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Figurations of Peripheries Through Arts and Visual Studies Peripheries in Parallax 1St Edition Maiju Loukola Online Ebook Texxtbook Full Chapter PDF
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Figurations of Peripheries Through
Arts and Visual Studies
This series is our home for innovative research in the fields of art
and visual studies. It includes monographs and targeted edited
collections that provide new insights into visual culture and art
practice, theory, and research.
and by Routledge
4 Park Square, Milton Park, Abingdon, Oxon, OX14 4RN
DOI: 10.4324/9781003348269
Typeset in Sabon
by MPS Limited, Dehradun
Contents
List of Contributors
Introduction
MAIJU LOUKOLA AND MARI MÄKIRANTA
PART I
Sites and sentients
PART II
Aesthetics and practices
PART III
Visual culture rearticulations
Index
Contributors
DOI: 10.4324/9781003348269-1
References
Ahmed, Sara. Cultural Politics of Emotion. Edinburgh: Edinburgh
University Press, 2004.
Borgdorff, Henk. The Conflict of the Faculties: Perspectives on
Artistic Research and Academia. Leiden: Leiden University Press,
2013.
Borgdorff, Henk, Peters, Peter & Pinch, Trevor. Dialogues between
Artistic Research and Science and Technology Studies. Routledge,
2020.
Braidotti, Rosi. Transpositions. On Nomadic Ethics. Cambridge: Polity,
2006.
Elo, Mika. “Three phases of artistic research”. Ruukku Journal.
Studies in Artistic Research, 18, 2022. http://ruukku-
journal.fi/en/issues/18/voices/mika-elo
Grosz, Elizabeth. Space, Time, and Perversion. Essays on the Politics
of Bodies. New York and London: Routledge, 1995.
Hall, Stuart. Representation: Cultural Representations and Signifying
Practices. London: Sage Publications & Open University, 1997.
Haraway, Donna. Staying with the Trouble. Making Kin in the
Chthulucene. Durham and London: Duke University Press, 2016.
Johansson, Hanna. “Liikkuvan kuvan materiaalisuus ja haptinen
representaatio”, in: Tarja; Lehtinen & Aki-Petteri Nuuttinen (eds.)
Representaatio: Tiedon kivijalasta tieteiden työkaluksi, Helsinki:
Gaudeamus, 196–213, 2010.
Keskinen, Suvi. “The ‘crisis’ of white hegemony, neonationalist
femininities and antiracist feminism”. Women's Studies
International Forum 68, 157–163, 2018.
10.1016/j.wsif.2017.11.001
Keskinen, Suvi, Skaptadóttir, Unnur Dís & Toivanen, Mari (eds.).
Undoing Homogeneity in the Nordic Region. Migration,
Difference, and the Politics of Solidarity. London: Routledge,
2019.
Keskitalo-Foley, Seija & Naskali, Päivi. “Tracing Places and
Deconstructing Knowledge”. In Seija Keskitalo-Foley, Päivi Naskali
& Pälvi Rantala (eds.) Northern Insights. Feminist Inquires into
Politics of Place, Knowledge and Agency. Rovaniemi: LUP, 7–15,
2013.
Latour, Bruno. Reassembling the Social. An Introduction to Actor-
Network-Theory. Oxford and New York: Oxford University Press,
2007.
Lummaa, Karoliina & Rojola, Lea. “Johdanto: Mitä posthumanismi
on?” In Karoliina Lummaa & Lea Rojola (eds.) Posthumanismi.
Helsinki: Eetos, 13–32, 2014.
Philippopoulos-Mihalopoulos, Andreas. Law and the City.
Abingdon/Oxon and New York: Routledge-Cavendish, 2007.
Probyn, Elspeth. Blush. Faces of Shame. Minneapolis: University of
Minnesota Press, 2005.
Puuronen, Vesa. Rasistinen Suomi. Helsinki: Gaudeamus, 2011.
