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FINAL Case Presentation (OB Ward)
FINAL Case Presentation (OB Ward)
FINAL Case Presentation (OB Ward)
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INTRODUCTION
CHAPTER I - PATIENT’S PROFILE
A. General Data
B. Admission Profile
C. History of Present Illness
D. Past Medical History
E. Heredo-Familial illness
F. Personal/Social History
CHAPTER IX - BIBLIOGRAPHY
INTRODUCTION
The infection can reach the GI tract by swallowing contaminated phlegm from a TB lung
infection or through the bloodstream and lymphatic system from the lungs. Less commonly, it
can be contracted by ingesting milk products contaminated with Mycobacterium bovis, a related
bacterium affecting cows. Gastrointestinal tuberculosis can affect several parts of the GI tract,
including the esophagus (rarely), stomach, small intestines, large intestines, rectum, and anus.
Common symptoms include abdominal pain, weight loss, fever, changes in bowel habits (more
commonly diarrhea than constipation), nausea, vomiting, dark tarry stools, paleness, anemia,
abdominal distention, ascites, enlarged liver and spleen, swollen lymph nodes, and an
abdominal mass. Between 6% and 38% of people with gastrointestinal tuberculosis also exhibit
lung symptoms such as a persistent cough with blood or mucus, exhaustion, high fever, night
sweats, unintentional weight loss, loss of appetite, and general sickness.
Risk factors for tuberculosis include close contact with individuals with gastrointestinal TB,
immigration from regions with high tuberculosis rates, working or spending time in hospitals,
homeless shelters, prisons, nursing homes, and residential homes for people with HIV.
Additional risk factors include smoking, heavy alcohol consumption, illicit drug use, and
conditions that weaken the immune system such as HIV, severe kidney disease, Crohn's
disease, head and neck cancer, and receiving an organ transplant.
CHAPTER I: PATIENT’S PROFILE
A. General Data
Name: A.P.
Gender: Male
Nationality: Filipino
Status: Married
B. Admission Profile
1-day PTC, vomiting 2x, abdominal pain, effectful food w/ associated fluke meals.
FH PTC, abdominal pain from epigastric area to RLQ then eventually quantized
10/10 radiate to back, resisted current consult @ER.
D. Past Medical History
Based on the interview, the patient has no past medical history, no allergies and
no current medication.
E. Heredo-Familial illness
During the interview, the presenters asked the patient if the patient family had a
history of illnesses or allergies. He said that his brother has gallstones disease,
his mother has hypertension and his father also has hypertension and stroke.
F. Personal/Social History
Patient A.P. was born on October 27, 1982, in San Pedro, Laguna. He has a wife
and three children living together under one roof. He is a company driver. He
said that he drinks and smokes occasionally. The patient stated that he has no
history of respiratory disorders or other related illnesses.
CHAPTER II - PHYSICAL ASSESSMENT
FOCUS FINDINGS
Skin + Pinkish
- Jaundice
Heart AP NRRR
Rectum/Genitalia Intact
Extremities +FEP
-Cyanosis
VITAL SIGNS:
10pm 38.5 - - - -
Digestive System
The digestive system, also known as the gastrointestinal (GI) tract, is a complex system responsible for
the breakdown, absorption, and assimilation of nutrients from food, as well as the elimination of waste
products. It comprises a series of organs that work together to facilitate these processes. It is responsible
for the breakdown and absorption of nutrients, water, and electrolytes while also serving as a vital barrier
against pathogens. Gastrointestinal tuberculosis (GI TB) occurs when Mycobacterium tuberculosis infects
the digestive tract, and its impact on digestive system physiology can be profound.
Mouth: Digestion begins in the mouth, where food is ingested, chewed, and mixed with saliva. Saliva
contains enzymes, such as amylase, which start the breakdown of carbohydrates.
Esophagus: After swallowing, food travels down the esophagus, a muscular tube that connects the
mouth to the stomach, through a process called peristalsis.
Stomach: The stomach secretes gastric juices containing enzymes and hydrochloric acid, which break
down proteins and kill bacteria. This creates an acidic environment. Muscular contractions mix food with
gastric juices, forming chyme, a semi-liquid mixture.
Small Intestine: Digestion continues in the small intestine with the help of enzymes from the pancreas
and bile from the liver. Nutrient absorption primarily occurs here, facilitated by villi and microvilli, tiny
finger-like projections that increase surface area.
Large Intestine: The large intestine absorbs water and electrolytes from the remaining chyme, forming
feces. It also houses beneficial bacteria that help ferment undigested carbohydrates, producing gases
and some vitamins.
