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ABSITE SLAYER
SECOND EDITION
 NOTICE
Medicine is an ever-changing science. As new research and clinical experience broaden our knowledge,
changes in treatment and drug therapy are required. The authors and the publisher of this work have
checked with sources believed to be reliable in their efforts to provide information that is complete
and generally in accord with the standards accepted at the time of publication. However, in view of the
possibility of human error or changes in medical sciences, neither the authors nor the publisher nor
any other party who has been involved in the preparation or publication of this work warrants that the
information contained herein is in every respect accurate or complete, and they disclaim all responsi-
bility for any errors or omissions or for the results obtained from use of the information contained in
this work. Readers are encouraged to confirm the information contained herein with other sources. For
example, and in particular, readers are advised to check the product information sheet included in the
package of each drug they plan to administer to be certain that the information contained in this work
is accurate and that changes have not been made in the recommended dose or in the contraindications
for administration. Titis recommendation is of particular importance in connection with new or infre-
quently used drugs.
 ABSITE SLAYER
SECOND EDITION

EDITORS

Dale A. Dangleben, MD, FACS

Trauma Medical Director
Associate Clinical Professor
Geisinger Commonwealth School of Medicine
Geisinger Holy Spirit
Camp Hill, Pennsylvania

Firas G. Madbak, MD, FACS, FCCP

Associate Professor of Surgery
Division of Acute Care Surgery
Program Director, Surgical Critical Care Fellowship
Medical Director, Surgical Intensive Care Unit
Department of Surgery
University ofRorida College of Medicine-Jacksonville
Jacksonville, Florida

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 Dedication
'This work is dedicated to my father, Arthur Charles Dangleben, who told me that education was
the gateway to a better future. Rest in peace, Dad.

Dale A. Dangleben, MD, FACS

This page intentionally left blank
 CONTENTS
Contributon ............................................................D:
Introduction: General Test-Taking Tips .................:si
1 Cell Biology ........................................................ 1
Christie Buonpane
2 Hematology ...................................................... 13
Christine Du
3 'lr&Diplant and Immunology ........................... 25
Patty T. Liu
4 Infection and Antibiotigs .................................41
Christie Buonpane and Dale A. Dangleben
5 Pharmacology................................................... 53
Shannon Brindle
6 Anesthesia......................................................... 63
PAtty T. Liu
7 Fluids/Eledrolyte9/Nutrition........................... 71
Firas G. MAdbak
8 Surgical Oncology ............................................ 91
RyAn A. LAwless
9 Wound Healing............................................... 105
DAie A. DAngleben
10 Head and Nedr................................................. 113
MAhdi Malekpour And Dale A. Dangleben
11 Breast •••••••••••••••••••••••••••••••••••••••••••••••••••••••••••••• 125
Christine Du
12 Endoai.ne ....................................................... 143
DAnielle Press
13 AbdomJnal Wall and HernJas......................... 167
Dale A. Dangleben
14 Stomach .......................................................... 185
Ginger L. Justice
15 Hepatobiliary.................................................. 203
MoustApha Dimachk And Firas G. Madbak
16 Panc:reu ..........................................................219
Duiree Raygor and Firas G. Madbak
17 Spleen..............................................................231
RyAn A. Lawless
18 Esophagus .......................................................241
Alexander Ghannam and Firas G. Madbak
19 Small Intestine ................................................261
Charles Walker and Dale A. Dangleben
20 Colorectal. .......................................................283
Charles Walker And Dale A. Dangleben
21 Trauma............................................................ 301
Dale A. Dangleben
22 Critical Care ...................................................331
Dale A. Dangleben and Firas G. Madbak
23 BUJ'llll............................................................... 349
KArin McConville
24 Vascular .......................................................... 361
Mahdi Malekpour
25 Pediatria ........................................................ 379
Scott Sylvester
26 Pla.tic: and Rec:omtruc:tive Surgery ...............405
Emily A. Peterson
27 Thorade: Surgery ............................................417
Dale A. Dangleben
28 Orthopedla ....................................................437
Yelena Bogdan
29 Neurosurgery..................................................449
DAie A. Dangleben
30 Obstetrigs and Gynec:ology ••••••••••••••••••••••••••••457
LinaFouad
31 Statiltl.c:s ..........................................................471
Dale A. Dangleben
32 Ethia and Profa1ionalinn ............................479
Robert D. Barraco And Stephen E. Lammers
Inda.....................................................................487
Yii
This page intentionally left blank
 CONTRIBUTORS

Rebert D. Barraco, MD, MPH, FACS, KCP Christine Du, MD, FACS
Chief Section of Geriatric 'Irauma and Trauma Lehigh Valley Health Network
Outreach LVPG- Transplant Surgery
Chair Institutional Ethics Committee Allentown, Pennsylvania
Lehigh Valley Health Network Chapter 2-Hematology
Department of Surgery Chapter 11-Breut
Allentown, Pennsylvania
Chapter 32-Ethia and Profasionalinn Lina Fauad, MD, FACDG
Assistant Professor of Obstetrics/Gynecology
Y1l1na Bagd1n, MD University of Florida College ofMedicine - Jacksonville
Orthopaedic Trauma Jacksonville, Florida
Clinical Associate Professor of Orthopaedics Chapter 30-0bstetria and Gynecology
Geisinger Co=onwealth School of Medicine
Camp Hill, Pennsylvania Aluander Ghannam, MD
Chapter 28-0rthopedica Resident Physician, General Surgery
Department of Surgery
Shannon Brindle, MD University of Florida College of Medicine-Jacksonville
PGYS Chief General Surgery Resident Jacksonville, Florida
Geisinger Medical Center Chapter 18-~ophagu9
Danville, Pennsylvania
Chapter 5-Pharmacology Ginger L. Justice, MD
Clinical Assistant Professor of Surgery Geisinger
Christle Buenpane, MD Co=onwealth School of Medicine
PGY4 General Surgery Resident Trauma, Acute Care Surgery, & Surgical Critical Care
Geisinger Medical Center Trauma Surgical Services
Danville, Pennsylvania Geisinger Holy Spirit Hospital
Chapter I-Cell Biology Camp Hill, Pennsylvania
Chapter 4-Infection and Anb'biotiai Chapter 14-Stomach

Dale A. Dangl1b111, MD, FACS Sh!ph111 E. Lamm1rs, PhD

Trauma Medical Director Helen H.P. Manson Professor ofthe English Bible
Associate Clinical Professor Emeritus, Lafayette College
Geisinger Co=onwealth School of Medicine Ethics Program Consultant
Geisinger Holy Spirit Lehigh Valley Health Network
Camp Hill, Pennsylvania Allentown, Pennsylvania
Chapter 4-Infection and Antibiotia Chapter 32-Ethla and Profe1111lonalhm
Chapter 9-Wound Healing
Chapter 10-HeadandNeck RJin A. Lawless, MD
Chapter 13-Abdominal Wall and Hemiu Acute Care Surgeon, Denver Health Medical Center
Chapter 1!1-Small Intelti.ne Assistant Professor of Surgery
Chapter 20-Colorectal University of Colorado School of Medicine
Chapter21-Trauma Aurora, Colorado
Chapter 22-Critical Care Chapter 8-Surgi.cal Oncology
Chapter 27-1borad.c Surgery Chapter 17-Spleen
Chapter 29-Neuroswgery
Chapter 31-StatUtiai PlttJ T. Uu, MD
Chief, Pancreas Transplantation, Lehigh Valley Health
Moustapha Dlmachk, MD Network
Chief Surgery Resident LVPG Transplant Surgery
Department of Surgery Allentown, Pennsylvania
University of Florida College of Medicine Chapter 3-Trllllllplant and Immunology
Jacksonville, Florida Chapter 6-Anesthe1la
Chapter 15-Hepatobillary
ll Contributors

