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Acute Bowel Obstructions
Acute Bowel Obstructions
Classification
It can be classified as mechanical and functional
I. Mechanical: bowel capable of contracting normally or excessivly proximal to a local
site of obstruction
Occlusive
Strangulation
Mixed (instussusception)
II. Functional: peristalsis maybe absent (paralytic ileus), or present in non-propelsive
form (mesenteric vascular occlusion, pseudo-obstruction)
Spastic ileus
Paralytic ileus
ALSO WE CAN DIVIDE INTO
For small bowel 80%
1. Extramural
Adhesions - mostly post-operative, 66% adhesion and 10-25% obstruct
Hernia- inguinal in particular
Neoplasm- external mostly
2. Mural
Strictures – crohn’s disease
İntussusception
Meckels diverticulum
3. Intraluminal
Gallstone ileus
Foreign body
Mechanical obstructions
Common causes of small bowel: adhesions and bands following abdominal surgery, external
hernia, intussusceptions, volvulus, neoplasm, obstruction of worms, strictures (IBD).
Common causes of large bowel: large bowel cancer, sigmoid diverticular disease, sigmoid
volvulus.
Functional obstructions
Common causes of small bowel: paralytic ileus after abdominal surgery, peritonitis,
mesenteric embolism or thrombosis with small bowel infarction, intestinal pseudoobstruction.
Common causes of large bowel: retroperitoneal hematome following lumber fracture or
lumber surgery, idiopathic
PATHOPHYSIOLOGY
The gastrointestinal tract secretes fluid later absorbs in the colon, when there is an
obstruction this fluid cannot reach the colon and cannot be reabsorbed
As a result there is a fluid loss in intravascular space into the gastrointestinal tract, this leads
to hypovolemia and shock. This abnormal loss of fluid is referred to as “third-spacing”
CLINICAL FEATURES
1. Crampy pain
2. Vomiting
3. obstipation
4. Abdominal distention
5. Also fever, tachycardia,
Detailed
PAIN
SBO: cramping/colicky, central
LBO: spasms, longer lasting, lower abdomen
The pain will be continous if perforation or strangulation is present
Absent in paralytic ileus
Patient finds relief by drowing legs up or fetal position.
VOMITING
Early in high SBO
Late in low SBO
Delayed or absent in LBO
Character: initially clear and becomes discolored and finally feculent (dark and foul
smiling)
CONSTIPATION
Early in LBO
Late in SBO
Absolute in complete obstruction
ABDOMINAL DISTENTION
Epigastric or hypogastric in SBO
Generalized in LBO
A FEW IMPORTANT POINTS about small bowel obstructions:
- Distal bowel obstruction will usually have less emesis
- Nausea and bilious emesis without pain can be common in proximal obstruction
- Obstipation occurs late
- Stinky emesis occurs as a late finding (bacterial overgrowth)
Inspection:
By inspection, need to look scars of previous surgery which can lead to adhesions, visible
peristalsis, swelling in case of hernia, abdominal tenderness, guarding, rebound
tenderness(peritonitis), hyperactive bowel sounds,
Tongue may be dry due to dehydration
Pulse, respiratory rate, blood pressure are usually normal at first. As the patient loses fluid
into the bowel and with vomiting, it can lead to tachycardia and low grade fever.
And overtime patient will have: anxious expression, sunken eyes, dry skin and mucous
membrane
Pathognomonic signs:
Wahl's sign: asymmetry of the abdomen, high tympanic sound over the distended bowel.
Sklyarov’s sign: splashing sounds in the small or large intestine during balloting
palpation of anterior abdominal wall.
Kywul’s sign: metallic sound over bloated intestinal loop during percussion of plesimer.
Schlange’s sign: visible peristals.
Spasokukotsky’s sign: during auscultation “sound of falling drop” above the stretched
intestinal loops.
Tsege Manteuffel’s sign: when setting the siphon enema all entered the liquid (up to 500
ml) quickly leaves the intestine does not contain impurities of feces and gases). Positive at
sigmoid volvulus
DIAGNOSIS
Blood test:
Electrolyte imbalances, hypokalemia (in U(urea) and E blood test)
Metabolic alkalosis due o vomiting stomach acid (in venous blood test)
Raised lactate which indicate bowel ischemia (venous blood gas or lab sample)
Leukocytosis- shows ischemia or perforation
Raised creatitine- may indicate acute kidney injury. Which is a common complication
due to the vomiting and dehydration.
Abdominal x-ray: may see dilated gas-filled loops or air fluid levels which can indicate
obstruction.
Erect chest x-ray: detects air under diaphragm due to intraobdominal perforation
***Contract abdominal CT scan: confirm the diagnosis, site and cause and detects
perforation.
TREATMENT
Conservative:
Spasmolytics, antiemetics
- No suspicion of ischemia or perforation
- Patient needs to be NIL by mouth
- IV fluids for hydrate and correct the electrolyte imbalances
- Nasogastric tube with free drainage
SURGERY
Depends on cause
- Hernia reparation
- Tumor resection
- Resection of blocked region and anastomosis( in patients with colorectal cancer who
may underdo hemicolectomy where half of the colon is removed)
- Stoma (opening of the bowels onto the skin
- Stenting (to reduce symptoms or sometimes done as a bridge to surgery)
DIFFERENTIAL DIAGNOSIS
Perforated peptic ulcer
Acute Appendicitis complicated with peritonitis
Acute Pancreatitis
Hepatic abscess
Renal colic
Perforative peptic ulcer Acute intestinal obstruction
Sharp acute diffuse pain Periodic acute diffuse pain as wavelike
Absence of hepatic dullness Constant vomiting and nausea without any
relief
Rigidity of anterior abdominal wall Abdominal distention
On Xray of the abdomen air above the liver On Xray Kloibers cups (airfluid levels)
(air sickle)
Splashing sound, increased peristalsis