Pyloric stenosis

Intro It condition of
: is the in which musculature
pylovic
-

antrum is
significantly nylcertrophied ,

the pyloric canal

is
lengthened resembling
olive
shade

Incidence : -
.
I in 1000 babies
· of
: q = 4: /
More in infants : Blood 28
B
group
·

Occ other defects like TOF CHD

genetic
·
.
asso a
,

Etiology Unknown
: -
o

·
Mother which are i
treated macrolide in

pregnancyserve landin levels

pHighed crostag
O

Iwide
.

·
nitc R .
levels

Clinical
Usually bett week to
-.
i
resents I 4 months
I
Manifestate
ins at 3-6 weeks
peals
E

Progressive
projectileiop /
feate
vomiting
most
imp
· ·
e

Forcible

tin.
ged
blood
May
be
·
Not loss
·
FTT
·
Jaundice
(interopyloric syndrome)
Facies alert
signs 8 anxious
hungry look
- · -

, ,

Slow & Shallow Alkalosis

breathing (If
·
:: Metabolic
· visible
gastric peristalsis to Rt)
·
palpable
in
tumou
70 % cases
Imperforated anus
TOF
 Hydrocephalus
It clinical ofraised
syndrome
Intro :
-

is a ICP =

ventriculomegaly From defective flow of

IY
Usually CSF
occurs

choroid
a)
varely by excessive secret· R.
by plexus
.
Op

wired
Etiology :-
Congenital Aq
Benign cyst
Tumour
-

Meningitis
·Aneuyan
is
·

infects Head
injury
·

malformation
Chiari-I Subarachanoid
hemorrhage
-
-

Defective
cong syndromes
-various of
absorption
-

CSF => TBM ·

Classification Obstructive Non. Obstructive

(Non-communicating) (Communicating)
-

Obstruction of CSF flow at ·

Obs outside ventricles

FO sub avachanoid
MORIO
Aq of S FO LG M space
·

, ,
 Spina bifida
Intro defect
: Birth in which there is
incomplete closure of
spinal
-

cord and
meriges
.

It first of
during
month
gestation
occurs .

ocele) S
of SB .
1

MeniMeni
ngocal e
There are three
types ca

· .
ngocculta
Incidence : - 1 in 1000 birth
SBO is MC (15 % )

Etiology : -

Genetic Environmental
Folate def
syndromic As
-
·

Drugs (phenytoin etc)

controlled
Poorly DM

Bifida
Spina Occulta

Defect in vertebral
body without
spinal cord &
meninges
.
·

Mildest & form 15 Si

MC
Usually
.
·
.

Diagosed accidentally
The too that does
split small not
protrude
·
is it .

normal
·

Overlying skin may

be
,
may
have hair ,
dimple or discol
vation
AsyMIstomatic 9
.

o
Harmless .
Meningocele

Meningomyocele
Congenital Talipes Equinovarus
Cleft Palate And Cleft Lip
Birth Initial assessment, presurgical evaluation and ortho- dontics
• 3–6 months Primary lip repair
• 9–12 months Palate repair (before speech develops)
• 2 years Speech assessment
• 3–5 years Lip revisions
• 8–9 years Initial intervention orthodontics
• 10 years Alveolar bone grafting
• 16 years Nasal revision
• 17–20 years Orthognathic surgery.