not talk during the test. HOW TO USE THE ECG 1. Prepare the skin. Prepare the subject's skin by wiping the chest area thoroughly with skin cleansing (alcohol) swabs. This removes any oil that may be on the patient's skin and which can cause drift in your ECG/EKG signals. HOW TO USE THE ECG 2. Find and mark the correct lead placements for the 12 lead ECG HOW TO USE THE ECG 3. Identify V1 and V2. • Identify the top of your subject's sternum. Approximately 4 centimeters below this, there is a ridge. This identifies the second intercostal space. Feeling down, you will come across the third and fourth intercostal spaces. Use a skin-safe marker to mark the fourth intercostal space as V2. • Mark V1 in the mirror position on the opposite side of the chest. HOW TO USE THE ECG 4. Find and mark V3 - V6. • V4 can be found one intercostal space lower than V2, in line with the middle of the clavicle. • Next, track along the torso to the subject's left to find V6 at mid auxiliary on the same horizontal level as V4. Mark V6. • V5 can be marked at midway between V4 and V6. • Mark V3 midway between V2 and V4. HOW TO USE THE ECG 5. Apply electrodes to the chest at V1 - V6 • Apply your electrodes to your 6 marks.
6. Connect wires from V1 to V6 to the recording device
HOW TO USE THE ECG 7. Apply limb leads. HOW TO USE THE ECG 8. Connect cable wires to one of the following: • An ECG machine placed at the patient’s side for an immediate recording (standard 12-lead ECG) • A cardiac monitor at the patient’s bedside for continuous reading; this kind of monitoring, usually called hardwire monitoring, is used in intensive care units • A small box that the patient carries that continuously transmits the ECG information by radiowaves to a central monitor located elsewhere (called telemetry) • A small, lightweight tape recorder–like machine (called ambulatory ECG monitoring or a Holter mon_x0002_itor) that the patient wears and that continuously records the ECG on a tape, which is later viewed and analyzed with a scanner HOW TO USE THE ECG HOW TO USE THE ECG HOW TO USE THE ECG HOW TO USE THE ECG HOW TO USE THE ECG • https://www.adinstruments.com/blog/correctly-place-electrodes-12- lead-ecg HOW TO USE THE ECG NORMAL ELECTRICAL CONDUCTION • Electrical impulse originates from SA node (near the superior vena cava in the right atrium) • Electrical impulse from SA node to AV node (CONDUCTION) → causes the atria to contract → ventricles filled with blood • Electrical impulse travels through Bundle of His and the Purkinje fibers (located in ventricular muscle) • Electrical stimulation causes mechanical contraction of ventricles (systole) • Cells repolarize and the ventricles relax (diastole) NORMAL ELECTRICAL CONDUCTION • DEPOLARIZATION • electrical stimulation • REPOLARIZATION • electrical relaxation • SYSTOLE • mechanical contraction • DIASTOLE • mechanical relaxation INTERPRETING THE ELECTROCARDIOGRAM WAVES, COMPLEXES, AND INTERVALS • P wave • represents atrial depolarization • N: 2.5 mm or less in height • N: 0.11 seconds or less in duration • QRS complex • ventricular depolarization • N: less than 0.12 seconds in duration • where atrial repolarization also occurs INTERPRETING THE ELECTROCARDIOGRAM WAVES, COMPLEXES, AND INTERVALS • QRS complex • Q wave • first negative deflection