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Fig. 198 Fig. 199
Fig. 200
TUBERCULOUS ARTHRITIS.
Tuberculous disease of the joints is one of the most frequent of
surgical lesions. It has produced characteristic appearances which
have been known under the name of “scrofula of joints,” until a
clearer recognition of the pathology of the condition led to the
abandonment of the term scrofula. Tumor albus, or white swelling,
was another term commonly applied to these lesions, because of the
anemic appearance of the surface of the swollen joint.
Tuberculous arthritis assumes different phases in proportion to the
involvement of the different component structures of the joint. Some
cases begin purely as a tuberculous synovitis, and may for a long
time be limited to the synovial structures. Others begin within the
spongy texture of the expanded joint ends of the long bones, the
disease spreading from such foci and involving everything in the
path which its products take in the effort to secure spontaneous
evacuation, products of softening and infection travelling in the
direction of least resistance.
It has been the writer’s custom to always follow Savory, in his
suggestion to students to let their mental pictures of consumption of
the lungs and pleuræ serve for illustration in similar disease of joints.
Thus the cancellous bone structure much resembles the lung tissue
in its spongy character. In both a capsule surrounds the mass of
tubercle, and in each, by breaking down of its contents, a cavity is
formed. Moreover, the pleura bears practically the same
resemblance and relation to the lung and the chest wall that the
synovialis does to the bone end and the joint cavity; as we may have
pleuritis with phthisis, so we may have synovitis with tuberculous
ostitis; and as adhesions tend to form in the pleural cavity, so also do
they in the synovial cavity. Furthermore, in each case obliteration of
deeper veins causes the more prominent appearance of the
subcutaneous veins, and as tuberculous pleurisy often terminates in
empyema, so does tuberculous hydrarthrosis often terminate in
pyarthrosis, perhaps with fungous ulceration. In almost every
feature, then, the progress and effect of tuberculosis in the lung and
bone end may be likened to each other.
In some clinics bone and joint tuberculosis constitute nearly one-
third of the total of cases treated. Joints of the lower limb are the
ones most frequently involved in children, while in the adult those of
the upper extremity are generally attacked. It is not often that more
than one joint is involved at one time. The relation of traumatism to
this disease has been frequently discussed, and is variously
regarded. The disease is more common in those who are
predisposed to it by environment or by heredity, in the latter case
hereditary evidences usually being well marked. In such predisposed
individuals, especially in the early years of life, severe injuries are
usually promptly repaired, while the milder traumatisms, which are
often frequent and to which too little attention is paid, seem often to
so far lower tissue resistance as to favor an infection to which the
individual is already favorably predisposed. The true position to take,
then, would appear to be this, that traumatisms rarely lead directly to
joint tuberculosis, but only indirectly by affecting tissue susceptibility.
Thus lesions which begin in the epiphyses lead to what is known
as osteopathic joint disease, while those which have their origin in
the synovia give rise to the arthropathic forms. The former are more
common in children and the latter in adults (Fig. 202).
Pathology.—In regard to the pathology of these conditions it does
not vary from that mentioned in the earlier portion of
this work in connection with the general subject of Surgical
Tuberculosis. The deposit of tubercle in the tissue whose resistance
has been weakened is followed by the formation of granulation
tissue, which, so long as the germs survive, tends to increase and to
make room for itself at the expense of surrounding tissue. At the
same time there occurs a tissue struggle by which the attempt is
made to throw around an active focus a protecting barrier, which in
soft tissues consists of condensed fibrous and connective tissue,
and, in bone, of a sclerotic capsule, as though the intent were to
imprison the disturbing cause, and, by completely enclosing it, effect
protection. When this attempt at encapsulation is successful
spontaneous recovery follows. It will be made successful, to some
extent at least, by treatment whose most important local feature is
physiological rest. On the other hand, when the attempt is
unsuccessful and the barrier is transgressed by granulation tissue,
the lesion will advance in the direction of least resistance, while its
progress will be made known, especially as it approaches the
surface, by very significant signs: adhesion of the overlying
structures and finally of the skin, with purplish discoloration of the
latter. Finally softening occurs with escape of granulation tissue,
which, so soon as it is freed from pressure, will grow more luxuriantly
and with more color, constituting the fungous granulation tissue, to
which German pathologists so often allude, or so-called “proud
flesh.” When this appears upon the surface it is soon infected with
pyogenic organisms, breaks down, and an abscess cavity results,
connecting with the original focus and its extensions. This may be so
placed as to lie outside the joint capsule, which, in some respects, is
fortunate for the patient. The joint function may then be
compromised to only a minor degree.
Fig. 202
Often the direction of least resistance is toward the joint cavity, this
fungous tissue loosening and perforating cartilage or periosteum
before it enters the joint. Having penetrated it again it grows
extensively until the cavity is distended, its rapidity of growth
diminishing with the degree of pressure produced by its
surroundings. This pressure will also make it less vascular, and
when such a joint is opened it at first appears pale and anemic. In
proportion as the joint distends it loses in motility, while should
recovery occur spontaneously or as the result of treatment this tissue
will to some extent disappear, to be replaced by adhesions by which
pseudo-ankylosis is produced. The extent of the intra-articular
involvement will cause obstruction to the deeper return circulation,
and thus is brought about the prominence with which the
subcutaneous veins appear. The degree of hydrarthrosis is
apparently not limited except by the distensibility of the joint. In the
articular or arthropathic forms there is always more or less synovial
outpour.
Fig. 203
Tuberculous panarthritis. (Ransohoff.)
Fig. 204
Fig. 207