Seppä, Anita. “Miten kertoa uudelleen ihminen ja edistys? Pohdintoja
eurooppalaisen korkeakulttuurin afrikkalaisesta alkuperästä”. In
Hanna Johansson & Anita Seppä (eds.) Taiteen kanssa maailman
äärellä. Kirjoituksia ihmiskeskeisestä ajattelusta ja
ilmastonmuutoksesta. Helsinki, PARVS & Taideyliopisto,
Kuvataideakatemia, 48–99, 2021.
Seppälä, Tiina. “Naiset, aktivismi ja feministinen solidaarisuus”.
Politiikasta.fi, 2017. http://politiikasta.fi/naiset-aktivismi-ja-
feministinen-solidaarisuus/
Sinevaara-Niskanen, Heidi. “Vocabularies for human development:
Arctic politics and the power of knowledge”. The Polar Record
51(2), 19, 2015.
Part I
Sites and sentients
Maiju Loukola, Mari Mäkiranta, and Jonna Tolonen
1 Performing with trees as
shifting attention
Annette Arlander
DOI: 10.4324/9781003348269-3
Practicing in a park
While preparing the artistic research project called Performing with
Plants,9 I was not fully aware of the extent of such interests. The
most important inquiries I wanted to explore were: 1) How can I
collaborate with nonhuman entities like plants? 2) How can I further
develop experiences from previous attempts at performing
landscape? 3) How can I create actions with plants, in which humans
can be invited to participate? The overarching research topic was:
How can I perform landscape today by collaborating with trees and
other plants, with an awareness of the insights generated by post-
humanist and new materialist debates? Thus, I asked questions such
as what can I do together with trees, how can I perform together
with trees for the camera on a tripod, and how can I appear in the
same image space with trees? I sought to combine my previous
practice of creating rough time-lapse videos to document changes in
the landscape with a focus on trees. As part of this three-year
project,10 I made time-lapse videos with specific trees, first both in
Helsinki and Stockholm, then in Stockholm only.11 In this text I
present one part of the project, performances with trees made in
Stockholm during the year 2018 (Figure 1.1), and I focus on some of
the ethical dilemmas involved.
Lafosse
Acute.
(1856)
Mammitis
Trasbot
Chronic.
(1883)
Catarrhal.
Saint Cyr
Phlegmonous or
(1874) Mammitis
interstitial.
Violet (1888)
Parenchymatous.
Galactogenous.
Acute
Primary mammitis (properly so- Lymphogenous.
called) Galactogenous.
Chronic
Lymphogenous.
Lucet (1891)
Hæmatogenous.
Acute
Lymphogenous.
Symptomatic mammitis
Hæmatogenous.
Chronic
Lymphogenous.
All these classifications are justified by the guiding ideas of the
writers, yet, as in every case of attempted systematisation, they have
the disadvantage of not being in entire agreement with clinical
experience.
For instance, the differences between catarrhal and
parenchymatous forms of mammitis are only of degree, and it is
difficult, therefore, to see why they should be divided into two
distinct varieties. The difference is in regard to the prognosis.
Similarly in practice it is difficult and sometimes impossible to
distinguish between an interstitial and a parenchymatous mammitis,
because all the tissues of the gland may be involved at a given
moment. The only factor which allows of differentiation is the
discovery of the point from which infection took place. Finally, it is
sometimes so difficult to distinguish between galactogenous and
lymphogenous mammitis that the attempt has had to be abandoned.
In gangrenous mammitis of milch ewes, for example, the infective
organism is found not only in the sinus and the galactophorous
canals, but also in the serosity of the interstitial tissue and of the
perimammary œdema.
Without doubt the causative agent of mammitis may enter the
gland by three principal channels—the galactophorous sinus, the
lymphatic plexus (after some injury), and the blood circulation. But
from the clinical standpoint it is not at all necessary to identify all the
causes in order to establish the classification.
