uro blood flow > death of brain tissue in 4-10 mine

. mimic labyrinths
Jarebellar
-

importion
⑦ head/neck pain
< 16-18m2/1009 tissue >
-
infarction in antr :

cerebellar edema - ↑ ICP - > Obstruct CSF >

-
hydrocephalus

ome/Nog tisme >

-
Ischemia wo infarction unless
prolonged On
-

Focal cerebral infarction pathways :

manifest

respiratory
-
as
Coma,
arrest rectly compressing,
the brainstern

① Nurotic Pathway
② Apoptotic Pathway INTRAVENOUS THROMBOUS

IVUTPA
-
(0 .

9 mg/kg ; 90mg max (

In stroke , prevent & mrghyumia -
10 % bolus

- remainder over 60 mins

.

First goal in the ty of stroke

>
- prevent/revere brain injury 0 .
6mg/kg >
- dosage used in Japan 2 other asian pop
.

Hemorrhagic Tenectplase (0 .
25
mg/kg IV holus over 55)
-

~ depressed level of consciousness -given wo need for a th infusion

~ higher initial BP

~
wonning of symptoms after out ENDOVASCAR REVASCULANIZATION

Thrombectomy for Basilar Occlusion

Ischemic -
safe upto 24h from symptom onset
-

at ment
~
deficit maximal

ANTITHROMBOTIC TREATMENT

subcutaneous Heparin Aspirin 0 clopidogrel

-
unfractimated & LMWH (may be concomitant) Aspirin Ticagre
Jeffective in
preventing and strike

BP should be lowered if
~ RP 220/O

/ w/malignant HIN

~concom mytadial ischemia

.

~ F p
> 185/110 & thomholytic therap is anticipated

Serum quicon
should be 60-150

Edema peaks on the

2nd or
d day
Embolic strokes

/occur suddenly

/max deficit at onset

Pulmonary Arteriovenous Malformations

Bubble-contrast echo
-

-
intracardiac shunt O

Dominant mechanism causing large-vessel brain ischemia

>
- Artery-Artery Embolic stroke