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SIMPLE ACUTE CATARRHAL CONJUNCTIVITIS.
Causes: irritant gases, smoke, dry air, dust, pollen, microbes, insects, seeds,
chaff, awns, sand, dust, quicklime, coal dust, hairs, whips, canes, branches,
stubble, fences, nails, claws, teeth, iodine, chlorine, bromine, formalin, turpentine,
rain, hail, sleet, glare of sun, ice, snow, white sand or clay, filaria, acari, infectious
diseases. Symptoms: closed lids, epiphora, redness diffuse on lids, ramified on
sclera, projected nictitans, milky flocculent discharge, later purulent, matted lashes
or tarsi. No photophobia. Fever variable. Lesions: exudate: cell proliferation:
papillæ enlarged: desquamation: swollen lymphoid bodies: hyperplasia: abscess:
ulcer. Treatment: remove cause, sterilized syringing, antiseptic and astringent
collyria, sublimate, zinc sulphate, lead acetate, pyoktannin, silver nitrate, morphia,
atropia, cocaine, vaseline, cupping, leeching.
Sequel of acute. Same causes. Old horses, young foals, damp lands, night dews,
frosts, eczema, follicular scabies. Lesions: swelling of lymph bodies in conjunctival
pouch, general congestion, chemosis, hypertrophied papillæ, clouded cornea;
Symptoms: as in acute form but less intense. Treatment: Remove cause, irritants,
damp soils, stables, etc., glare of light, heat, etc., astringent antiseptic lotions,
atropia, mercury oxide, counter-irritation, setoning, cupping, leeching.
More purulent and more infective through dust, tongues, rubbing posts, kennels,
swill; a class due to different microbes. Prevalence in dogs, swine, horses, sheep,
goats, cats. Symptoms: acute conjunctivitis with excess of pus, follicular swelling
and enlarged lymph bodies in cul de sac. Diagnosis. Treatment: Astringent and
antiseptic lotions, injected often: silver nitrate: pyoktannin.
In cow pox, horse pox, and above all sheep pox, the infection
sometimes falls on the conjunctiva, giving rise to the formation of the
specific eruption on that membrane. The coexistent eruption on
other parts of the body (udder, heels, hairy portions of skin) of the
specific variolous eruption, furnishes the means of a satisfactory
diagnosis. The lesions in the eye develop rapidly to an extreme
severity. The lids are swollen and closed, lachrymation abundant,
and early mixed with pus, and when exposed the conjunctiva is
strongly congested with circumscribed areas of elevation. In cow pox
these are circular in form, raised above the surrounding mucosa,
having a deep red areola and a paler, flattened center. In sheep pox
the elevations have the same general character, but are liable to be
more numerous and confluent, and tend to permanent opacities,
cicatrices, and perforations of the cornea with loss of the eye. Short
of perforation, internal inflammations are not uncommon.
Treatment. At the outset this form may be aborted by the
application of silver nitrate solution (2 ∶ 100) or sodium hyposulphite
(1½ ∶ 100). Otherwise the local treatment is like that for simple
conjunctivitis, cooling astringent and sedative lotions, and if need be,
derivatives and eliminants.
APHTHOUS CONJUNCTIVITIS. PHLYCTENULAR
CONJUNCTIVITIS.
Closely allied to the last, are those cases in which vesicles appear
on the conjunctiva. In exceptional cases these are seen during an
epizootic of foot and mouth disease, while in other instances they are
associated with eczematous eruptions on other parts of the body,
particularly in dogs. In the human subject conjunctival vesicles are
often associated with tuberculosis, but this has not been recorded of
animals so far. The disease usually makes a rapid eruption, with
symptoms of extreme inflammation, and its duration is largely
determined by the general disease.
Beside the local treatment by astringent or antiseptic and sedative
collyria it may be desirable to correct the hepatic, digestive or other
disorder on which the eczema depends, or to improve the general
health by a course of bitters or even of cod liver oil.
DIPHTHERITIC CONJUNCTIVITIS IN BIRDS.
A disease of the mouth, fauces, nose and eyes, associated with the
formation of false membranes, has been long recognized in birds
(hens, ducks, pigeons, etc.), and is known to bird-fanciers by the
names of roup, and diphtheria. The disease is characterized by the
presence of a local inflammation in patches, associated in the early
stages, with a free serous discharge, but, later, with the formation of
white, or grayish, fibrinous exudate, which may be at first firm and
smooth, later soft and cheese like, and still later in many cases more
or less necrotic and sloughing. In Dr. Moore’s experience this is more
common on the conjunctiva than on the nares or fauces. The interior
of the eye was not involved in the inflammation. Beneath the false
membrane the mucosa was devoid of epithelium and the underlying
tissue was red, raw and angry, bleeding readily when handled, and
infiltrated with an excess of lymphoid cells.