The symptoms allow of a division only into acute and chronic
mammitis. Careful examination of the general condition of the
patients will afterwards allow cases of primary mammitis to be
distinguished from secondary or symptomatic mammitis such as
occurs in tuberculosis. Finally, consideration of the conditions under
which a particular case of mammitis has appeared, and study of the
symptoms in detail (peculiarities of the milk, local temperature,
hardness of the tissues, œdematous infiltration, etc.) will in most
cases indicate whether the mammitis be parenchymatous or
interstitial.
This system really differs little from that adopted by Lucet in his
work on Mammitis.
The classification adopted in the following pages is as follows:—
Parenchymatous or
Primary galactogenous.
Acute Interstitial or lymphogenous.
Secondary or Hæmatogenous.
symptomatic. Galactogenous.
Mammitis
Simple.
Primary Parenchymatous.
Chronic Interstitial.
Parenchymatous.
Secondary
Interstitial.
We shall leave on one side everything concerning secondary
symptomatic mammitis, the study of which merges into that of the
general diseases from which it arises.
ACUTE MAMMITIS.
Causation. The general cause of acute mammitis, like that of
chronic mammitis, is infection by pathogenic organisms, whether
such organisms enter by the usual natural path, viz., the
galactophorous sinus and excretory apparatus, by the lymphatic
path, owing to some accidental injury, or, again, by the blood
circulation.
Infection of the lymphatics undoubtedly plays a part in superficial
and interstitial inflammations, and it is proved that certain
microorganisms may pass into the milk, as it has been proved that
they pass through the kidney.
But if infection is the determining cause, certain secondary
favouring influences must not be overlooked.
Thus lactation is an almost indispensable condition. It is true that
some cases of mammary inflammation apart from lactation have
been described, but they have been the result of violence, accidental
or mechanical.
Accumulation of milk in the udder (overstocking) has
unquestionably a certain influence in the large milk-yielding
animals, not because it directly produces inflammation, but because
milk then escapes spontaneously; and as the udder cannot be
entirely evacuated without external assistance, the entrance is kept
permanently open for the passage of germs, which are freely
transferred to the teat from the litter and surrounding objects.
Cold, or rather chills, also act in a complex manner, particularly by
disturbing vaso-motor control. Different forms of mechanical
violence, such as blows, crushing strains, wounds, etc., may
immediately and directly set up local or general inflammation.
Bacteriological investigation has proved that numerous and varied
microorganisms can be found in the milk or interstitial exudates in
cases of mammitis, but only a few special forms have been proved to
be specific: streptococcus of contagious mammitis of milch cows, and
micrococcus of contagious gangrenous mammitis of ewes (Nocard).
Pathology. The pathogenic results produced by infective
organisms depend on their number and power of reproduction, and
on the activity of their life products.
The most immediate and regular result of acute mammary
infection is coagulation of the milk within the udder by
decomposition of the lactose, and the formation of lactic or even of
butyric acid. The acini and excretory canals are dilated by coagula,
and can no longer expel their products of secretion, so that the
colonies of microorganisms develop there in full security. The active
epithelial cells undergo granular degeneration and disappear, whilst
the walls of the glands become infiltrated and large numbers of
leucocytes are poured forth around the glandular culs-de-sac.
The tissues being thus affected, the virulent organisms penetrate
from the acini into the interstitial tissue, and from this time onwards
the lesions become mixed.
Inversely, should infection originate in the lymphatic spaces, a
time arrives when the organisms make their way from the interstitial
tissue into the acini, with a similar result in the end.
The development of the lesions may be arrested or may pass on to
suppuration, or even gangrene, of the parenchymatous lobules. Cases
happen in which infection is so rapid and severe that the successive
stages cannot even be identified, and gangrene appears without any
preliminary stages at all. Luckily the commonest forms are less
serious.
Symptoms. Acute mammitis is characterised by its sudden
appearance, more or less acute general symptoms (dulness, fever,
and loss of appetite), and variable local symptoms. When the
practitioner is able to follow the development of the disease
throughout, he may sometimes distinguish well-marked signs, which
permit the two clinical varieties to be distinguished.
