Professional Documents
Culture Documents
Maternity
Maternity
Learning Outcomes:
- Describe the scope of perinatal nursing in Canada
- Describe current trends in perinatal health
- Consider how the social determinants of health influence the health of women and newborns
and discuss approaches needed to address health inequities in Canada
- Multiple births
- Inequities in access to quality maternity care still exist in Canada, particularly in rural, remote,
inner-city, and First Nations, Metis and Inuit communities
- Increase in low birth weight and preterm births
- Breast feeding rates
- Inductions and caesarean delivers
Learning Outcomes
- Describe the processes of fertilization and the development of the normal embryo and fetus
- Discuss the importance of preconception and prenatal care
- Describe expected maternal and family adaptations to pregnancy
Preconception Care
Goals of preconception care include:
• To optimize the health of all women PRIOR to entering into pregnancy
• To identify / modify risk factors (medical, behavioural, social) in order to improve health prior
to conception
Components of preconception care include
• Health Promotion
• Risk assessment and interventions
1. The placenta
2. The membranes
3. Amniotic fluid
4. Umbilical cord
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1. The Placenta
• Chorionic Villi attach placenta to uterine wall
• Serves as the lungs, kidney, and digestive tract
• Continues to grow throughout the pregnancy. At the end of 38 - 40 weeks, the placenta usually
is approximately 15 - 20 cm in diameter and 2 - 3 cm in depth. It covers about half of the
mineral uterine wall, and weighs approximately 400 - 600 gm (1 pound)
• Oxygen and nutrients osmose from the maternal blood to the embryo through the placenta
• Most substances (including alcohol and nicotine) are able to cross from maternal blood to the
fetal circulation through the placenta
• There is no direct enhance of blood between the embryo and the mother during pregnancy
• Blood flow through the placenta increases with gestation (i.e., is approximately 50ml/min at
10 weeks and approximately 500-600ml/min at 40 weeks)
• Uterine perfusion, and this placental circulation, is most efficient when the women lies on her
left side. This position takes pressure off the inferior vena cava, preventing blood from being
trapped in the woman’s lower extremities. When lying on her back, pregnant women may
begin to experience supine hypotension
• A healthy placenta usually weighs approximately 400-600gm (1lb) at birth
2. The Membranes
• The chorionic membrane develops from the chorionic villi. It
is the outermost fetal membrane that serves to form a sac that
contains amniotic fluid
• There is also a second membrane lining the chorionic
membrane, known as the amniotic membrane
• At birth, these membranes (both fused together as one) can be
seen covering the fetal surface of the placenta, giving this
surface a shiny appearance.
• There is no nerve supply in this membrane, therefore, when they eventually rupture, neither the
mother or the infant experience any pain
• The amniotic membrane is involved in the production of amniotic fluid
3. Amniotic Fluids
• Is constantly being newly formed and reabsorbed by the amniotic membrane, so it never
becomes stagnant.
• The major method of absorption of amniotic fluid occurs because the cutis continually
swallows the fluid. This fluid is then reabsorbed into the fetal blood stream from the fetal
intestine
• Most women will have approximately 80-1200 ml of amniotic fluid at term
• Polyhydramnios (more than 200ml of fluid) can sometimes occur when a fetus is unable to
swallow the amniotic fluid (i.e., with esophageal atresia or anencephaly). It is also commonly
seen in women who are diabetics (hyperglycaemia causes excessive fluid shifts into the
amniotic space)
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• When the fetal kidneys begin to function, fetal urge also adds to the quantity of the amniotic
fluid. A disturbance of fetal kidney function may result in oligohydramnios (a reduction in the
amount of amniotic fluid to less than 300ml)
• The main function of amniotic fluid is to:
• (a) shield the fetus against pressure or blow to the mother’s abdomen,
• (b) protect the fetus from temperature changes,
• (c) acid in muscular development because it allows the fetus freedom to move, and
• (d) protect the umbilical cord from pressure, which in turn protects fetal oxygen supply
Fetal Circulation
- Fetal circulation differs from extrauterine circulation because the fetus derives oxygen and
excretes carbon dioxide not from gas exchange in the lung but from gas exchange in the
placenta
- Blood arriving from the placenta to the fetus is highly oxygenated (carried through the
umbilical vein). This vessel is called a vein rather than an artery even though it us carrying
oxygenated blood. This is because it is carrying blood toward the fetal heart
- Blood is then shunted through specialized structures (i.e., ductus venous, foramen oval, ductus
arteriosus) to ensure that blood flows to the most important organs of the body first (i.e., brain,
liver, heart, and kidneys.)