after the P wave • N: less than 0.04 seconds in duration • R wave • first positive deflection after the P wave • S wave • first negative deflection after the R wave INTERPRETING THE ELECTROCARDIOGRAM WAVES, COMPLEXES, AND INTERVALS • T wave • represents ventricular repolarization • usually the same direction as QRS complex • U wave • represents repolarization of Purkinje fibers • usually smaller than P wave • sometimes seen in patients with hypokalemia, HPN, or heart disease INTERPRETING THE ELECTROCARDIOGRAM WAVES, COMPLEXES, AND INTERVALS • PR interval • measured from beginning of P wave to beginning of QRS complex • N: 0.12 to 0.20 seconds in duration • ST segment • measured from end of QRS complex to the beginning of T wave • QT interval • represents total time for ventricular depolarization and repolarization • measured from beginning of QRS complex to end of T wave • N: 0.32 to 0.40 seconds in duration INTERPRETING THE ELECTROCARDIOGRAM WAVES, COMPLEXES, AND INTERVALS • TP interval • measured from end of T wave to the beginning of next P wave • no electrical activity, graph remains flat -> isoelectric line • PP interval • atrial rhythm and atrial rate • RR interval • ventricular rhythm and ventricular rate INTERPRETING THE ELECTROCARDIOGRAM DETERMINING HEART RATE FROM ECG • 1 minute-strip = 60 seconds • 300 large boxes (0.2 seconds each) • 1500 small boxes (0.04 seconds each) • Count the number of small boxes within an RR interval and divide that 1500 by that number • E.g. There are 25 small boxes within the RR interval, HR= 1500/25 = 60 BPM INTERPRETING THE ELECTROCARDIOGRAM DETERMINING HEART RATE FROM ECG
• Count number of RR intervals in 6 seconds then
multiply that number by 10 • 3 second-intevals = 15 large boxes NORMAL SINUS RHYTHM • Ventricular and atrial rate: 60 - 100 bpm • Ventricular and atrial rhythm: Regular • QRS shape and duration: Usually normal, but may be regularly abnormal • P wave: Normal and consistent shape • PR interval: Interval between 0.12 - 0.20 seconds • P:QRS ratio: 1:1 NORMAL SINUS RHYTHM NORMAL SINUS RHYTHM SINUS NODE DYSRHYTHMIAS SINUS BRADYCARDIA • Ventricular and atrial rate: Less than 60 • Ventricular and atrial rhythm: Regular • QRS shape and duration: Usually normal, but may be regularly abnormal • P wave: Normal and consistent shape • PR interval: Interval between 0.12 - 0.20 seconds • P:QRS ratio: 1:1 SINUS NODE DYSRHYTHMIAS SINUS BRADYCARDIA • Causes: • Vagal stimulation (vomiting, suctioning, severe pain) • Medications (calcium channel blockers, beta blockers) • ICP (hyper-brady-brady) • Myocardial infarction • Management: • Atropine to block vagal stimulation SINUS NODE DYSRHYTHMIAS SINUS BRADYCARDIA SINUS NODE DYSRHYTHMIAS SINUS BRADYCARDIA SINUS NODE DYSRHYTHMIAS SINUS TACHYCARDIA • Ventricular and atrial rate: greater than 100 bpm but usually less than 120 bpm • Ventricular and atrial rhythm: Regular • QRS shape and duration: Usually normal, but may be regularly abnormal • P wave: Normal and consistent shape, may be buried in preceding T wave • PR interval: Interval between 0.12 - 0.20 seconds • P:QRS ratio: 1:1 SINUS NODE DYSRHYTHMIAS SINUS TACHYCARDIA • Causes: • Physiologic/Psychological stress ( acute blood loss, anemia, exercise, anxiety) • Meds that stimulate sympathetic response (catecholamines) • Stimulants (caffeine, nicotine) • Illicit drugs (cocaine, ecstasy) • Postural orthostatic tachycardia syndrome (POTS) • Management: • Beta blockers or calcium channel blockers SINUS NODE DYSRHYTHMIAS SINUS TACHYCARDIA SINUS NODE DYSRHYTHMIAS SINUS TACHYCARDIA SINUS NODE DYSRHYTHMIAS SINUS ARRHYTHMIA • Ventricular and atrial rate: 60-100bpm • Ventricular and atrial rhythm: Irregular • QRS shape and duration: Usually normal, but may be regularly abnormal • P wave: Normal and consistent shape • PR interval: Interval between 0.12 - 0.20 seconds • P:QRS ratio: 1:1 SINUS NODE DYSRHYTHMIAS SINUS ARRHYTHMIA • Causes: • Increases with inspiration and decreases with expiration • Heart disease or valvular disease Management: • Usually not treated SINUS NODE DYSRHYTHMIAS SINUS ARRHYTHMIA SINUS NODE DYSRHYTHMIAS SINUS ARRHYTHMIA ATRIAL DYSRHYTHMIAS PREMATURE ATRIAL COMPLEX • “Heart skipped a beat.” • Ventricular and atrial rate: Depends on the underlying rhythm • Ventricular and atrial rhythm: Irregular due to early P waves, shorter PP interval, followed by a longer than usual PP interval • QRS shape and duration: QRS that follows the early P wave may be normal or absent (blocked PAC) • P wave: There may be an early or different P wave seen • PR interval: Interval between 0.12 - 0.20 seconds • P:QRS ratio: 1:1 ATRIAL DYSRHYTHMIAS PREMATURE ATRIAL COMPLEX • Causes: • Caffeine, alcohol, nicotine • Hypokalemia • Atrial ischemia • Often seen with sinus tachycardia Management: • If unfrequent, no treatment is necessary ATRIAL DYSRHYTHMIAS PREMATURE ATRIAL COMPLEX ATRIAL DYSRHYTHMIAS PREMATURE ATRIAL COMPLEX ATRIAL DYSRHYTHMIAS ATRIAL FLUTTER • Ventricular and atrial rate: Atrial rate: 250-400 bpm; Ventricular rate: 75-150 bpm • Ventricular and atrial rhythm:Atrial and ventricular rhythms are regular but ventricular rhythm may be irregular • QRS shape and duration: Usually normal, but may be abnormal or absent • P wave: Saw-toothed shape (F waves) • PR interval: Difficult to determine • P:QRS ratio: 2:1, 3:1, 4:1 ATRIAL DYSRHYTHMIAS ATRIAL FLUTTER • Causes: • COPD • Thyrotoxicosis • S/P Open heart surgery Management: • Administration of adenosine • Electrical cardioversion to convert rhythm to sinus rhythm ATRIAL DYSRHYTHMIAS ATRIAL FLUTTER ATRIAL DYSRHYTHMIAS ATRIAL FLUTTER ATRIAL DYSRHYTHMIAS ATRIAL FIBRILLATION • Ventricular and atrial rate: Atrial rate: 300-600 bpm; Ventricular rate: 120-200 bpm • Ventricular and atrial rhythm: Highly irregular • QRS shape and duration: Usually normal, but may be abnormal • P wave: No discernible P waves; irregular undulating waves that vary in amplitude and shape are seen • PR interval: Cannot be measured • P:QRS ratio: Many:1 ATRIAL DYSRHYTHMIAS ATRIAL FIBRILLATION • Causes: • Structural heart disease in people of advanced age • Pericarditis, Myocarditis • CAD • Heart failure • Sepsis • RHD • Mitral stenosis ATRIAL DYSRHYTHMIAS ATRIAL FIBRILLATION • Management: • If patient is unstable with ventricular rate > 150 bpm, prepare for immediate cardioversion • If stable, calcium channel blockers, digoxin, procainamide may be administered • Anticoagulation therapy to prevent emboli ATRIAL DYSRHYTHMIAS ATRIAL FIBRILLATION ATRIAL DYSRHYTHMIAS ATRIAL FIBRILLATION JUNCTIONAL DYSRHYTHMIAS PREMATURE JUNCTIONAL COMPLEX • Ventricular and atrial rate: Depends on the underlying rhythm • Ventricular and atrial rhythm: Irregular due to early P waves, shorter PP interval, followed by a longer than usual PP interval • QRS shape and duration: QRS that follows the early P wave may be normal or absent (blocked PAC) • P wave: May be absent, may follow QRS, or may occur before QRS but with • PR interval: Less than 0.12 seconds • P:QRS ratio: 1:1 JUNCTIONAL DYSRHYTHMIAS PREMATURE JUNCTIONAL COMPLEX • Causes: • Digitalis Toxivity • Heart failure • CAD Management: • If unfrequent, no treatment is necessary JUNCTIONAL DYSRHYTHMIAS PREMATURE JUNCTIONAL COMPLEX JUNCTIONAL DYSRHYTHMIAS PREMATURE JUNCTIONAL COMPLEX JUNCTIONAL DYSRHYTHMIAS JUNCTIONAL RHYTHM • The AV node becomes the pacemaker of the heart • Ventricular and atrial rate: 40-60 bpm • Ventricular and atrial rhythm: Regular • QRS shape and duration: Usually normal, but may be abnormal • P wave: May be absent, may follow QRS, or may occur before QRS, may be inverted • PR interval: Less than 0.12 seconds • P:QRS ratio: 1:1, 0:1 JUNCTIONAL DYSRHYTHMIAS JUNCTIONAL RHYTHM • Causes: • Increased vagal tone • Complete heart block Management: • Same as for sinus bradycardia JUNCTIONAL DYSRHYTHMIAS JUNCTIONAL RHYTHM VENTRICULAR DYSRHYTHMIAS PREMATURE VENTRICULAR COMPLEX • “Heart skipped a beat.” • Ventricular and atrial rate: Depends on underlying rhythm • Ventricular and atrial rhythm: Irregular due to early QRS, creating one RR interval that is shorter than the others • QRS shape and duration: Duration is 0.12 seconds or longer; shape is bizarre and abnormal • P wave: May be absent, or in front of QRS. If P wave follows the QRS, P wave shape may be different • PR interval: Less than 0.12 seconds • P:QRS ratio: 1:1, 0:1 VENTRICULAR DYSRHYTHMIAS PREMATURE VENTRICULAR COMPLEX • Causes: • Increased workload of the heart • Digitalis toxicity • Hypoxia • Electrolyte imbalances Management: • Lidocaine or amiodarone VENTRICULAR DYSRHYTHMIAS PREMATURE VENTRICULAR COMPLEX VENTRICULAR DYSRHYTHMIAS PREMATURE VENTRICULAR COMPLEX VENTRICULAR DYSRHYTHMIAS VENTRICULAR TACHYCARDIA • Three or more PVCs in a row, occurring at >100 bpm • Ventricular and atrial rate: Ventricular rate is 100-200 bpm • Ventricular and atrial rhythm: Usually regular • QRS shape and duration: Duration is 0.12 seconds or longer; shape is bizarre and abnormal • P wave: Very difficult to detect so atrial rate and rhythm may be indeterminable • PR interval: Very irregular, P waves are seen • P:QRS ratio: Difficult to determine but if P waves are apparent, less P waves: more QRS VENTRICULAR DYSRHYTHMIAS VENTRICULAR TACHYCARDIA • VT is an emergency because the patient is usually (although not always) unresponsive and pulseless. • Causes: • Larger MIs Management: • If pulseless, initiate CPR. • If with pulse, administer amiodarone. If ineffective, initiate synchronized cardioversion. VENTRICULAR DYSRHYTHMIAS VENTRICULAR TACHYCARDIA VENTRICULAR DYSRHYTHMIAS VENTRICULAR TACHYCARDIA VENTRICULAR DYSRHYTHMIAS VENTRICULAR FIBRILLATION • Most common dysrhythmia in patients with cardiac arrest • Ventricular rate: Greater than 300 per minute • Ventricular rhythm: Extremely irregular, without specific pattern • QRS shape and duration: Irregular, undulating waves without recognizable