A. Interstitial Mammitis.—This form, which might perhaps
also be termed peri-mammitis when it primarily affects the
subcutaneous lymphatic spaces, has also received the names of
phlegmonous and lymphogenous mammitis.
It is characterised by alarming general symptoms, and particularly
by a rise in temperature of 2°, 4°, or even 5° Fahr., with all its
consequences, such as loss of appetite, stoppage of rumination,
acceleration of breathing and circulation, slight tympanites,
constipation, and by the thrusting of the hind limb on the affected
side away from the centre line. The animals groan when forced to
move.
These symptoms sometimes precede by a considerable interval the
appearance of the local changes, which consist in painful swelling of
one or two quarters, rarely of more.
The perimammary subcutaneous tissue is infiltrated, œdematous,
painful on palpation and preserves the imprint of the finger. The teat
is tense, swollen, very tender, and of reddish colour. In the grave
forms the swelling extends forwards under the abdomen in the
direction of the umbilicus, and backwards towards the perineum.
The local temperature is abnormally high, the secretion of milk in the
diseased gland is modified or checked, and sometimes this
phenomenon extends by reflex action to the neighbouring quarters,
although the latter may not themselves be affected. The
inflammation rarely extends from one quarter to another, because
the lymphatic plexuses do not anastomose (Fig. 237).
The animals lose appetite and fall away rapidly.
Resolution may occur after from five to eight days. By degrees all
the symptoms then become less marked. The appetite returns, pain
diminishes, the fever drops, and the lesions gradually disappear, but
the yield of milk rarely regains its former amount.
Suppuration may occur; sometimes a superficial subcutaneous
abscess forms, more rarely, a deep-seated, interstitial abscess,
originating in the connective tissue or lymphatic spaces. With a
superficial abscess, the local symptoms again revive to a slight
extent; these are present in a more marked degree where the abscess
is deep-seated. An extremely sensitive œdematous swelling forms,
the skin covering which is at some point of a deep-red tint, whilst
fluctuation gradually appears.
In cases of deep-seated abscess formation the general condition
becomes alarming; the affected quarter is tense throughout, hard
and very sensitive.
Deep-seated suppuration is difficult to detect, and exploratory
punctures with a fine needle may be necessary before the diagnosis
can be made.
Local or diffuse gangrene forms a rare complication. It is due to
the vessels of one or several glandular lobules becoming obliterated
or thrombosed.
Such a termination is indicated by extreme aggravation of the
general symptoms, feebleness of the heart and great weakness of the
patients, who fall into a condition of coma. Locally the udder
remains œdematous, the skin becomes of a blackish-violet colour,
whilst the local temperature falls and the animals die from
exhaustion and intoxication.
Parenchymatous Mammitis.—Parenchymatous mammitis
when mild is also termed catarrhal mammitis. It is in reality true
primary mammitis; interstitial being primarily and practically
perimammary lymphangitis.
In this case infection occurs through the teat, and may be localised
in the sinus or excretory apparatus, giving rise to galactophoritis, but
it usually extends to the acini. Inflammation of the mammary tissue
is therefore direct and primary. It rapidly extends, however, through
the glandular wall into the interstitial tissue, thus setting up (from
the anatomo-pathological standpoint) a mixed mammitis. Clinical
distinction between this form and interstitial mammitis is at first
easy.
The symptoms usually occur in the following order:—Swelling of
the affected quarter or quarters; appreciable increase in size and
sensitiveness; the presence at first of curdled milk in the
galactophorous sinus, then of clots mixed with slightly red tinged
serosity; complete cessation of the yield of milk, and suppuration in
the secreting portions of the gland.
The general symptoms appear only after the objective signs, and
vary greatly in intensity, according to the case. As in the interstitial
form, there may be marked fever, loss of appetite, cessation of
rumination, groaning, and difficulty in walking.
In some grave forms, where development is peracute, infection
spreads rapidly from the glandular to the interstitial tissue, and
subcutaneous, subabdominal, or perineal interstitial œdema occurs
secondarily.
