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Laporan Kasus RA1

ASITES
PENYAJI : - Margareth
Margareth Sitorus
- Meliani
- Ida Meita Sagala
- M. Kamal Hafiz
- Syed M. Kamal
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LEMBAR PENGESAHAN
Telah dibacakan pada tanggal : 7 Mei 2015
Nilai :
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ii

KATA PENGANTAR

Puji dan syukur kami panjatkan kehadirat Tuhan Yang Maha Esa, yang
telah memberikan berkat dan karunia-Nya sehingga penulis dapat menyelesaikan

laporan kasus ini dengan judul ASITES

Penulisan laporan kasus ini adalah salah satu syarat untuk menyelesaikan
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iii

DAFTAR ISI

Lembar Pengesahan.......................................................................................... i
Kata Pengantar ................................................................................................. ii
Daftar Isi ........................................................................................................... iii
Bab 1 Tinjauan Pustaka ................................................................................... 1
1.1. Latar Belakang ........................................................................................ 1
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BAB 1
TINJAUAN PUSTAKA
1.1 Latar Belakang
Asites adalah akumulasi cairan di dalam rongga peritoneum. Kata asites
berasal dari bahasa yunani askites dan askos yang berarti kantong atau perut.
Asites dapat disebabkan oleh banyak penyakit. Penyebab asites dapat digolongkan
ke cirrhotic asites dan non-cirrhotic asites1.
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disebabkan oleh tuberkulosis peritoneal, dan sisanya disebabkan oleh penyebab


lain seperti sindrom nefrotik dan penyakit pankreas 2,3.

2,4
1.4 Patofisiologi
1.4.1 Hipertensi portal → peningkatan tekanan vena porta merangsang pelepasan
sitokin vasodilator seperti NO, prostasiklin, adenosin, endotoksin) yang
selanjutnya mengakibatkan vasodilatasi perifer dan splanchnic. Vasodilatasi
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memudahkan terjadinya akumulasi protein dan albumin di rongga


peritoneum. Peningkatan tekanan osmotik ini menarik cairan dari
intravaskuler ke rongga peritoneum. Selain itu, tumor hepar yang besar yang
mengkompresi atau tumbuh ke dalam vena porta atau vena hepatik akan
mengakibatkan hipertensi portal dan asites.
 Peritoneal carcinomatosis
 Pseudomyxoma
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tidak bisa menyerap bilirubin. 17 Ikterus dapat menjadi penunjuk beratnya


kerusakan sel hati. Ikterus terjadi sedikitnya pada 60 % penderita selama
perjalanan penyakit.
b. Timbulnya asites dan edema pada penderita sirosis 12 Ketika liver kehilangan
kemampuannya membuat protein albumin, air menumpuk pada kaki (edema)
dan abdomen (asites). Faktor utama asites adalah peningkatan tekanan
hidrostatik pada kapiler usus . Edema umumnya timbul setelah timbulnya
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atau TB paru milier, menelan spuutm yang terinfeksi atau penyebaran


langsung dari kelenjar limfe, dan organ intraabdominal ( terutama ileum
terminal dan caecum)
Adenosin Deaminase (ADA) merupakan salah satu pemeriksaan yang dapat
digunakan untuk mendiagnosa TB Peritoneal dengan tingkat sensitifitas 97%
dan spesifisitas 98% . Adenosin deaminase adalah suatu enzim
aminohidrolase yang mengubah adenosin menjadi inosin dan terlibat dalam
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Kemudian difokuskan untuk mendeteksi penyakit hati kronis/sirosis hepatis


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Cairan asites dikategorikan menjadi eksudat dan transudat. Berikut adalah


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umumnya anekoik homogen, dan usus tampak bergerak bebas. 9 Asites yang
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merah akibat trauma akan bersifat heterogen dan akan membeku, tetapi jika
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10

Butt Chiari Syndrome Serositis


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11

Cirrhosis
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C. Large Volume Paracentesis (LVP)


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13

E. Peritoneovenosus Shunt (PVS) / LaVeen Shunt


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terapi pengganti spironolakton yang dapat digunakan adalah Amiloride. Dosis


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albumin tinggi seperti putih telur dan tidak mengkonsumsi zat yang bersifat
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