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Fetal Maturation
Respiratory system
Neurological System
- Development of alveoli - Spinal cord disorders
- Breathing practice
- Surfactant
Musculoskeletal System
- Age of viability - Bone ossification
- Lung maturity and betamethasone - Fetal movements
Gastrointestinal System
Integumentary System
- Meconium - Lanugo
- Vitamin K synthesis - Vernix caseosa
Renal System
Immune System
- Urine excretion - Maternal antibioses and passive immunity
- Oligohydramnios
Obstetrical Terms
• Gravida: a women who is pregnant
• Multigravida: a women who has had two or more pregnancies
• Multipara: a women who has completed two or more pregnancies to 20 weeks of gestation or
more
• Nulligravida: A women who has never been pregnant
• Nullipara: a women who has not completed a pregnancy with a fetus beyond 20 weeks
• Postterm: a pregnancy that goes beyond 41 weeks gestation
• Preterm: a pregnancy that has reached 20 weeks gestation, but before completion of 37 weeks
gestation
• Primapara: a women who has completed one pregnancy with a fetus who reach 20 weeks
gestation or more
• Fullterm: a pregnancy between 37 weeks - 41 weeks gestation
• Viability: The capacity to live outside the uterus occurring about 22 - 25 weeks gestation
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Ovarian Changes:
- Ovulation ceases
Breast Changes:
- Breast tenderness
Cardiovascular Changes:
- Cardiac output, blood volume and heart rate
- Peripheral blood flow changes
Respiratory Changes:
- Nasal congestion
- Shortness of breath
Body Temperature:
- Slight decrease due to hormonal changes
GI Changes
- Nausea/vomiting
- Gastric reflux/heartburn
- Hypertrophy and bleeding of gums
- Increased saliva production
- Decreased pH of saliva
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Urinary Changes:
- Fluid retention
- Urinating frequently
Skeletal Changes:
- Change in center of gravity while ambulating leads to postural changes
- Back pain
High Risk Pregnancy: When the life or health of the mother and/or fetus is jeopardized during
pregnancy
- Although most pregnancies and births are considered low risk, there are still some births that
are categorized as high risk due to maternal or fetal complications
- To prevent morbidity and mortality, timely identification of these risks, along with timely
implementation of appropriate interventions, is required
Third Trimester:
- Fetal movement routing
- Non-stress test (NST)
- Contraction Stress Test (CST)
- Biophysical Profile (BPP)
Gestational Conditions
- There are many disorders that can develop during pregnancy, which can place the woman and
her fetus at serious risk
- The next several slides will provide you with a brief overview of the disorders listed below
- Pre-eclampsia
- Gestational diabetes mellitus
- Hyperemesis Gravidarum
- Ectopic Pregnancy
- Placenta Priva
- Placental Abruption Rh incompatibility
Pre-eclampsia / Eclampsia
• A serious condition that affects about 5% of pregnant
women, and typically starts after 20 weeks gestation
• Elevated BP is the primary symptom, but there may be other
symptoms such as protein in the urine, liver or kidney
abnormalities, persistent headaches, or vision changes
• Pre-eclampsia causes blood vessels to constrict, which can
result in reduced blood flow / injury to the liver, kidneys, brain, and uterus.
• Decreased blood flow to the uterus can lead to poor growth of the fetus, too little amniotic
fluid, placental abruption, or premature birth.
• Changes in blood vessel endothelial cells also causes capillaries to “leak” fluid into the
interstitial spaces. This results in fluid retention, rapid weight gain, and excessive swelling of
the face, arms, hands, legs, and feet.