QRS complexes • P wave: Very difficult to detect so atrial rate and rhythm may be indeterminable • Characterized by absence of audible heartbeat, palpable pulse, and respirations. • Cardiac arrest and death are imminent if not corrected. VENTRICULAR DYSRHYTHMIAS VENTRICULAR FIBRILLATION • Causes: • MI • Digoxin, epinephrine toxicity • Electric shock Management: • If pulseless, initiate CPR. Follow protocols for defibrillation, intubation, and administration of epinephrine or vasopressin, lidocaine, or amiodarone. VENTRICULAR DYSRHYTHMIAS VENTRICULAR FIBRILLATION VENTRICULAR DYSRHYTHMIAS VENTRICULAR FIBRILLATION VENTRICULAR DYSRHYTHMIAS VENTRICULAR ASYSTOLE • Commonly called flatline • QRS shape and duration: Absent • P wave: May be apparent for a short duration • Characterized by absence of audible heartbeat, palpable pulse, and respirations. • Fatal VENTRICULAR DYSRHYTHMIAS VENTRICULAR ASYSTOLE • Causes: • MI • Cocaine Overdose Management: • Initiate CPR. VENTRICULAR DYSRHYTHMIAS VENTRICULAR ASYSTOLE CONDUCTION ABNORMALITIES FIRST-DEGREE ATRIOVENTRICULAR BLOCK • Ventricular and atrial rate: Depends on underlying rhythm • Ventricular and atrial rhythm: Depends on underlying rhythm • QRS shape and duration: Usually normal but may be abnormal • P wave: In front of the QRS complex; shows sinus rhythm, regular shape • PR interval: Greater than 0.20 seconds; PR interval measurement is constant • P:QRS ratio: 1:1 CONDUCTION ABNORMALITIES FIRST-DEGREE ATRIOVENTRICULAR BLOCK CONDUCTION ABNORMALITIES FIRST-DEGREE ATRIOVENTRICULAR BLOCK CONDUCTION ABNORMALITIES SECOND-DEGREE AV BLOCK, TYPE I (WENCKE-BACH) • Ventricular and atrial rate: Depends on underlying rhythm • Ventricular and atrial rhythm: Starting from the RR that is the longest, the RR interval gradually shortens until there is another long RR interval. • QRS shape and duration: Usually normal but may be abnormal • P wave: In front of the QRS complex; shape depends on underlying rhythm • PR interval: PR becomes longer with each succeeding ECG complex until there is a P wave not followed by a QRS • P:QRS ratio: 3:2, 4:3, 5:4, and so forth CONDUCTION ABNORMALITIES SECOND-DEGREE AV BLOCK, TYPE I (WENCKE-BACH) CONDUCTION ABNORMALITIES SECOND-DEGREE AV BLOCK, TYPE I (WENCKE-BACH) CONDUCTION ABNORMALITIES SECOND-DEGREE AV BLOCK, TYPE II • Ventricular and atrial rate: Depends on underlying rhythm • Ventricular and atrial rhythm: RR interval may be irregular depending on the P:QRS ratio • QRS shape and duration: Usually anormal but may be normal • P wave: In front of the QRS complex; shape depends on underlying rhythm • PR interval: PR interval is constant for those P waves just before QRS complexes • P:QRS ratio: 2:1, :1, 4:1, 5:1, and so forth CONDUCTION ABNORMALITIES SECOND-DEGREE AV BLOCK, TYPE II CONDUCTION ABNORMALITIES THIRD-DEGREE AV BLOCK • Ventricular and atrial rate: Depends on the escape and underlying rhythm • Ventricular and atrial rhythm: PP interval is regular and RR interval is regular, but PP interval is not equal to RR interval • QRS shape and duration: Depends on escape rhythm • P wave: Depends on underlying rhythm • PR interval: Very irregular • P:QRS ratio: More P waves than QRS complexes CONDUCTION ABNORMALITIES THIRD-DEGREE AV BLOCK CONDUCTION ABNORMALITIES THIRD-DEGREE AV BLOCK