Firu G. Madblk, MD, FACS, FC<P Danielle Press, MD FACS

Alisistant Professor of Surgery General and Endocrine Surgeon
Division of Acute Care Surgery ChristianaCare
Program Director, Surgical Critical Care Fellowship Department of Surgery
Medical Director, Surgical Intensive Care Unit Newark, Delaware
Department of Surgery Chapter 12-Endocrine
University of Florida College of Medicine-Jacksonville
Jacksonville, Florida Desiree Raygor, MD
Chapter 7-Fluidai/Electrolytea/Nutrition Resident physician
Chapter 15-Hepatobillary Department of Surgery
Chapter 16-Pancreu University of Florida College of Medicine
Chapter 18-Esophagus Jacksonville, Florida
Chapter 22-Critical Care Chapter 16-Pancreu

Mahdi Malekpour, MD Smtt Sylvestlr, MD

Geisinger Medical Center Burn Fellow
Danville, Pennsylvania 1he Johns Hopkins School of Medicine, Department of
Chapter 10--Head and.Neck Plastic and Reconstructive Surgery
Chapter 24-Vuc:ular Baltimore, Maryland
Chapter 25-Pediatriai
Karin Mc:Carnille, MD
Trauma Surgeon Charles Walker, MD
Chapter 23-Bumi Chief General Surgery Resident
Geisinger Medical Center
Emily A. Peterson, MD Danville, Pennsylvania
Plastic Surgeon Mount Nittany Physician Group Chapter 1!1-Small Intestine
Reconstructive and Cosmetic Surgery State Chapter 20-Colorectal
College, Pennsylvania
Chapter 26-Plastic and Recomtructive Surgery
 INTRODUCTION: GENERAL TEST-TAKI NG Tl PS

1. A good night's sleep before exams brings renewed spirit and concentration to
complete the task.
2. Simple meditation or yoga on the morning of the exam can help with stress and
anxiety release and promote focus.
3. Do not carb-load for breakfast because you may crash and get fatigued during the
exam, so balance it out.
4. It is not a one-night preparation, nor one week, nor one month. It is a continuous
year-round schedule.
5. Do not second-guess yourself.
6. Do not choose an answer because you have not heard of it.
7. There is no 100% item on test. Each year a pattern is seen: some commonly asked
questions do not show up, but common things are common. You have to know the
basics.
8. Do not overthinkthe questions. Keep it simple.
9. The night before the test should not be devoted to an "all-nighter"or intense
review. Read a few things to ease your conscience, but spend time having a good
meal and, more importantly, getting a good night's sleep.
10. Beware of the urge to change answers. Statistically, your first answer is correct
more often than a changed answer.
11. If you know an answer is correct but can't remember why, the reason may not
matter. For the sake of the test, so you don't get too hung up on one single
question, just answer the question and move on.
12. Don't get bogged down by wordy or long questions. Often the last sentence or 2
tells the primary question being asked.
13. The best prep for the ABSITE is RESIDENCY and a small amount of daily formal
study.
Always do what you do in real life: stabilize patients before the operating room,
never send an unstable patient to the CT scanner, and always remember to
differentiate sick patients from nonsick patients.
14. "Get to 2": these exams are usually about narrowing down the answers to 2 likely
choices. Then go back to look for the clues to sort out these final options.
1s. Study hard in order to treat your patients in the best way possible, not to take an
exam.
16. As noted in tip #9, it is a bad idea to try to study the night before an ABSITE exam.
This can lead to finding information that you have not totally mastered and may
affect your confidence for the exam. Ifyou are going to review a topic, choose a
topic in which you are well versed to boost your confidence for the exam.
17. Layer your clothing for the exam. You never know what the room temperature will
be like.
18. Eat breakfast, but avoid eating heavy foods. Bring snacks to the test.

11
Ill Introduction: General Test-Taking Tips

19. Questions are generally"fluff free."There is little fluff in the questions. If they
wanted you to know more, they would have told you! The absence of clues toward
a particular decision is a clue that you should NOT be moving in that direction.
20. Consider bringing Tylenol and ibuprofen for muscle aches or headaches.
21. The test writers love the"thoughtless trap." For example, they will give you a
patient with colon cancer who needs an operation, but they will also mention the
patient had an Ml last week. You have to factor the Ml into your decision. Read the
questions carefully: there is usually more than enough time.
22. Remember, the ABSITE is an endurance test. Pace yourself wisely and take a short
break if necessary to get back on track.
23. They want you to get it right! Only a handful of questions are designed to separate
out the ninety-ninth percentile from the ninety-eighth percentile.

Dale A. Dangleben, MD, FACS

Firas G. Madbak, MD
CHAPTER 1
Cell Biology
Christie Buonpane

Test Taking Tip

Often, complex cell biology is hidden within a question seemingly about a clinical scenario. If you can parse the implied
question from the background without wasting time on superfluous infonnation, you will have the best chance at efficiently
making progress through tough questions.

CELL MEMBRANE
Which cell wall component increases membrane fluidity?
Cholesterol
What are the 3 main lipid classes found in the cell membrane?
Phospholipids, cholesterol, and glycolipids
What percentages ofprotein, carbohydrate, and lipid compose the plasma membrane?
Protein: 60%, carbohydrate: 1% to 10%, and lipid: 40%
What are the most common phospholipids in the plasma membrane?
Phosphatidylethanolamine and phosphatidylcholine
Which portion ofthe cell wall provides capacitance (ability to store charge)?
Lipid portion of plasma membrane
Which portion provides the ability to resist charge?
Protein portion
What is the difference between surface antigens in the ABO system and the ULA
system?
ABO = glycolipids
HLA = glycoproteins
Name the adhesion molecules that anchor a cell to other cells:
Desmosomes
Name the adhesion molecules that anchor a cell to extracellular matrix molecules:
flemidesmosomes
Cell-cell occluding junctions that form a water-impermeable barrier:
Tight junctions
Tuxic portion oflipopolysaccharide complex:
Lipid A
2 CHAPTER 1 Cell Biology

What synthesizes <:AMP and serves as a se<:ond messenger to activate various c;:ell
enzymes and proc;:essesf
Adenylate cyclase

