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Hafiza Report
Hafiza Report
agilus»
diagnostics
PATIENT NAME HAFIZA BEGUM REF, DOCTOR : DR, RATHINDRA NATH BISWAS
coDE/NAME & ADDRESS IC000111042 ACCESSION NO :0269XD000334 |AGE/SEx :48 Years Female
sURE & CURE DIAGNOSTICS |PATILNT D HAFIM2405750 DRAWN :04/04/2024 12:56:53
ALARING NO, 2440, PANDUA SIATION BAZAR, CLIINT PATIENTID: RECEIVED :04/04/2024 16:15:48
POST OFICE LANE,P,S, PANDUA, REPORTED :04/04/2024 19:48:18
HOOGHLY 712149 ABHA N0
9434562382
HAEMATOLOGY - CBC
Page 1 Of 8
agilus>
diagnostics
PATIENT NANE HAFIZA BEGUM REF. DOCTOR: DR, RATHINDRA NATH BISWAS
CODE /NANE & ADDRESS :CO00111042 ACCESSION NO:0269XD000334 AGE/SEX :48 Years Fernale
SURE & CURE DIAGNOSTICS 0PATIENT ID HAFIM2405750 DRAWN 04/04/2024 12:56:53
BEARING NO, 2440, PANDUA STATION BAZAR, cUENT PATIENT ID: RECEIVED 04/04/2024 16:15:48
POST OFFICE LANE,P.S. PANDUA,
ABHA NO REPORTED :04/04/2024 19:48: 18
HOOGHLY 712149
9434562382
Page 2 of 8
agilus>»
diagnostics
PATIENT NAME: HAFIZA BEGUM REF, DOCTOR: DR, RATHINDRA NATH BISWAS
CoDE/NAME &ADDRESS :CO00111042 IAGE/SEX 48 Years Female
ACCESSION NO :0269XD000334
SURE & CURE DIAGNOSTICS
BEARINGNO, 2440, PANDUA STATION BAZAR, 0PATIENT ID :HAFIM2405750 DRAWN :04/04/2024 12:56:53
POST OFFCE LANE,P.S. PANDUA, CLIENT PATIENT ID: RECEIVED :04/04/2024 16:15:48
HOOGHLY 712149 ABHANO IREPORTED :04/04/2024 19:48:18
9434562382
HAEMATOLOGY
CBC WITH ESR (CBC+PS+ESR) EDTA WHOLE BLOOD/SMEAR
ERYTHROCYTE SEDIMENTATION RATE (ESR),EDTA
BLOOD
E.S.R 45 High 0 - 20 mm at 1 hr
METHOD:MODIFIED WESTERGREN
Interpretation(s)
ERYTHROCYTE SEDIMENTATION RATE (ESR),EDTA BLOOD-TEST DESCRIPTION:
Erythrocyte sedimentatlon rate (ESR) Is a test that Indirectly measures the degree of inflammation present in the body. The test actually measures the rate of fall
(sedimentation) of erythrocytes in a sample of blood that has been placed into a tall, thin, vertical tube. Results are reported as the millimetres of clear fluid (plasma) that
are present at the top portion of the tube after one hour, Nowadays fully automated instruments are available to mcesure ESR.
ESR IS not diegnostic
inflammatory it isa isnon-specific
condition,CRP superior to test
ESR that may itbeis elevated
because in anumber
more sensitive of different
and reflects a moreconditions. It provides
rapid change. general information about the presence of an
TEST INTERPRETATION
Increase in: Infections, Vasculities, Inflammatory arthritis, Renal disease, Anemia, Malignancies and plasma cell dyscrasias, Acute allergy Tissue injury, Pregnancy,
Estrogen mecication, Aging.
Finding a very accelerated ESR{>100 mnm/hour) in patients with ill-defined symptoms directs the physician to search for a systemic disease (Paraproteinemias,
Disseminated malignancies, connective tissue disease, severe infections such as bacterial endocarditis).
in pregnancyBRI in first trimester is D-48 mm/hr(62 if anemic) and in second trimester (0-70 mm fhr(95 if anemic), ESR returns to nomal 4th week post partum.