Lymphatic System: The lymphatic system is integral to immune function. Lymph nodes, lymphatic
vessels, and lymphatic organs help filter and circulate lymph fluid, which carries white blood cells and
other immune cells to fight infections and remove toxins.
CHAPTER IV - PATHOPHYSIOLOGY
CHAPTER V - MEDICAL MANAGEMENT
TX (Treatment):
Piptozabactum: Given
before surgery to prevent
postoperative infections,
which can be more
severe in patients with
tuberculosis.
Paracetamol:
Tuberculosis can cause
fever and pain.
Paracetamol helps
manage these
symptoms.
Omeprazole: Patients
with gastrointestinal
tuberculosis may have
gastric involvement or
may be on medications
that can cause gastric
irritation. Omeprazole
helps reduce gastric acid
secretion and prevent
irritation.
For Laparoscopic
appendectomy: If the
patient has
gastrointestinal
tuberculosis and
presents with symptoms
suggestive of
appendicitis, an
appendectomy may be
indicated. However, it's
crucial to differentiate
between appendicitis and
other abdominal
pathologies that mimic its
symptoms, including
tuberculosis.
VS Q2: Tuberculosis can
cause fluctuations in vital
signs, and closely
monitoring them every 2
hours allows for early
detection of any
deterioration or
complications.
Dehydration is a concern
in patients with
gastrointestinal
tuberculosis, and close
monitoring of fluid intake
and output is essential to
prevent further
complications.
Please insert foley
catheter now: This
might be indicated if the
patient is unable to
urinate due to abdominal
distension or other
factors related to
gastrointestinal
tuberculosis, ensuring
adequate urinary
drainage during surgery.
Maintain JP drain,
measure output q4 and
record: Jackson-Pratt
drain maintenance and
monitoring of output
every 4 hours help
prevent fluid
accumulation and detect
any signs of infection or
bleeding.
Maintain IFC, measure
output q1 and record:
Intermittent Foley
catheter maintenance
and hourly monitoring of
urine output help assess
renal function and fluid
balance.
Regular wound care is
essential for preventing
infection and promoting
wound healing in patients
undergoing surgery for
gastrointestinal
tuberculosis.
VS q2 and record: Vital
signs are monitored
every 2 hours to assess
the patient's stability and
response to treatment.
I&O qshift: Intake and
output are measured and
recorded every shift to
monitor fluid balance and
renal function.
Close observation for
signs of abdominal pain,
hypotension (systolic
blood pressure less than
90 mmHg), tachycardia
(heart rate greater than
90 bpm), and decreased
urine output (less than 30
cc/hr) helps detect
complications such as
bleeding, sepsis, or renal
impairment.
A. Hematology
DIFFERENTIAL COUNT
Eosinophils 0.0-0.05
Basophils 0.0-0.01
Stabs 0.0-0.05
Date: 05-23-2024
DIFFERENTIAL COUNT
Basophils 0.0-0.01
Stabs 0.0-0.05
Date: 05-26-2024
B. URINALYSIS
Ph 5.0 5.0-7.5
Sugar +2 Negative
MICROSCOPIC EXAM
Date: 05-23-2024
C. ELECTROLYTES
RESULT REFERENCE VALUES
Date: 05-23-2024
D. CLINICAL CHEMISTRY I
E. LABORATORY REPORT
Date: 05-23-2024
Route of adjustments to
n: IV s: Hypersensitivity
to paracetamol
: HCI (prodrug of
Analgesics paracetamol).
Antipyretics hepatocellular
insufficiency.
Brand Name: responsible for acid reflux. It's Diarrhoea peptic ulcer
Nursing Management
1. Patient Safety:
Verify patient identity and surgical site with the surgical team.
Position the patient safely and comfortably to prevent pressure injuries.
Maintain a sterile environment to prevent infections.
2. Monitoring:
Continuously monitor vital signs, including heart rate, blood pressure, oxygen saturation,
and temperature.
Monitor fluid balance, including input and output of fluids, to maintain hemodynamic
stability.
Assist the anesthesiologist in monitoring the patient's response to anesthesia.
3. Documentation:
Transfer the patient to the Post-Anesthesia Care Unit (PACU) for close monitoring.
Frequently monitor vital signs and compare them to baseline values.
Assess pain levels and administer prescribed pain medications.
Ensure the patient's airway is clear and monitor for signs of respiratory distress.
4. Documentation:
CHAPTER IX - BIBLIOGRAPHY
https://www.healthline.com/health/abdominal-tuberculosis
https://www.mayoclinic.org/diseases-conditions/tuberculosis/symptoms-causes/syc-20351250
https://www.ncbi.nlm.nih.gov/books/NBK556115/