CELL STRUCTURES

Name the thin filaments that interact with myosin to cause muscle contraction:
Actin
Name the thkk :filaments that slide along actin utilizing ATP:
Myosin
lntennediate filament found in hair and nails:
Keratin
Intennediate filament found in muscle:
Desmin
Intennediate filament found in fibroblasts:
Vimentin
Form specialized cellular structures such as mitotic spindles, cilia, and neuronal
axons; form lattic;:e inside the cell to aid in transport of organelles in cell:
Microtubules
Specialized microtubule that forms spindle fibers during cell division:
Centriole
Structural component of cell that synthesizes exported proteins:
Rough endoplasmic reticulum
Structural component of cell that detoxifies drugs and is involved with lipid/steroid
synthesis:
Smooth endoplasmic reticulum
Structural component of a cell that uses carbohydrates to modify proteins and targets
proteins to lysosomes:
Golgi apparatus
Structure inside the c;:ell that has a double membrane with an outer membrane that is
continuous with the rough endoplasmic reticulum:
Nucleus
Structure inside the nucleus with no membrane where ribosomes are made:
Nucleolus
Cell structure responsible for energy production:
Mitochondria

GENETICS

Consists of proteins, histones, and double-stranded helical DNA:

Chromosomes
 Nucleus \ \Cytoplasm
DNA mRNA Protein
tumovar turnover

Transcription /
Nudear envelope i\ I RNA
! I I Protein
degradation
RNA~~'~­ Posttranslational Actlw
transcript / processing I mRNA A ~ mRNA ~ Protein modification protein

RNA
transport

Transcriptional Posttranscrlptional Translational Posttranslational

control control control control

FIGURE 1-1. Fnurmajor stepsin themntrol ofeukaryoticgene l!XJlression. Transaiptional and posttransaiptional cootid detenninethe level ofmRNAthat ismdable to makea ~in, whereas tran~ational and posttranslational mntrol determine the
flnal ootarne cffunctional proteins. Note that posttransalptional and posttranslational contro~conslst ofsmralsteps (l/eproduadwlthptrmlsslon tiDm Brunlam/IF(.AndmenDK, BJ/Jlm"Tll,ttal: Sdi~ Prlnc/pltsofSwyer;, 1lthtd. Ntw Yo.rJ:. Nr.
McGtrMHm; 2019.) ~
""
a·
i
,..
I:

-
.::..s
...
4 CHAPTER 1 Cell Biology

Adenine and guanine are examples of:

Purines
Cytosine, thymidine, and uracil are examples of:
Pyrimidines
Process by which ribosomes use mRNA as a template for synthesis of proteins:
Translation
Process by which RNA polymerase uses a DNA strand for synthesis ofmRNA:
Transcription
Place where transcription takes place:
Nucleus
Sequence ofthe start codon:
AUG
Coils ofDNA that are the bask units of DNA pukaging:
Nucleosomes
Small basic proteins that nonspecifi.cally bind with DNA segments:
Histones
Formed bythe coiling of 6 or more nucleosomes by the histone Hl:
Solenoids
Proteins are synthesized from:
mRNA
Enzyme involved in the unwinding of DNA:
DNA helicase
Enzyme used to catalyze the formation of the RNA primers used to initiate DNA
synthesis:
DNAprimase
Enzyme that links DNA fragments by degrading RNA primers:
DNAligase
Type ofmutation that results in a single amino acid change from a point mutation:
Missense mutation
Type ofmutation resulting in a change in a single base pair:
Point mutation
Type ofmutation occurring from a point mutation that results in replacement of an
amino acid with a stop codon:
Nonsense mutation
Type ofmutation that oa:urs with the addition or deletion of a few base pairs:
Frameshift mutation
Technique by which DNA can be amplified a billion-fold by utilizing synthesized
primers/oligonucleotides to complement a strand ofDNA:
Polymerase chain reaction
 Cell Biology CHAPTER 1 5

Nonwding regions that interrupt eukaryotk genes:

Introns
Process by which introns are removed from an RNA transcript:
Splicing

RECEPTORS AND SIGNALS

Platelet-derived growth factor, epidermal growth factor, and transforming growth
factor alpha belong to this receptor family:
Tyrosine kinase receptor
Activated by calcium and diacylglycerol:
Protein kinase C
Activated by cAMP:
Protein kinase A
Enzyme that wnverts membrane phosphoinositols into IP3 and DAG:
Phospholipase C

Death signal
(eg, TNF or Fas)

Death
t Plasma
receptor - - ~membrane

Death
receptor
aipling Cytochrome c
I
pathway release

\
Activation
caspase cascade
I
ot •
Apoptotic target cell

Normal target cell

FIGURE 1-2. Allmpllfied Ylew ofthe apoptosls pathways. Extracellular death receptor pathways Include theactivation of Fasand tumor neaosls factDr reaptorsand consequent
activati111 of the caspase pathway.The intracellulardeath pathway indicatesthe release of cytodmrne cfrom mitochondria,which also triggersthe actiYoltion of the G1spase cascade.
[).iring apoptosis,celb undugo DNA fragmentltion, nudear and cell m"11brane breakdown, and are eventIJallydigested by ~er cells. (Reproduad with pt:tmWion from l!JuniaJtrli K.
Andtrsen DK, Biliar TR, et Di: Sd!wattn Prindf*s ofSuigtry, 11th ed. Ntw Witk, N~ McGtaw Hil;}()19.)
6 CHAPTER 1 Cell Biology

Mediates release of calcium from sauoplasmk retkulum in muscle, endoplasmic

reticulum, and mitochondria:
IP3
Works with calcium to activate protein kinase C:
DAG
Enzyme that breaks down ATP to c;AMP with release of pyrophosphate:
Adenylate cyclase
Most critical wmponent in neovasc;ularization in tumor metastases:
VEGF receptor
Cellular proc;ess under the precise wntrol of different extracellular and intracellular
signals and follows a fixed sequenc;e of events leading to c;ell death:
Apoptosis
Steroid hormones bind rec;eptor in:
Cytoplasm
Thyroid hormone binds rec;eptor in:
Nucleus
Examples of c;AMP-dependent hormones:
TSH,ACTH

CELL TRANSPORT
Type of c;ell transport that uses c;onc;entration gradient as a driving fora:
Diffusion (C02, 0 2 , and urea)
Type of diffusion that utilizes a carrier and is saturable:
Facilitated diffusion
Type of c;ell transport that requires ATP for energy:
Active transport

CELLULAR METABOLISM
In glywlysis, I gluoose molec;ule generates:
2 ATP and 2 pyruvate molecules
Name of cycle where NADH and FADHl are created from the 2 pyruvate molec;ules
produc;ed from the breakdown of glucose:
Krebs cycle
Overall number of ATP generated from I molec;ule of gluwse:
38 ATP: 36 from Krebs cycle + 2 ATP from glycolysis
Proc;ess by which amino acids and lactic add via the Cori cycle are wnverted into
gluwse:
Gluconeogenesis
Largest site of gluwneogenesis:
Liver
 Cell Biology CHAPTER 1 1

Muscle RBC
WBC
Protein Nerve
~Glucose
pyruvate Kidney
Muscle

! -
Alanine --- - •
I
I
I

Lactate + Pyruvate

Fatty
acid

Glucose-alanine cycle Cori cycle

FIGURE 1-J. The recycling ofpenpheral lactate and p)TINate for hepatkgluconeogenesl1l1 a<IOm~~hed by the Clrlcyde. Alanlnellfthln skr!lml musclemnalso be used as
a preaJrsor fur hepatic gluconeogenesl~ During star1ation, sudi fatty acid provides fuel soorm fur basal hl'patic enzymatic function. R~ red lfood ml; W&. white blood CEii.
(/leprodU«IJ with petmissionfromBrU1Jiamli FC. Amlffifn DK, l!illiar TR, etri:5chwarrnPrincip/eJof5URpy, 11rlr eri h ~ Nr. M<GtmvHi/l;lOF9J

What can be used for gluconeogenesis?