Decreased in: Potycythermia vera, Sickle cell anemia
LIMITATIONs
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agilus> dagnostics
PATIENT NAME: HAFIZA BEGUM REF. D0CTOR : DR, RATHINDRA NATH BISWAS
CODE/NAME &ADDRESS :C000111042
ACCESSION NO :0269XD000334 AGE/SEX 48 Years Fernale
SURE & CURE DIAGNOSTICS
0PATIENT ID HAFIM2405750 DRAWN .04/04/2024 12:56:53
BEARING NO, 2440, PANDUA STATION BAZAR,
POST OFFICE LANE,P.S. PANDUA, CUENT PATIENT ID: RECEIVED :04/04/2024 16.15:48
HOOGHLY 712149 |ABHA NO !REPORTED :04/04/2024 19:48:18
9434562382
BIOCHEMISTRY
VER EUNCTIONLPROFILE, SERUM
Interpretation(s)
LIVER FUNCTION PROFILE, SERUM
Bilirubin is a yellowish pigment found in bile and is a breakdown product of normal heme catabollsm. Bilirubin is excreted in bile and urine, and elevated levels may give
yeliow discoloration in jaundice.Elevated levels results from increased bilirubin production (eg, hemolysis and ineffective erythropoiesis), decreased biirubin excretion (eg,
obstruction and hepatitis), and abnormal bilrubin metabolsm (eg, hereditary and neonatal jaundice). Conjugated (direct) bilirubin is elevated more than unconjugated
(indirect) bilirubin in Viral hepatitis, Drug reactions, Alcohollc lver disease Conjugated (direct) billrubin is also elevated more than unconjugated (indirect) bilirubin when
there is some kind of biockage af the bile ducts like in Galstones geting into the bile ducts, tumors &Scaring of the bile ducts. Increased unconjugated (indirect) bilirubin
may be a resut of Hemolytic or pernicious anemia, Transfusion reaction &a common metabolic condition termed Gilbert syndrome, due to low levels of the enzyme that
attaches sugar molecules to bilirubin,
AST is an enzyme found in varnous parts of the body. AST is found in the liver, heart, skeletal muscle, kidneys, brain, and red blood cells, and it is commonly measured
dinically as a marker for liver health, AST levels increase during chronic viral hepatitis, blockage of the bile duct, cirrhosis of the liver,liver cancer,kidney failure,hemoytic
anemia,pancreatitis,hemochrofmatosis. AST levels may also increase after a heart attack or strenuous activity.ALT test measures the amount of this enzyme in the blood,ALT
is found mainly in the iver, but also in smaller amounts in the kidneys, heart,muscles, and pancreas.It is commonly measured as a part of a diagnastic evaluation of
hepatocellular injury, to deterrn1ne Iver health.AST levels increase during acute hepatitis,sometimes due to a viral infection,ischemia to the liver.chronic
hepatitis, obstruction of bile ducts,cirrhosis.
ALP is a tein found in almost allbody tissues.Tissues with higher amounts of ALP include the liver, bile ducts and bone.Elevated ALP levels are seen in Biliary obstruction,
Osteoblastic bone tumors,osteomalacia, hepattis, Hyperparathyroidism,Leukemia, Lymphoma, Pagets disease, Rickets, Sarcoidosis etc. Lower-than-normal ALP levels seen
in Hypophosphatasia, Malnutrition,Protein defldency, Wllsons disease.
GGT is an enzyme found in cetl membranes of many tissues mainly in the liver,kidney and pancreas.It is also found in other tissues including intestine,spleen, heart, brain
and seminal vesicles.The highest concentration is in the kidney, but the liver is considered the source of normal enzyme activity.Serum GGT has been widely used as an
index of liver dysfunction.Elevated serum GGT sctivity can be found in diseases of the lver,llary system and pancreas.Conditions that increase serum GGT are otbstructive
liver disease,high aicohol consumption and use of enzyme-inducing drugs etc.
Total Protein aso known as total protein1, is a biochernical test for measunng the total arnount of proteirn in serum.Protein in the plasma is made up of albumin and
agilus>> diagnostics
globulin.Higher-than-normal levels may be due to cinflammation or infection,including HIV and hepatitis Bor C, Multiple myeloma,
disease.Lower-than -nomal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage).Burns,Glomerulonephritis,Liver disease, Malabsorption, Malnutritlon,Nephrotic
syndrome, Protein-losing enteropathy etc.
Albumin is the most abundant protein in human blood plasma.It is produced in the llver.Albumin constitutes about half of the blood serum proteln.Low blood albumin levels
(hypoalbuminemia)can be caused by:Liver disease ike cirrhosls of the liver, nephrotic syndrome,protein-losing enteropathy, Burns,hemodilution,increased vascular
permeability or decreassed lymphatic clearance, malnutrition and wasting etc
agilus>» diagnostics
BIOCHEMISTRY - LIPID
Page 6 Of 8
agilus>» dragnostics
PATIENT NAME: HAFIZA BEGUM REF. D0CTOR : DP, RATHINDRA NATH BrsWAS
cCODE/NAME & ADDRESS :C000111042 ACCESSION NO : 0269XDO00334 AGE/SEX 48 Years Female
sURE & CURE DIAGNOSTICs DRAWN 04/04/2024 12:56:53
BEARING NO. 2440, PANDUA STATION BAZAR, PATIENT ID HAFIM2405750
POST OFICE LANE,P.S. PANDUA, CUENT PATIENT ID: RECEIVED 04/04/2024 16:15:48
HOOGHLY 712149 ABHA NO REPORTED :04/04/2024 19:48:18
9434562382
CHOLESTEROL, TOTAL
339
271.2
Dordertinetigh
203.4
128 152
135.6
desirable
67.8
Interpretation(s)
Serum lipid profile is measured for cardiovascular risk prediction. Lipid Association of India recommends LDL-C as primary target and Non
HDL-C as co-primary treatment target.