• The condition can progress slowly or rapidly from mild symptoms to severe, life-threatening
Eclampsia (a convulsive state), if not diagnosed and treated promptly.
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Pathophysiology of Pre-eclampsia
- The spiral arteries of the uterus (located in the endometrium) play an important role in
providing blood flow to the growing placenta and fetus.
- Normally, the spiral arteries widen during pregnancy to increase blood flow to the placenta.
This widening is thought to be influenced by how well the trophoblast burrowed into the
uterus during the time of implantation. If the
trophoblast failed to implant correctly, the spiral
arteries will fail to widen, and instead, stay
narrow as the pregnancy progresses.
- Over time, this causes the placenta to become
very oxygen deprived. In response, the placenta
releases substances (i.e., pro-inflammatory
proteins) into the maternal circulation in an
attempt to increase blood flow to it.
- However, these substances are very toxic to
maternal endothelial cells (cells that line the
inside of blood vessels throughout the body and
organs).
- When the maternal endothelial cells become
damaged, vasospasm of the blood vessel walls
results. This causes the blood vessel to become
rigid and much narrower than normal, which leads to maternal hypertension.
- Damaged endothelial cells also result in an increase in permeability of the blood vessel wall.
This allows fluid from the vascular system to leak into the interstitial spaces.
- This leads to fluid retention, rapid weight gain, and excessive swelling / pitting edema of the
face, arms, hands, legs, and feet.
Hyperemesis Gravidarum
Pathophysiology
• This is an extreme condition that causes long-lasting intense nausea, vomiting, and weight loss
during pregnancy.
• It is different than the usual morning sickness that occurs during pregnancy in that the
symptoms are intense and persistent, leading to weight loss, dehydration, and electrolyte
imbalances.
• Usually develops during the 4th – 6th weeks of pregnancy and may last throughout the majority
of the pregnancy. The nausea is continuous and women who are suffering from the illness are
frequently exhausted over time, which considerably influences their daily life.
• While the exact cause of this disorder is not fully understood, it is thought to be related to the
rapid rise in hCG levels.
• Complications that may arise from excessive vomiting include dehydration, renal impairment,
malnutrition, and electrolyte imbalance.
Ectopic Pregnancy
• Is when implantation occurs outside of the uterine cavity (i.e., inside the fallopian tube, on the
surface of the ovary, inside the abdomen, or in the cervix)
• The most common site of an ectopic pregnancy is in a
fallopian tube.
• Often no symptoms at the time of implantation (i.e., there is
no menstrual flow, the woman may experience nausea and
vomiting, and a pregnancy test for HcG will be positive).
• However, at 6 – 8 weeks gestation, the embryo grows large
enough to rupture the slender fallopian tube, resulting in
tearing of blood vessels and severe intraperitoneal bleeding.
• When the fallopian tube ruptures, the woman may feel a
sharp, stabbing pain in one of her lower abdominal
quadrants at the time of rupture, followed by scant
vaginal spotting. The scant amount of vaginal bleeding can
be deceptive, as most blood may be expelled into the pelvic
cavity rather than into the uterus.
• The ectopic pregnancy may only be diagnosed once the woman begins to experience
hypovolemic shock (rapid thready pulse, rapid respirations, decreased blood pressure, pale,
cool clammy skin), hence why all ectopic pregnancies are considered an emergency.
Placenta Previa
• A condition when the placenta is implanted abnormally in the uterus. It is the most common
cause of PAINLESS BLEEDING in the third trimester of pregnancy.
• Occurs in various degrees:
• Low lying placenta (placenta is implanted in the lower portion of the uterus but does not
cover the cervical os)
• Marginal implantation (placenta edge approaches the cervical os)
• Partial placenta previa (placenta occludes a portion of the cervical os)
• Total placenta previa (i.e., totally obstructs the cervical os)
• Most often diagnosed early during the first fetal ultrasound. When diagnosed, the mother
should be advised to AVOID COITUS, to get adequate rest, and to call her health care
provider at any sign of vaginal bleeding.