Yes: lactic acid, amino acids, glycerol
No: free fatty acids, lipids
Name the breakdown product of fat metabolism that cannot be converted back into
pyruvate:
Acetyl CoA
Lipases act on lipids to form:
Fatty acids and monoacylglycerols
Fatty add utilization:
Short chain = direct transport to liver
Long chain = packaged into micelles into lymph

CELL CYCLE
Most variable part of the cell cycle that determines cell cycle length:
Gl
Part of cell cycle where protein synthesis and DNA replication oa:ur:
s
Growth factors affect the cell during this phase of the cell cycle:
Gl
Phase that helps maintain genomic stability:
G2
Part of qcle where cell divides:
M
I CHAPTER 1 Cell Biology

B/CDK1

Mitosis

Fl&URE 1-4. The CEllcyde and i3mntrol system.Mis themitmisphase, >Mien the nudeus and the cytoplasm divide; Sis the phase >Mien D~ ~ duplicrted; Gl ~the gap between
Mand S; G2 ~\lie gap between Sand M. Ammplex of cyclin and cyclin-dependent ~nase (CDKl cootdsspe<ific ev•m~ of ead! phase. 'Mthout cydin, CDK is inactive. llfferent cydinf
CDK cll!lplexes are shCMTI around \lie cen cycle. A. B. D, and Estand fa" cydln A. cyclln B, cydln ll, and cyclln E, respectfvely. (~with ptm1il!/ol1 !mm Btunlfl//rl/ K. Andtrstn
DK,BillituTR,etal:Xhwartz'sPrillcipksofSURJt:ry. 11rhed ~lbtk, Nr. McGtw!Hil/;lOF9J

Tumor cells are most sensitive to radiation during this stage of the cell cycle:
M
Phase of mitosis where chromosomes shorten, nucleolus and nuclear envelope
disappear, and spindle apparatus forms:
Prophase
Phase of mitosis where centromeres align on the equatorial plate, spindle fibers attach
to the centromeres, and centromeres duplicate:
Metaphase
Phase of mitosis where chromatids migrate to opposite poles:
Anaphase
Phase when nucleolar and nuclear envelope re-form and chromosomes decondense:
Telophase

MULTIPLE CHOICE QUESTIONS

1. Erythrocytes use glycolysis primarily as a source of energy in the form of
A. ATP to power active membrane transport
B. ATP to maintain cytoskeleton integrity
C. NADH to power protein synthesis
D. NADPH to initiate DNA replication
E. NADH to reduce oxidized glutathione
 Cell Biology CHAPTER 1 !I

2. A defeci in cholesterol metabolism or other soun:es of bile would uuse difficulties

in digestion because bile is needed for
A. Emulsification of dietary fat for easier access of stomach lipases
B. Denaturation of dietary proteins for easier digestion by proteases
C. Micelle incorporation of lipids for easier digestion by lipases
D. Neutralization of stomach acid
E. Stimulation of pancreatic secretions
3. Cyclins are proteins that serve as signals to control progression of cells around the
cell cycle. Cydin signals are transmitted via
A. Histone acetylases
B. Protein kinases
C. DNA methylases
D. Specific proteases
E. Small interfering RNAs (siRNA}
4. Which of the following is a correct match?
A. G cell-pepsinogen
B. Chief cell-gastrin
C. Parietal cell-HCl and intrinsic factor
D. Mucous cells-cholecystokinin
5. Platelet activation, muscle contraction, pancreatic secretion, and glycogen
degradation act via which intracellular signal mechanism?
A. cAMP second messenger signaling
B. Calmodulin-induced calcium release
C. Protein kinase A activation
D. IP3- and DAG-induced activation of protein kinase C
6. Base deficit and serum lactate correlate with mortality in trauma by reflecting
which of the following systemic changes from normal physiology?
A. Myoglobin-induced ATN progressing to renal failure
B. Hypoperfused end organs relying on energy generated via anaerobic metabolism
C. Skeletal muscle sarcomere-unregulated release of calcium and diacylglycerol
D. Injured organ trauma-induced apoptosis releasing corresponding intravascular
waste cellular products
7. Which of the following clinical scenarios regarding metabolism is false or
implausible?
A. An elderly patient on indomethacin, oxazepam, aspirin, and acetaminophen
becomes jaundiced after overwhelming UDP-glucuronic acid transferase enzymes
B. A 26-year-old female on oral contraceptives conceives after a course of antibiotics
C. A 56-year-old with atrial fibrillation on warfarin is admitted with spontaneous
hematemesis after starting ciprofloxacin/metronidazole therapy for diverticulitis
D. A traumatically injured 38-year-old with no past medical history develops coma
and cerebral infarction from profound hypoglycemia within 30 minutes of injury
10 CHAPTER 1 Cell Biology

8. Whidi medianism explains ultraviolet light as a risk fiu;tor for skin uncers?
A. UV-Blight is absorbed by DNA strands, causing pyrimidine dimers
B. Increased number of melanocytes after prolonged tanning leads to proliferation
errors
C. Vitamin D activation includes free radicals as a side product
D. Sunlight induces collagen breakdown, leading to sheer stress injury
9. Sdect the incorrect statement from the following.
A. Aerobic metabolism provides the most efficient, most proliferative process to
convert glucose into ATP in humans
B. Hepatocyte metabolism of toxins includes cytochrome P-450 enzymes, UDP-
glucuronyl transferases, glutathione S-transferases, and sulfotransferases
C. The entirety of chromosomal DNA is contained within the nucleus in formation
with histone proteins
D. Phase I reactions change endogenous substances' solubility, while phase II
reactions change their chemical structure
10. Whidi of the following is true?
A. The Na/K ATPase transports 3 Na+ in for every 2 K+ out
B. Na+ is the most common intracellular cation
C. Cl- is the most common extracellular anion
D. K+ is high in concentration in the extracellular fluid
11. Once the Golgi apparatus modifies proteins with carbohydrates, it then transports
them to the cdl membrane and they are:
A. Secreted
B. Targeted to lysosomes
C. Both
D. Neither
12. Choose the answer with the correct pairing:
A. Mitochondria: major site of ATP production
B. Krebs cycle: occurs in the nucleus
C. Gluconeogenesis: occurs in the mitochondrial matrix
D. Free fatty acids: substrate for gluconeogenesis
13. Microtubules are involved in:
A. Muscle contraction
B. Transport of organelles
C. Formation of hair and nails
D. Engulfment of particles for degradation
14, Protein kinase A:
A. Is an intracellular enzyme activated by Ca++
B. Hydroxylates enzymes and proteins
C. Is an intracellular enzyme activated by cAMP
D. Is an intracellular enzyme activated by diacylglycerol (DAG)
 Cell Biology CHAPTEl 1 11