The udder is turgid, tense, shining, and of reddish-violet colour in
places, as if a deep-seated abscess were developing.
Pressure on the galactophorous sinus causes the flow of reddish-
grey milk, sometimes fœtid or of gangrenous odour. The animals
seem exhausted, show signs of profound intoxication, are unable to
rise, and appear paralysed.
But besides these grave forms are others, in which the patients
seem scarcely to suffer: appetite is preserved and all the vital
functions are in full activity. Only the local signs are of importance.
This variability in the clinical symptoms of acute forms of
mammitis is entirely due to differences in the pathogenic infecting
organisms.
Parenchymatous mammitis may end in resolution in three to four
days, with progressive but slow return to the physiological condition.
This termination is announced by the gradual disappearance of all
the symptoms and the return of milk secretion. It is, however, quite
exceptional for the former condition to be fully restored, and in many
cases the affected quarter must be regarded as lost from the
physiological standpoint.
It gradually becomes hardened, sclerotic and atrophied.
Suppuration is very common. It attacks the galactophorous sinus,
the excretory canals, and even the acini. If obstructions occur in the
course of the collecting vessels, or if evacuation is not artificially
stimulated by milking, the pus collects in the depths of the gland,
and enormous diffuse abscesses may form at the expense of the
mammary tissue.
Circumscribed or diffuse gangrene, as a primary condition, is
rarer. Infective organisms rapidly invade even the depths of the
gland, the interstitial and subcutaneous tissue, and thrombosis due
to infection or intoxication occurs, followed by gangrene. Death
results from infection or intoxication.
Complications such as necrosis of the abdominal tunic, of the
fibrous tissue enveloping the mamma, and of the muscular layers on
the inner surface of the thighs, may occur in the suppurative forms.
Diagnosis. The diagnosis of acute mammitis is easy, and the
interstitial forms (mammary lymphangitis) can be distinguished
from the parenchymatous forms very early in the attack.
Careful examination suffices to differentiate between this
condition and mammary congestion or primary chronic mammitis.
The examination, however, must be much more thorough and
searching when a specific disease (such as tuberculous mammitis) is
in question.
Prognosis. The prognosis of acute mammitis is always grave,
whatever form the disease may assume, for, if the animal’s life is not
invariably endangered, its economic value is always affected.
Moreover, should superficial or deep-seated abscesses form,
prolonged suppuration may follow, resulting in loss of condition and
enormous depreciation.
Lesions. The lesions of interstitial mammitis are similar to those
of ordinary lymphangitis, the condition originating near the teat and
gradually extending to the layers of connective tissue between the
acini, mammæ, etc.
In the parenchymatous form the inflammation may remain partial,
and be localised in particular tracts of glandular tissue. The secreting
epithelium, when infected, exhibits cloudy swelling, becomes
loosened, and disappears; the margin of the gland and the interstitial
divisions become infiltrated with enormous numbers of white blood
corpuscles, and are the seat of suppurative processes which end in
the production of small acinous abscesses. By the union of
neighbouring abscesses large branching collections of pus are
produced, and lead to partial or total destruction of tracts of the
parenchyma, of the connective tissue divisions, vessels and
aponeuroses.
The abscess tends to break through the skin, which becomes
inflamed and ulcerated, or, when the microorganisms are of slight
virulence, the tissues may react, so that the abscess becomes
surrounded with a thick indurated wall, and finally encysted.
Treatment. Very numerous methods of treatment have been
proposed, an admission which, in itself, suggests that no perfect one
has been discovered. No infallible system, in fact, exists of arresting
the disease and restoring the parts to their normal condition.
From a prophylactic standpoint, mammitis can be avoided by
placing the animals under proper hygienic conditions, paying special
attention to cleanliness, avoiding overstocking, and treating
excoriations or injuries to the teat or udder as soon as they appear.
Once acute mammitis has developed, general and local treatment
must both be attempted.