• Bleeding usually starts to occur in the 30th week of gestation
• Betamethasone (a steroid that hastens fetal lung maturity), may be prescribed if the fetus is less
than 34 weeks.
• Birth must occur as soon as possible when:
• (1) the fetus reaches 37 weeks of age,
• (2) An amniocentesis analysis for lung maturity shows a positive result (i.e., favorable
lecithin-sphingomyelin ration (L/S ratio),
• (3) bleeding occurs again,
• (4) labour begins, or
• (5) the fetus shows symptoms of distress.
• After birth, the mother monitored carefully for:
• (1) post-partum hemorrhage (because the lower segment of the uterus does not usually
contract as efficiently as the upper segment), and
• (2) endometritis (because the placental site is close to the cervix, the portal of entry for
pathogens.
Rh Incompatibility
• Rh incompatibility occurs when an Rh-negative mother carries a fetus with an Rh positive
blood type
• There is usually no connection between fetal blood and maternal blood during pregnancy, so
the mother should not be exposed to fetal blood.
• However, if a villus ruptures, small amounts of fetal blood can occasionally enter the maternal
circulation.
• When this happens, the mother will develop Rh + antibodies. These antibodies will then attack
the fetus as though the fetus was a foreign invasion.
• To reduce the number of maternal Rh antibodies being formed (which could negatively affect
all future pregnancies), the woman is given an Rh immune globulin shortly after birth (i.e.,
RhoGam)
(1) The Passenger: Movement of the passenger (I.e the fetus) is determined by several
interacting components including:
Fetal presentation
- The term presentation refers to the part of the fetus that enters the pelvic inlet first and leads
through the birth canal during labour
- The three main presentations are: 1- cephalic (head first) 2- breech (buttocks or feet first) and
3- transverse (shoulders first)
- The term presenting part refers to the part of the fetal body that is first felt by the examining
finger during a vaginal examination (I.e. cephalic presentation - presenting part is usually the
occiput; breech presentation - usually the sacrum; should presentation- scapula)
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Fetal lie
- This refers to the relation of the spine of the fetus to the spine of the mother
- There are 3 primary lies:
1. Longitudinal (vertical) (spine of the fetus is parallel with mother’s spine
2. Oblique (spine of fetus is diagonal to mother’s spine
3. Transverse (horizontal), or (spine of the fetus is either at a right angle to the mother’s
spine)
- Birth cannot occur if the fetus remains in a transverse lie
Fetal attitude
- Refers to the relation of the fetal body parts to one
another (posture)
- Normal attitude (general flexion): back of the
fetus is rounded, chin is flexed on the chest, thighs
are flexed on the abdomen, and legs are flexed at
the knees, arms are crossed over the thorax, and
the umbilical cord lies between the arms and the
legs
- Any deviation from the normal attitude can pose
problems during the labour / birth process
Fetal Position
- This refers to the relation of the presenting part to the
four quadrants of the mother’s pelvis
- The first letter of the abbreviation indicated location of
the presenting part in the right (R) or left (L) side of the
mother’s pelvis
- The second letter stands for the specific presenting part
of the fetus (O = occiput; S = sacrum; M = mentum; and
Sc = scapula)
- The third letter stands for the location of the presenting
part in relation to the anterior (A), posterior (P), or
transverse (T) portion of the paternal pelvis.
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Mechanisms of Labour
- Passage of a fetus through the birth canal involves several different position changes, termed
the cardinal movements of labour, which include the following:
- Engagement & Descent (i.e., the downward movement of the biparietal diameter of the fetal
head to within the pelvic inlet),
- Flexion (i.e., when the head reaches the pelvic floor it bends forward onto the chest),
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hastening the dilatation process). If membranes rupture before the fetal head is engaged in the
pelvis, prolapsed cord may result.
- Your textbook provides detailed information with regards to some of the more common
complications that you will see while working in a birthing center. Please review the
following in your text as there may be some questions on this in the future.
- Preterm Labour & Birth
- Post term Pregnancy, labour, and birth
- Induction of Labour
- Augmentation of Labour
- Caesarean Birth
- Vaginal Birth after Caesarean (VBAC)
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