15. What is the wrreci action of tight junctions?

A. Allow communication between cells
B. Composed of connexon subunits
C. Include occludin and claudin proteins
D. Stabilize cell-to-cell adhesion and are attached to the actin cytoskeleton

ANSWERS
1. Answer: A. B would be correct if it listed NADPH instead of ATP. C is incorrect
because the cell does not make its own proteins. D is incorrect because RBCs lack
nuclei and therefore do not replicate any DNA. E would be correct if it listed NADPH
instead of NADH.
2. Answer: C. The stomach does not produce lipases. Bile micelle incorporation is not
related to protease activity. Bile does not affect the acidic pH of stomach effluent.
Pancreas secretions are stimulated by hormones, not bile.
3. Answer: B. Histone acetylases and DNA methylases play a role in DNA configuration,
while proteases are not involved in cell messaging. siRNA is part of gene expression,
not directly related to cell messaging.
4. Answer: C. G cell = gastrin, chief cell = pepsinogen, parietal cell = HCl and intrinsic
factor, mucous cell = mucus/bicarbonate.
5. Answer: D. B is incorrect because IP3 binding to endoplasmic reticulum releases
calcium. A and C are incorrect because they belong to the protein kinase A system.
6. Answer: B. Lactate production is associated with hypoperfusion in trauma.
Myoglobin can cause ATN or renal failure in trauma but should not directly alter BD
or lactate. Sarcomere release of calcium is implicated in malignant hyperthermia.
7. Answer: D. D is the incorrect scenario. Glycogen stores can supply the necessary
glucose for anaerobic metabolism even in intense need for 20 to 90 minutes, after
which it is depleted; thus, anaerobic metabolism would attempt to meet the needs of
the patient in scenario D.
8. Answer: A. B is incorrect because melanocyte number is constant as part of neural
crest migration as an embryo. C is incorrect because free radicals are not involved. D
is incorrect because collagen is unrelated to DNA sequence.
9. Answer: D. Phase I reactions change chemical structure, while phase II reactions
change solubility.
10. Answer: C. The Na/K ATPase transports 3 Na+ out for every 2 K+ in. K+ is the
most common intracellular cation. K + is low in concentration in the extracellular
fluid (4 mEq/L).
11. Answer: C. The proteins are secreted or targeted to lysosomes.
12. Answer: A. Mitochondria are the major site for ATP production. The Krebs cycle
occurs in the mitochondrial matrix. Gluconeogenesis occurs in the cytoplasm.
Free fatty acids cannot be used for gluconeogenesis because acetyl CoA cannot be
converted back to pyruvate.
13. Answer: B. Microtubules are involved in the formation of specialized cell structures
such as cilia and axons, transport of organelles within the cell, and cell division. Actin
and myosin are involved in muscle contraction. Intermediate filaments such as keratin
12 CHAPTER 1 Cell Biology

are involved in the formation of hair and nails. Phagosomes and endosomes engulf
particles for degradation.
14. Answer: C. Protein kinase A is an intracellular enzyme activated by cAMP. It
phosphorylates other enzymes and proteins, not hydroxylates. Protein kinase C is
activated by Ca++ and DAG.
15. Answer: C. Gap junctions allow communication between cells and are made up of2
connexon subunits. Tight junctions include proteins such as occludin and claudin and
form a water-impermeable barrier between cells. Adherens junctions stabilize cell-to-
cell adhesion and are attached to the actin cytoskeleton.
CHAPTER 2
Hematology
Christine Du

Test Taking Tip

Hematology requires lots of memorization. Important topics to look over include bleeding disorders and the various anticoagu-
lants.These are basic questions you don't want to miss.

THE COAGULATION PATHWAY

Sequence of the intrinsic pathway of coagulation:
Prekallikrein + HMW kininogen + factor XII + exposed collagen - activates factor
XI - activates factor IX, combines with factor VIII - activates factor X, combines
with factor V - t converts prothrombin (factor II) into thrombin. Thrombin converts
fibrinogen into fibrin.

I ~nae
Vascular Injury
I
~ Surface Inflammation
Complement activation
~FactorXlla
i
nssue factor +
Kalllkrem .,..__ Prekallikrein
Fibrinolysis

HMW klninogen ? Physiologic

factor VII
Surface

Tissue factor-Factor VIia

FactorX

Prothrombin - - - - - - - • .. /Factor XIII

(factor II)
I Ca2 ... Fibrin
Flbrinogen ~I Fibrin
FactorXllla +
X-Linked fibrin

FIGURU·1. Sdifmallc oldie coagulallonsystem. HMI'( high maecular weight (RtplOl/lm! wnh ptl/IJb:!/on flom Bllinlaldl FC, MdMtn DK, Bllllar 1/1, tt tt 5dnw1rrn ~
ofS<itrpy. 9rhtd. NewYork. NY: Mr.<ilrrwllil;201(})
14 CHAPTER 2 Hematology

Sequence of the extrinsic pathway of ooagulation:

Factor VII + tissue factor - t activates factor X, combines with factor V -t converts
prothrombin into thrombin. Thrombin converts fibrinogen into fibrin.
Whiclt factor is the oonvergence point and oommon to both the extrinsic and intrinsic
pathways of coagulation?
Factor X
What does the prothrombin oomplex oonsist of?
Factor V. X, platelet factor 3, and prothrombin catalyze the formation of thrombin
What function does thrombin have?
Activates factors V and VIII, activates platelets, and converts fibrinogen into fibrin and
fibrin split products
Whiclt factor has the shortest half-life?
Factor VII
What factor can be used to differentiate a oonsumptive ooagulopathy from
hepatoc;:ellular disease?
Factor VIII:C; consumptive coagulopathy will have reduced levels of all factors, and
hepatocellular disease will have reduced levels of all factors except factor VIII
Whiclt factors are known as the labile factors (activity lost in stored blood)?
Factors V and VIII
What function does factor XIII have?
Cross-links fibrin
What does protein C do?
Degrades fibrinogen and factors V and VIII (vitamin K dependent)
What does protein S do?
Acts as protein C cofactor (vitamin K dependent)
What does Von Willebrand factor (vWF) do?
Links collagen to the Gplb receptor on platelets
What is the function of antithrombin III?
Binds heparin, inhibits factors IX, X. XI, and thrombin
Where does tissue plasminogen activator oome from, and what does it do?
Released from endothelium, and it converts plasminogen into plasmin
What does plasmin do?
Degrades fibrinogen, fibrin, and factors V and VIII
What is the natural inhibitor of plasmin called, and where does it come from?
Alpha-2 antiplasmin; comes from the endothelium
What are the vitamin K-dependent factors?
Factors II, VII, IX, and X and proteins C and S
What function does tissue factor pathway inhibitor have?
Inhibits factor X
 liematclogy CHAPTER 2 15

