Professional Documents
Culture Documents
Oxygenation Reviewer
Oxygenation Reviewer
Staggering statistics
• Pulmonary Diseases
•Lung CA -
•TB – •Pneumonia –
•Chronic Airflow Limitation (formerly COPD) –
Staggering statistics
•Cardiovascular Diseases – # 1 killer
•HTN – 65 million Normal Oxygenation Process
•Artheriosclerosis Inspiration
•Arteriosclerosis • Diaphragm and intercostal muscles contract
•Stroke • Thoracic cavity size increases
•Hypercholesterolemia • Volume of lungs increases
• 107 million - a risk factor for CVD • Intrapulmonary pressure decreases
•AMI – 7.5 Million per year, 460,000 die • Air rushes into the lungs to equalize
pressure
• Americans paid 393.5 billion in 2005 for
CVD related medical costs Expiration
•Diaphragm and intercostal muscles relax
•Lung volume decreases
•Intrapulmonary pressure rises
•Air is expelled
Gas Exchange
• Occurs after the alveoli are ventilated
• Pressure differences (gradient) on each
side of the respiratory membranes affect diffusion
• Alveoli:
Respiratory System
• PO2 100mmHg
Process of Breathing
• PCO2 40mmHg • Venous blood:
•Inspiration
• PO2 60mmHg
•Air flows into lungs
• PCO2 45mmHg
•Expiration
• O2 diffusion from alveoli pulmonary blood
•Air flows out of lungs
vessels
Normal Oxygenation Process
• CO2 diffusion from pulmonary blood vessels
• Cardiovascular: alveoli
Adequate O2 Balance
Normal
Oxygenation Process
• Systemic:
• Maintenance of adequate O2 balance Gas • Flaring of nares
Exchange
• Substernal or intercostal retractions
Oxygen Transport
• Cyanosis
• Transported from the lungs to the tissues
Abnormal Respiratory Patterns
• 97% of O2 combines with RBC Hgb
•Tachypnea (rapid rate)
oxyhemoglobin carried to tissues
•Bradypnea (abnormally slow rate)
• Remaining O2 is dissolved and transported
•Apnea (cessation of breathing)
in plasma and cells (PO2)
•Kussmaul’s breathing
Normal Oxygenation Process
•Cheyne-Stokes respirations
• Cell environment / O2 carrying capacity:
•Biot’s respirations
• O2 Carrying capacity of blood is expressed
by:
• HEART • Wheezes
• Irreversible (no cure) -.. cases vary from U – unable to tolerate activity
from mild – severe
N – nutrition poor (Weight loss esp. emphysema)
G – Gases abnormal (PCO2>45 and PO2 <90..
• Managed w/ lifestyle and medications respacidosis
D – dry or productive cough esp. chronic bronchitis
TYPES:
COPD used now “catch all” term for diseases that Complications in COPD
limit airflow
1. Heart diseases: heart failure
Chronic Bronchitis (blue bloaters) – tend to
2. Pneumothorax: spontaneous air sacs
have cyanosis due to low of oxygen and tend to
edema. 3. Lung Infection : PNA
- Hyperinflation/ became inflames/ produce 4. Increased risk of lung cancer
mucus/narrowing of the air sacs
- Abnormal diaphragm (flattens)
DIAGNOSED
- Low amount of oxygen (cyanotic) ->
1. Spirometry – a test where a patient
increasing the amount of RBCs (causes the blood
breathes into a tube which measures:
to become to thick) -> increase pressure in
pulmonary artery (pulmonary hypertension) back- How much colume the lungs can hold
flowing to the right side of heart -> affect the during inhalation
liver(incongestion) leads to left side failure and
bloating comes. How much and how fast air volume is
exhaled
Emphysema (Pink puffers) – hyperventilate
for compensation (lack of oxygen ) and maintain Measuring FVC (Forced Vital Capacity)- low
pink complexion. No cyanosis, Barrel chest from reading -> restrict breathing -> largest amount of air
accessory muscle usage) exhaled after breathing in deeply in one second
- (lack of oxygen, increases of CO2 retention) Measuring Forced Expiratory – volume: how
much air a person can exhale w/in 1 second low
- Alveoli sac lose elasticity due to inflames; reading shows severity
response in body-> air gets trapped in the sacs
- Hyperinflation (diaphragm to flatten) due to
retaining air volume
*used bronchodilator 1st (dilate airways)then
corticosteroid inhaler and RINSE mouth after use
cortico… due to developing thrush
NURSING INTERVENTIONS
Phosphodiestrace – 4inhibiters: “Roflumilast” (used
1. Monitor Respiratory System for ppl w/ chronic bronchitis and helps decreased
copd exarcebation… (not bronchodilator)
- Listening to lung sounds (may need suction)
Sideffect: cause suicide ideation, weight loss
- Monitor sputum production (collect if
ordered( @risk for PNA Methylxanthines: “Theophylline” (given orally many
times).. type of bronchodilator, used long term w/
- Keep O2 Sat 88-93% (pt w/ copd are pts w/ severe copd
stimulated to breathe due to low oxygen level rather
than high carbon dioxide levels.. but if give them *narrow therapeutic range : 10-20 mcg/mL :
too much O2, the breathing will stop -> increased Digoxin toxicity and low effects of
hypoventilate and CO2 will become toxic Lithiumt Dilantin
MEDICATIONS: REGIME
“Chronic Pulmonary Medications Save Lungs!”
Corticosteroids : decreased inflammation and
mucous production.. oral or IV, or inhaled.. it used
many times w/ bronchodilators
- Prednisone
- Solumedrol
- Pulmicort
- Symbicort : steroid and long acting
bronchodilator
Side Effects: easy bruising, hyperglycemia and
increased risk infection(long-term use- boneloss)