Risk Stratification for ASCVD (Atheroselerotic cardiovascular disease) by Lipid Association of India
Risk Category
Extreme risk group A.CAD with > 1feature of high risk group
B. CAD with > 1feature of Very high risk group or recurrent ACS (within I year) despite LDL-C <or =
S0 mg/dl or polyvascular disease
Very High Risk 1. Established ASCVD 2. Diabetes with 2 major risk factors or evidence of end organ damage 3
Familial Homozygous Hypercholesterolemia
High Risk I. Three major ASCVID risk factors. 2. Diabetes with I major risk factor or no evidence of end organ
damage. 3. CKD stage 3B or 4. 4. LDL >190 mg/dl 5. Extreme of asingle risk factor. 6. Coronary
Arery Calcium - CAC >300 AU. 7. Lipoprotein a>= S0mgldl 8. Non stenotic carotid plaque
Moderate Risk 2 major ASCVD risk factors
Low Risk 0- major ASCVD risk factors
Major ASCVD (Atherosclerotic cardiovascular disease) Risk Factors
. Age > or 45 years in males and > or =55 years in females 3. Current Cigarette smoking or tobacco use
2. Family history of prematureASCVD 4. High blood pressure
5. Low HDL
Newer treatment goals and statin initiation thresholds basedon the risk eategories proposed by LAl in2020.
Risk Group Treatment Goals Consider Drug Therapy
Page 7 Of 8
agilus>» diagnostics
PATIENT NAME: HAFIZA BEGUM REF. D0CTOR:DR, RA THINDRA NATH BISWAS
CODE /NAME & ADDRESS : CO00111042 |ACCESSION NO :0269XD000334 | AGE/SEX 48 Years Fernale
SURE & CURE DIAGNOSTICS
PATTENT ID HAFIM2405750 DRAWN 04/04/2024 12:56:53
BEARING NO. 2440, PANDUA STATION BAZAR,
CUENT PATIENT ID: RECEIVED :04/04/2024 16:15:48
POST OFFICE LANE,P.S. PANDUA,
HOOGHLY 712149 ABHA NO REPORTED :04/04/2024 19: 48:18
9434562382
**End Of Report**
Please vist www.agilusdiagnasttcs.com for related Test Information for this accession
Page 8 Of 8
MC5746
agilus>» dognostics
NEPHELOMETRY
Interpretstlon(s)
C-REACTIVE PROTEIN, SERUM (QUANTITATIVE)- TesttoDescription:
detet inflammation due to acute conditionsor to monitor the severity of disease in chronic conditions. CRP S one of
A CRP test measures the amount of CRP in the blood 4-6 hours of
Its rapld response to trauma or infection.Synthesis of CRP increases withir
the proteins commonty referred to as acute phase reactants. CRP is distinguished by
onset of inflammation, reaching peak values within 1-2 days. CRP levels also fall quickly afte resolution of inflammation since its half life is 6 hours. levels ir
different tests that measure CRP and each test measures a different range of CRP
This standard CRP test is not to be confused with a hs-CRP test. These are twoof protein observed In diseases that cause significant inflammation.
the blooc for different purposes. The standard CRP test measures hlgh levels
Test Interpretation: inflammation but will not identify its location or the cause.
Increased CRP level: In creasing amount of CRP in the blood suggests the presence of
SUspected bacteríal Infection: a high CRP level can confirm that you have a serlous bacterlal ínfection.,
have a chronic inflammatory disease or that treatment has not been effective.
Chronic inflammatory disease: high levels of CRP suggest a flare-up if you recovery from surgery, myocardial infarction, transplantation, infammatory bowel
disease,
Test1nc for dicated in the following clinical situations - monitoring
s indica
also prove useful In determinling disease progress or the effectveness of
infectious diseases. Measuring and charting C-reactive protein values can
rheumatic diseases and
treatments
of birth control pills or hormone replacement therapy (.e., estrogen). Higher leveis
of
CRP leves can be eievated in the later stages of pregnancy as well as with the use in people who have cancer.