LABORATORYTE.STS AND DATA

PT measures the function of these factors:
Factors II, V, VII, and X and Dbrinogen
What 2 factors are not measured by the PTT?
Factors VIl and XIII
PTT measures the function of these factors:
Factors II, V, VIII, IX, X. XI, and XII and fibrinogen
What is the normal value for bleeding time, and what does it imply?
Normal bleeding time ranges from 3 to 9 minutes and implies platelet counts >50,000/mL
and normal platelet function
What test aids in detecting circulating anticoagulants, qualitative abnormalities of
fibrin, inlu'bition of fibrin polymerization, and measures the clotting time of plasma?
Thrombin time
Patients bleeding after a large number of blood transfusions should be considered
to have:
Dilutional thrombocytopenia (vs. hemolytic transfusion reaction)
What factors are common to both PT and PTTt
Factors :o:. V. and X and :ftbrinogen

1. vascular phase 2. Platelet phase

(Vasoconstrlctlon) (Platele1e aggrngate)
Common pathway
I Intrinsic pathway I Plothrombln I Extrinsic pathway I
~
~~~~
Clotting factors
Thrombin
@
Clotting factors
VIII, IX, X, XI, XII I VII
Fibrin

3. Coagulation phase (Clot formation)

(Clot rnlmctlon) 4. Flbrfnolysle (Clot destruction)

R&llll.!-J. l!iolo;yafr.mosmThe4~ic~~i~tolinitblocldlassfrim1nirjumlvmd11'illlllmll<lldindl.lll!Vil0.11rn.mri<lion.~
plugflmln!m,~m d(lfirrniltliJl,~dftbllnrtfi15. ~WilflparrJ!rlalm~K.~mOK,Bll/Jat 111,tttt Sdill'MrtfltldJ*sd~ll!lltd 111.w llJ/t/IY:
Mt.<irllwlfl;101a)
16 CHAPTER 2 Hematology

How many hours must elapse after the last dose of IV heparin before the PT <:an be
reliably measured?
Minimum of 5 hours
Sequence of physiologic reactions that mediate hemostasis following vascular injury:
1. Vasoconstriction
2. Platelet activation/adherence/aggregation
3. Thrombin generation

HYPERCOACiULABILITY DISORDERS
What is Virchow triad?
Stasis, endothelial injury, and hypercoagulability
What is the most common cause of acquired hypercoagulability?
Smoking
What is the most common inherited hypercoagulable state?
Factor V Leiden
What is the treatment for hyperhomocysteinemia?
Vitamin B-12 and folate
Name the prothrombin gene defect causing spontaneous venous thrombosis:
Prothrombin gene defect G20210A

PLATELET FUNCTION AND DYSFUNCTION

What is the normal life span of a platelet?
7to10 days
Formation of a platelet plug requires these 2 electrolytes:
Calcium and magnesium

Vascular endothelial
l,..ury

Platelet hemostatic Vasoconstriction

function
Subendothellal collagen

.... Coagulation activation

Platelet adhesion secretion via tissue factor-
factor VIia
(Reversible) .... ADP, serotonin,
Ca2+, flbrinogen
Platelet aggregation secretion i
IXa,Xa
(Irreversible) .... ADP. serotonin, Complexes on
Ca2.., ffbrinogen activated platelets

FIGUIU-J. Sdlematicofplab!letactivatim and thrombus

functim. ADP. adenosine diphoiphate. (llqxoduC«I with
Platelet aggregation
.... i
Thrombin
pmnJss/on lirlm IITunkvrdl F(, Andmtn DK, BJl1lar Ill, et oJ:
5d!WllM PrindpltJ of5wyey, 9lh ed ~ YotA; N~ Mdimw
Platelet-fibrin +
thrombus -------Fibrinogen
ff81; 2010)
Another random document with
no related content on Scribd:
 Inhoudsopgave

VOORWOORD. VII
GEBEZIGDE LITTERATUUR. XI
INHOUD. XIII
LIJST VAN ILLUSTRATIES. XV
VERBETERINGEN. XVI
I. INDIANEN-BEVOLKING VAN WEST-INDIË. 1
Inleidende beschouwingen. 1
Inhoud der verhalen 7
Lijst der in dezen bundel opgenomen mondelinge
overleveringen der Indianen. 14
Indianen-Vertellingen. 66
1. De sage van Haboeri (W.) 66
2. De oorsprong der eerste menschen (C.) 73
3. De oorsprong van het menschdom (W.) 75
4. De oorsprong der Caraïben. (C.) 76
5. Hoe de Caraïben gekweekte planten leerden
kennen. (C.) 77
6. De dochter van den geestenbezweerder. 79
7. Hoe lichaamspijnen, dood en ellende in de wereld
kwamen. (C.) 81
8. Het hoofd van den Boschgeest en de nachtzwaluw.
(A.) 83
9. De vrouw, die een Boschgeest nabootste. (A.) 84
10. De Geest van een schimmelplant* redt een 86
Indiaansch meisje. (C.)
11. Een jagoear, die in een vrouw veranderde. (A.) 89
12. De man met een Baboen-vrouw. (A.) 91
13. Schildpad, die Boschrat er in liet loopen. (C.) 94
14. De bedrieger bedrogen. (C.) 95
15. Tijger en Miereneter. (C.) 96
16. Hariwali en de Wonderboom. (A.) 98
17. De legende van den Ouden man’s val. 103
18. Amanna en haar praatzieke man. (C.) 105
19. De zon en zijn beide tweelingzoons. (C.) 107
20. De Legende van den Vleermuis-berg. (M.) 111
21. De Uil en zijn schoonbroeders vleermuis. (W.) 112
22. De Lichtkever en de verdwaalde Jager. (C.) 114
23. De bina, de weder in het leven geroepen vader en de
slechte vrouw. (W.) 116
24. Hoe een jong Warrau-Indiaantje uit de handen der
Caraïben ontkwam. (W.) 119
25. Sluit de oogen en doe een wensch. (C.) 121
26. De gelukspot. (W.) 122
27. De honigbij en de zoete drank. (W.) 124
28. De piaiman en de stinkvogels*. (A.) 125
29. Hoe het ongeluk over de menschen kwam. De
geschiedenis van Maconaura en Anoeannaïtoe. (A.) 131
30. De kolibri, die tabak brengt aan den eersten piaiman.
(W.) 140
31. Het ontstaan der vrouwennaties. 145