The older practitioners were in the habit of bleeding from the
mammary or jugular vein. Since their time, however, objections have
been made to bleeding because acute mammitis has been proved to
be of an infectious character, and, therefore, it is undesirable to
lower the patients’ resisting power.
This reasoning, however, appears to be erroneous. Little by little
the advantages of bleeding, both in intoxications and infections, have
been recognised, and one thing at least is beyond dispute, namely, its
action on fever. Undoubtedly, it must not be resorted to without
judgment, nor should it be freely employed in debilitated animals;
but in well-nourished patients its effect on fever and on the
accompanying respiratory and circulatory disturbance is immediate.
We, therefore, recommend moderate bleeding from the jugular.
Bleeding from the mammary vein entails too great a risk of
infection to be commendable.
Purgatives and diuretics diminish or prevent accidents such as
intoxication and the complications resulting from temporary
suspension of the digestive function.
Local treatment is more or less efficacious in mammary infection.
To relieve pain and check infection it should be of an emollient and
antiseptic character. Ointments containing 10 per cent. of carbolic
acid, boric acid or iodine, or 12½ per cent. of camphor, opium or
belladonna, are of real service during the first stages, particularly of
mammary lymphangitis and interstitial mammitis.
Repeated applications of 10 per cent. carbolic glycerine have
similar advantages.
In the less acute forms originating in the parenchymatous tissue,
mild ointments of plumbic iodide, Goulard’s extract, or mercury may
also be used if precautions are taken to prevent the animals from
licking, and so poisoning themselves.
When the tendency to suppuration is marked, vesicants hasten the
development of the abscess and facilitate puncture. The most
commonly used are the 33 per cent. tartar emetic ointment or the 10
per cent. biniodide of mercury ointment.
If, on the other hand, the mammitis is of the interstitial type, with
severe subcutaneous œdema, extending over the belly and towards
the perineum, good results often follow deep firing in points over the
swollen region. The points should be widely spaced, venous branches
being avoided. In this way numerous ducts are formed by which the
toxic and septic liquid which causes the œdema is enabled to escape.
This method of treatment can be supplemented by the
simultaneous use of antiseptic ointments.
Finally, in mammitis of the parenchymatous type, where there is
no marked tendency to invade the interstitial tissue, the most
important point is to wash out the interior of the gland, and even the
acini as far as possible, with antiseptic fluids. Practically this is
difficult to effect, because such irrigation must be performed
aseptically, and cannot properly be left to the cowmen.
In current practice, therefore, one often has to be content with
stripping the udder every hour. Milk clots which accumulate in the
sinuses and galactophorous canals are broken down by soft pressure,
and withdrawn with more or less difficulty. By repeated milking they
are prevented from accumulating in the galactophorous sinus and
canals, a very important point. Neglect of this precaution enables the
colonies of microorganisms to develop uninterruptedly in the culs-
de-sac, whereupon the coagula formed of casein obstruct the
excretory channels and complications develop despite all external
treatment.
By repeated friction of the udder as in milking the advantages that
would be produced by washing out the gland from the direction of
the acini are secured, and thus the ascending infection is checked.
The diseased udder must always be emptied before making
antiseptic injections, which would otherwise be useless.
Should the practitioner decide to face the practical difficulties of
injections, he must take care that his instruments are aseptic; that
the solutions employed are always at or about bodily temperature;
that these solutions are incapable of irritating even tissues so tender
as the epithelium of the acini or of the galactophorous canals; and,
finally, that the drugs employed will not coagulate the milk within
the gland.
Bearing in mind these points, the practitioner will do well to
restrict himself to the use either of boiled water, physiological salt
solution (·9 per cent.); alkaline 3 per cent. solution of borate of soda;
or ·05 per cent. of fluoride of sodium. Every precaution having been
taken, from 12 to 20 ounces of liquid can be injected into each
quarter, according to its size. The solutions should be made to
penetrate as far as possible into all portions of the gland by gently
manipulating the parts, and should again be withdrawn in about a
quarter of an hour.