The lesions however were by no means constant. In some even of
the fatal cases, the exudate was merely muco-purulent, while in
others the false membrane was quite abundant.
The gravity of the affection also varies in different outbreaks or in
the same outbreak at different times. Sometimes it runs a very rapid
and fatal course, while at others it becomes chronic and
comparatively dormant and the great majority recover.
In its more virulent form it is readily inoculable on birds, mice,
rats, and rabbits, while the milder cases are not easily propagated in
this way.
The false membrane contains a variety of saprophytes, and among
them the pathogenic bacillus, which may be obtained in pure culture
by inoculating it on a rabbit or mouse. Emmerich believed this
bacillus to be identical with the Klebs-Löffler bacillus of diphtheria in
man, but Löffler recognizes it as essentially distinct.
The bacillus diphtheriæ avium is 0.8 to 1.5μ long, by 0.8 to 1.2μ
broad. The ends are oval so that short specimens seem round. In
bouillon cultures they form chains or clumps. They stain in aniline
dyes, most deeply at the poles. Are not stained by Gram’s method.
They are non-motile, ærobic, grow in agar, and alkaline bouillon, but
render the latter acid in one or two days. Do not grow on peptone
gelatine, nor produce gas with sugars. Are killed in 5 minutes at 58°
C. (137° F.). Are killed by dryness in 24 hours, by sulphuric acid
(0.25 ∶ 100) in 10 minutes, by lime water in 1 minute, and by sulphur
fumes.
Pathogenesis. Eight rabbits inoculated subcutaneously with 0.1 cc.
of the bouillon culture died in 18 to 36 hours. Of several white and
gray mice inoculated only one died. Inoculations of mature hens
subcutem and on the nose had no effect. Inoculation of a six weeks
old chicken caused death in 4 days with bacilli in the liver and blood
(Moore). Inoculation of pigeons produced the disease (Löffler). It
seems doubtful whether the milder forms part with their virulence to
birds, when cultivated artificially, or whether a special susceptibility
is required in order to render the inoculation effective.
Prevention. Besides the general conditions of good hygiene,
cleanliness, pure air, and pure water, the strict exclusion of the germ
is the great desideratum. New fowls should not be taken into a flock,
when they show any indication of disease in the mouth, throat, nose
or eyes, nor when they come from a flock in which such signs of
disease can be found. When examination of the flock, from which
they are sold, cannot be made, the new fowls should be placed by
themselves in quarantine until proved sound. Sound flocks should
not be allowed to wander at large and mingle with the birds that are
unsound, or open to suspicion. Neither should they be allowed to
come in contact with manure from suspected poultry yards. If
disease of air passages, mouth or eyes appears, separate at once the
diseased fowl, and sprinkle roost, house and yard with dilute
sulphuric acid, quicklime, or other disinfectant.
Treatment. Beside separation and disinfection the local use of
antiseptics to the surface divested of the false membrane gives the
best promise. Boric acid solution (4 ∶ 100) or sublimate solution (1 ∶
5000) may be applied to the eye. For the nose and mouth somewhat
stronger applications may be made.
Prevention of infection to man. The essential difference of the
Klebs-Löffler bacillus of diphtheria in man, and the microbe of this
affection in fowls does not exclude the necessity of avoiding
contagion from birds to man. Among reported cases of such infection
are: (1) That at Wesselhausen, where 4 attendants on the fowls
contracted the disease from the sick fowls at a time when no other
cases existed in the human population: (2) That of Sebdon where 6
persons suffered and 10 fowls fed by a hospital attendant also
suffered: (3) That of Tunnis in which diphtheria prevailed in fowls,
and soon also in those who fed them producing an extended
epidemic: (4) That of Jacksonville, Ill., where a diphtheritic chicken,
conveyed the disease, with fatal effect, to a child which fondled it
(Moore).
These and other similar instances seem to show that the disease of
the bird may become so virulent as to be communicated to man, and
the disease of man so potent as to be transmissible to the bird. Every
precaution therefore should be taken to prevent infection passing
from one to the other.
FOLLICULAR CONJUNCTIVITIS.