32. Het gebroken ei. 146
33. De geest van den pasgeborene. 146
34. De huid van den Reuzenslang of Hoe de vogels hun 147
 tegenwoordig gevederte kregen.
35. Een waarschuwing voor de vrouwen. (A.) 148
36. Hoe een man van zijn luiheid genezen werd. (W.) 155
37. Zwarte Tijger, Wau-oeta en de gebroken boog. (W.) 157
38. De Legende van Letterhoutstomp. 162
39. De Legende van Arimoribo en Jorobodie. (C.) 165
40. Uitdrijven van een priester uit den Indiaanschen
hemel. 170
41. Uitdrijving der Indianen uit den Hemel der Paters. 174
42. Bezoek van Caraïben aan Macoesiland. (C.) 178
43. Legende van Paramaribo. 179
44. De Legende van Post Sommelsdijk. 180
45. Einde van den Indiaanschen broederoorlog. (A.) 182
46. De groote bloedzuigende vleermuis. (A.) 183
47. Legende van Mapajawari of de uitroeiing der
menscheneters. (C.) 184
48. Migratie-legende van den Kasi’hta-stam der Creek-
Indianen, 189
II. West-Indische neger-folklore. 197
Inleidende beschouwingen. 197
De Surinaamsche Anansi-tori’s en hare oorsprong. 203
LIJST DER NEGERVERTELLINGEN. 235
Inhoud der Surinaamsche Negervertellingen. 237
De anansi-tori en het bijgeloof. 246
VERTELLINGEN DER SURINAAMSCHE
STADSNEGERS. 258
1. Anansi, die een half dorp verovert. 258
2. Spin en de Prinses. 266
3. Het huwelijk van Heer Spin. 271
4. Anansi, Tijger en de doode Koe. 273
 5. Anansi en zijn kinderen. 276
6. Hoe Spin zijn schuldeischers betaalt. 277
7. Een feest bij de Waternimf. 281
8. Anansi en Kat. 282
9. Spin en Krekel. 285
10. Heer Spin als Geestelijke. 286
11. Heer Spin als roeier. 287
12. Spin neemt Tijger gevangen. 289
13. Heer Spin en Hond. 291
14. Tijger’s verjaardag. 293
15. Spin voert den Dood in. 295
16. Spin wedt, Tijger te berijden. 297
17. Verhaal uit het leven van vriend Spin. 299
18. Anansi als Amerikaan verkleed. 303
19. Heer Spin en de Waternimf. 305
20. Anansi, Hert en Kikvorsch. 306
21. Heer Spin als landbouwer. 308
22. Anansi en de Bliksem. 310
23. Ieder volwassen man moet een rood zitvlak hebben. 315
24. Hoe Anansi aan schapenvleesch wist te komen. 318
25. De geschiedenis van Fini Foetoe, Bigi bere en Bigi
hede. 321
26. Legende van Leisah I. 323
27. Legende van Leisah. II. 325
28. Verhaal van het land van „Moeder Soemba”. 327
29. Boen no habi tangi. 330
30. Geschiedenis van Kopro Kanon*. 332
31. De Meermin of Watramama. 335
32. De Boa in de gedaante van een schoonen jongeling. 337
 33. Het huwelijk van Aap. 339
DE ANANSI-TORI DER SURINAAMSCHE
BOSCHNEGERS. 342
Hoe Heer Spin door zijn bekwaamheid als
geneesheer de mooie dochter van den Landvoogd
wist te krijgen. 345
NEGER-VERTELLINGEN UIT HET WEST-INDISCHE
EILANDENGEBIED. 350
Curaçaosche Negervertellingen. Cuenta di Nansi. 350
Nansi en Temekóe-Temebè. 354
Creoolsche folk-lore van St.-Eustatius. 360
Braha- Nanci en Braha-Toekema. 362
Neger-vertellingen van Jamaica. Nancy-Stories. 367
1. Annancy in Krabbenland. 371
2. Reiger. 373
3. Annancy, Poes en Rat. 377
BIJVOEGSELS. 379
I. NEGER-SPREEKWOORDEN. 379
Suriname. 379
West-Afrika. 381
II. AVOND OP HET WATER in Sierra Leone 384
Spin, Olifant en Hippopotamus. 389
III. DIEREN-FABEL, 393
Wie zijn Krokodil’s verwanten? 393
VERKLAREND REGISTER. 396
A. 396
B. 397
C. 399
D. 400
E. 400
F. 400
G. 401
H. 401
I. 402
J. 402
K. 403
L. 406
M. 407
N. 408
O. 408
P. 409
R. 411
S. 411
T. 413
V. 414
W. 415
Y. 416
Z. 416
 Colofon
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Metadata

Titel: Mythen en
sagen uit
West-Indië
Auteur: Herman Info https://viaf.org/viaf/45474713/
van
Cappelle
Jr. (1857–
1932)
Illustrator: Willem Info
Antonius https://viaf.org/viaf/3295167202597667930008/
Josef
Backer
(1901–
1971)
Aanmaakdatum 2023-11-14
bestand: 20:38:53
UTC
Taal: Nederlands
(Spelling
 De Vries-
Te Winkel)
Oorspronkelijke 1926
uitgiftedatum:

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De volgende verbeteringen zijn aangebracht in de tekst:

Bladzijde Bron Verbetering Bewerkingsafstand

n.v.t. GEILLUSTREERD GEÏLLUSTREERD 1/0
VII symphatieke sympathieke 2
XII, 165, [Niet in bron] ) 1
243, 408
XII, 400,
407 . [Verwijderd] 1
Passim. [Niet in bron] . 1
XII Volkenkunden Volkenkunde 1
XII Smithonian Smithsonian 1
XII reports report 1
XIII, 350,
350 St. Eustachius St.-Eustatius 3
XVI wreedzaam wreedzame 2
XVI vreedzaam vreedzame 2
XVI, 26,
46, 58,
158, 203,
253, 276,
287, 328,
356, 403,
405, 406,
410, 410,
412 , . 1
2 wreedzame vreedzame 1
4, 76, 76 wêer weêr 2/0
5 hebben heb 3
7 nêergelegd neêrgelegd 2/0
7 geïdialiseerd geïdealiseerd 1
8 Mythen-cathegorie Mythen-categorie 1
9, 207 beinvloed beïnvloed 1/0
11 met [Verwijderd] 4
12 beinvloeden beïnvloeden 1/0
13 Travells Travels 1
14, 92,
119, 222,
276, 287,
329, 355 — 1
17 af komstig afkomstig 1
17 voor voort 1
20, 165,
214, 214,
243, 334 . , 1
24 lichamspijnen lichaamspijnen 1
25 Anoeannaitoe Anoeannaïtoe 1/0
28 verweringskorst verweeringskorst 1
28 Indiaaan Indiaan 1
29, 409 over dag overdag 1
29, 164,
165 Caraiben Caraïben 1/0
31 demaan de maan 1
Passim. [Niet in bron] „ 1
Passim. Penard’s Penards 1
33 Caraibische Caraïbische 1/0
33 de de de 3
33 heuvel hemel 2