It must always be remembered that failure to observe the above
precautions may make matters worse instead of better, and therefore
that intra-mammary injections can only be of value when carried out
by a skilled person.
In otherwise hopeless cases there remains as a last resort total or
partial ablation of the mamma. This operation is advisable in cases of
diffuse gangrene, or of intense massive suppuration, where there is
imminent danger of death from infection.
Directions for its performance will be found in Möller and Dollar’s
“Regional Surgery” (uniform with the present volume), p. 454.
CHRONIC MAMMITIS.
This disease occurs in different parts of France, and has also been
seen in Germany.
Causation. Lafosse in 1856 attributed it to the dark and dirty
condition of the sheep-folds, a cause which certainly contributes to
its propagation, though it is not the determining cause of the disease
itself. The latter is a specific micrococcus discovered in 1875 by
Rivolta, and thoroughly studied by Nocard in 1886 and 1887.
Symptoms. The course of the symptoms offers a certain analogy
to that of septic engorgements and interstitial mammitis or
mammary lymphangitis. As a rule only one gland is infected, but
generally symptoms at once appear, indicating an extremely
dangerous condition, viz., peracute mammitis. The patient suddenly
becomes dull and entirely loses appetite, rumination ceases and
respiration is short and jerky, although the bodily temperature does
not always rise to any marked extent.
Local symptoms soon develop. The udder assumes a violet-red tint
and becomes the seat of an erysipelatous swelling; the local
temperature rises, but as the disease progresses it gradually falls
again. Milk secretion ceases.
All these appearances rapidly become aggravated. The patient lies
down; the œdematous swelling extends to the belly and even to the
chest and thighs; the local temperature falls, indicating the
imminence of gangrene; the teat becomes contracted, and the pulse
is very frequent and almost imperceptible. From time to time the
animal grinds its teeth.
The bodily temperature next falls to 98° or even 96° Fahr. (37° or
36° C.), and the animal shows extreme prostration. The
subcutaneous swelling extends as far as the sternum in one direction
and the quarters and perineum in the other. The udder crackles
under the finger. Death occurs without a struggle.
All these symptoms follow as a rule in barely more than twenty-
four hours. Nevertheless, in certain cases, the disease lasts for three,
four or five days. Cases of spontaneous recovery are exceptional. The
gangrenous part may become delimited and slough away, leaving an
enormous suppurating wound, which slowly cicatrises. Even though
the animals survive, they never recover condition, but remain
weakly, so that, from a monetary standpoint, death would have been
preferable. Moreover, the lambs are starved and require a foster-
mother.
Lesions. Post-mortem examination reveals œdematous
infiltration of the udder and surrounding connective tissue, and
often extensive, diffuse gangrene. The serosity is of a reddish colour,
and sections of the diseased udder of a violet tint. The tissues of the
udder and the serous liquid contain the specific micrococcus.
It is very small, and stains readily by the Gram-Nicolle method. It
grows rapidly in liquid and solid media, liquefies gelatine, and
quickly renders neutral media alkaline.
The injection of a few drops of culture into the udder of a milch
ewe reproduces the typical disease. In any other species it is without
effect. Infection occurs through the open extremity of the teat, or the
medium of a wound, and the microorganism is so virulent that it
rapidly invades all the tissues.
Treatment. No antiseptic treatment appears capable of checking
the course of the disease.
Surgical treatment alone is of any value, and consists in ablation of
the udder, followed by antiseptic dressing. Only a portion of the
gland is removed: an elliptical incision is made, including the
diseased teat, the skin is then dissected free so as to form a flap; the
diseased tissue is isolated; last of all, the vessels are ligatured. The
consequences of operation are less grave than might be expected,
considering the size of the wound, which heals with fair rapidity.
Moussu has frequently practised this radical method of treatment
without losing a single case. The remaining portion of the udder
becomes hypertrophied, and is often quite capable of secreting
sufficient milk for the nourishment of one lamb.