CRP have elso bbeen observed in people who obese. CRP can also be increased individuals for
Recommendation: The hs-CRP test precisely detects lower levels of the protein than that measured by the standard CRP test and is also used to evakuate
disease, It measures CRP in the range from 0.15 to 20 ma/L.
risk of cardiovascular
Limltation: present, Levels may not increase in conditions like pregnancy, angina, seizures,
CRP leves n autotmune diseases may show ittie or no increase unless infection is Interpreted without a complete clinical history and evaluation.
asthma, common colc. The main limitation of CRP Is in its non-specific response and should not
**End of Report**
Please visit www.agilusdiagnostics.com for related Test Information for this accession
Page 1 Of 2
MC-5746
agilus>»
PATIENTNAME : HAFIZA BEGUM REF, DOCTOR : DR. RATHINDPA NATH BISWAS
TODE /NAME & ADDRESS :CO00111042 IAGE/SEx 48 Years Female
ACCESSION NO :0269XD000338
sURE &CURE DIAGNOSTICS PATIENT ID DRAWN 04/04/2 024 12:59 19
HAFIM2405750
AFARING NO. 2440, PANDUA STATION BAZAR. 04/04/2024 16.1845
CUENT PATIENT 10: RECEIVED
POST OFFICE LANE,P.S. PANDUA,
ABHA NO
REPORTED :04/04/2 024 19:37:06
HOOGHLY 712149
9434562382
Page 2 of 2
MICROBIOLOGY
GRAN STAIN
SPUTUM
SPECIMEN SOURCE NUMBER OF
MODERATE PUS CELLS, FEW EPITHELIAL CELLS, MODERATE AND
AND CHAINS
GRAM STAIN GRAM POSITIVE COCCI SEEN IN PAIRS, TETRADS
FEW GRAM NEGATIVE BACILLI SEEN.
MICROSCOPIC EXAMINATON
METHOD: GRAMS STAIN +
Interpretation(s) in cinical
GRAM STAIN-GRAM STAIN the only method ermployed for the diagnostic iderntification of bacteria
Gram stain is the mostst important staining method in bacteriology. It is the first and usually stained smears from clinical specimens involves consideration of staining dark
of clinical specimens. Interpretatlon of gram which stain
specimens. It also serves to assess the quality two categories of genera: the Gram-positive,
of particular host cell types. It distinguishes two types of cells, Further details of the
bacteria
characteristic,morphology of the etiological agent and presence Gram-variable, and tend to show a mixture of the resuits s
light red. A few species are bserved. Comparing Gram stairn resuit to culture
purple. and the Gram-negative, which stain comma shaped Gram negative bacilli) are also
any other special features, including unusual shapes (such as
as monitoring quality assurance.
an exce llent internal method for
**End of Report**
for this accession
www.agilusdiagnostics.com for related Test Information
Please visit
Page 1 of 2
Dr.Himadri Mondal, MD
Consutant Microbiologist
agilus>
BEGUM
PATIENT NAME : HAFIZA femole
ADDRESS :CO00111042
/NAME &
CODE DIAGNOSTICS AIEH
SURE & CURE PANDUA SIAION IAA
QEARING NO, 2440,
PANDUA.
P.S.
POST OFFICE LANE, ABIIA
HOOGHLY 712149
9434562382
REPORTING
cONDITIONS OF LABORATORY TESTING &
belongs to the patient 5. AGILUS Diagnostics confirrs that all tests have been
sample
1. It is presumed that the test requisition form. perforned or assayed with highest quality standards, clinical
named or identified in the test safety & technical integrity.
reported as per the
2. All tests are performed and AGILUS Directory of Services. 6. Laboratory resuts should not be interpreted in
isolation,
turnaround time stated in the be correlated with clinical inforrnation and be
unforeseen it rnust
3. Result delays could oCcur due to / equipmnent interpreted by registered rnedical practitioners only to
circumstances such as non-availability of kitsdowntime or any determine final diagnosIS.
breakdown / natural calamities / technical 7. Test results may vary based on tirne of
collection,
other unforeseen event.
physiological condition of the patient, current rmedication or
performed if:
4. Arequested test might not be nutritional and dietary changes. Plese consult your doctor
i. Specimen received is insufficient or inappropriate or call us for any clarification.
ii, Specimen quality is unsatisfactory Test results cannot be used for Medico legal purposes.
8
ii. Incorrect specimen type care
specimen 9. In case of queries please call cUstorner
iv. Discrepancy between identification on (91115 91115) within 48 hours of the report.
container label and test requisition form
Agllus Dlagnostics Ltd
Fortis Hospítal, Sector 62, Phase VIII,
Mohali 160062
Page 2 Of 2
Dr.Himadri Mondal, MD
Consultant Microbiologist