33 19 20 2
34 van daan vandaan 1
39 hebben heeft 4
40, 106,
106, 122 broer broêr 1/0
Passim. [Niet in bron] ” 1
41 jaguar jagoear 2
44 ,* *, 2
45 kalabassen kalebassen 1
47 Indíaansche Indiaansche 1/0
54 Missisippi-stroom Mississippi-stroom 1
55, 189 Ogleterpe Oglethorpe 2
55 Tchikili Tchikilli 1
56 mais maïs 1/0
60 Tot In 3
61 bij hij 1
62 Natuur Natur 1
63, 72 papagaai papegaai 1
66 Pallissade-palm Palissadepalm 2
69 antwoorde antwoordde 1
72 Onmiddelijk Onmiddellijk 1
74 gopend geopend 1
76 weér weêr 1/0
78, 125,
183, 184,
229, 264,
283, 294,
313, 321,
332, 336,
354 .* *. 2
78 lotgenoten lotgenooten 1
78 Allepäntepo Allepántepo 1/0
81 Jorokas Joroka’s 1
81, 117,
128, 233,
410 , [Verwijderd] 1
84 ”. .” 2
84, 148,
314, 314 mee meê 1/0
86 broers broêrs 1/0
96 lachtte lachte 1
97 Tamanoe Tamanoea 1
112 schoonbroeder schoonbroeders 1
112 broêr broêrs 1
114 schoonbroers schoonbroêrs 1/0
117 vermoordde vermoorde 1
121 hadden had 3
122 verweringsprodukten verweeringsprodukten 1
123 çassave-gerecht cassavegerecht 2/1
124, 364,
391 „ [Verwijderd] 1
127 van een van een van een 8
128 weer weêr 1/0
131 Agouti Agoeti 1
133, 141 schreidde schreide 1
133 Anoennaïtoe Anoeannaïtoe 1
134 uw Uw 1
135 Kakoutji Kaikoutji 1
137 wraken wrake 1
139 Anoeannitoe Anoeannaïtoe 2/1
142, 142 hem hen 1
142 zij hij 1
142 zouden zou 3
150 probeerden probeerde 1
151 weefpatroon vlechtpatroon 5
157 Tobe-heroanna Tobe-horoanna 1
158, 255,
264, 269,
269, 276,
376 [Niet in bron] , 1
158 tijger Tijger 1
159 ”* *” 2
162 niet [Verwijderd] 5
163, 164 Caraibisch Caraïbisch 1/0
164 Marorwijne Marowijne 1
164 Corantyn Corantijn 2
164 Marorvijne Marowijne 2
166 slotten slotte 1
170, 340 ” [Verwijderd] 1
183 Nu nu 1
184 uitroeiïng uitroeiing 1/0
185 zijn’s zijn 2
188 vluchtten vluchten 1
189 kanibalen kannibalen 1
189 achtiende achttiende 1
189 Kanibalenstam Kannibalenstam 1
189 Kanibalen Kannibalen 1
189 Giorgia Georgia 1
196 Ogletherpe Oglethorpe 1
198 dède hóso déde-hóso 2/1
201 negervertelingen negervertellingen 1
202 - [Verwijderd] 1
207 Neger-engelsche Neger-Engelsche 1
210 [Niet in bron] in 3
212 lostte loste 1
212, 239 Jamaïca Jamaica 1/0
220 Boesi-gramman Boesi-granman 1
229 Python’s Pythons 1
229 synonym synoniem 2
230 Aequtoriaal Aequatoriaal 1
231 Rabit Rabbit 1
233 Sierre Sierra 1
236 Temekoe-Temebe Temekóe-Temebè 2/0
239 Awarì-bang Awari-bángi 3/1
240 Negerengelsch Neger-Engelsch 2
243 Je Je 0
244 von van 1
246 lachtten lachten 1
246 anansi tori’s anansi-tori’s 1
247 kreool Creool 1
247 déde hóso déde-hóso 1
251 déde-hoso déde-hóso 1/0
251 arratta aratta 1
252 wachvrouw waschvrouw 1
252 Idiaansche Indiaansche 1
253, 296,
363 neer neêr 1/0
254 abormalen abnormalen 1
256, 256,
256 u U 1
267, 268 Jou Jouw 1
268, 277, jou jouw 1
284
269 „ ” 2
269 ” ,„ 2
272 anansi’s Anansi’s 1
276 Ma’ ’Ma 2
276 julie jullie 1
278 [Niet in bron] Vos 4
282, 295 anansi Anansi 1
282 anansì Anansi 2/1
282 Pína Pina 1/0
296, 304,
310, 311,
312, 312,
314, 314 Anansí Anansi 1/0
299, 307 anansí Anansi 2/1
303 broeders Broeders 1
303 zusters Zusters 1
303 Kosi Kósi 1/0
303 [Niet in bron] ,” 2
307, 338 wordt word 1
310, 310 10.000 10,000 1
311 . : 1
311 ! ? 1
313 buikpuin buikpijn 2
314 . ? 1
319 korsten kortsten 1
320 huigelachtige huichelachtige 2
322 broertjes broêrtjes 1/0
324 moeder Moeder 1
324 Vader’s Vaders 1
324 vaders Vaders 1
326 schuim Schuim 1
329, 329 Ma ’Ma 1
331 uit uit uit 4
332 ! : 1
333, 334,
334 Minimini Mininimi 2
334 patient patiënt 1/0
334 Kapro Kopro 1
338 wij Wij 1
338 er Er 1
340 gebruike gebruikelijke 5
344 Hoevell Hoëvell 1/0
345 spin-vertelling spinvertelling 1
348 verergde verergerde 2
348 hoelanger hoe langer 1
349 Spin’s Spins 1
350 voornaamsche voornaamste 2
350 dagens dagen 1
354 papiemento papiamento 1
357 guyave guave 1
365 St. Eustatius St.-Eustatius 1
370 kamplaats kampplaats 1
370 banencultuur bananencultuur 2
373 vrienden vriend 2
376 hollandsche Hollandsche 1
379 zachtzinige zachtzinnige 1
380 Iffi iffi 1
381 Houd Houdt 1
381 okro okra 1
381, 381 houd houdt 1
383 zend zendt 1
386 melodiën melodieën 1
390 houdt houd 1
394, 394 veèren veêren 1/0
396 Neger-engelsch Neger-Engelsch 1
396 Amalavica Amalivaca 2
399 [Niet in bron] : 1
399 Eunctus Eunectus 1
399 Piaaiman Piaiman 1
400 Mrs Mrs. 1
400 Dokoen Dokóen 1/0
400 Dokoenboom Dokóenboom 1/0
401 NE. N.E. 1
401 donkerebruinen donkerbruinen 1
402 Auruba Aruba 1
402 Dominico Dominica 1
402 Onca onca 1
403 Anacardicum Anacardium 1
405 Kopra Kanon Kópro Kanón 3/1
405 ,) ), 2
405, 416 [Niet in bron] - 1
406 kankantree kankantrie 1
407 Maconoura Maconaura 1
407 waarmêe waarmeê 2/0
408 Massoewa Masoewa 1
408 Winnamoeroe Winnamoroe 1
409 Ooloekwa-toelala Oeloekwa-toelala 1
409 [Niet in bron] ( 1
409 Okro Okra 1
409 ( [Verwijderd] 1
409 ) [Verwijderd] 1
410 Ipomaea Batatas Ipomoea batatas 2
413 [Niet in bron] L. 3
414 Verweerings-korst Verweeringskorst 1
414 Zuid-America Zuid-Amerika 1
415 Blondii blondii 1
415, 416 N.-E. N.E. 1
416 Magroveboomen Mangroveboomen 1

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