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Clinical Protocols in the Treatment of Caries and Complication in Kids and Adolescents
Clinical Protocols in the Treatment of Caries and Complication in Kids and Adolescents
• Infected dentin - the fluorescence has an intense red or dark red color,
which covers the entire carious dentin;
• Partially infected dentin - selectively removed to soft dentin - pink
fluorescence with localized limited red fields in the area of suprapulp
dentin;
• Affected dentin – selectively removed to firm dentin – pale pink
fluorescence, localized only in small fields from the bottom of the cavity (in
the area of the suprapulp dentin), and in the rest of the cavity lack of
fluorescence;
• Healthy dentin - non-selectively completely removed carious dentin - lack
of red fluorescence.
Protocols for Diagnostics
and Treatment of Deep
Dentinal Caries Lesion:
D3b (Peneva)/ code 05 ( ICDAS II)
D4 (Peneva)/ code 06 ( ICDAS II) – closed asymptomatic pulpitis
D3b (Peneva) code 05 - ( ICDAS II)
dentin caries affecting the dentin in the 0-no sealant or filling
inner half of the dentin without affecting 5-cavitation with visible carious dentin
the pulp
D4 (Peneva) code 06 ( ICDAS II)
dentin caries affecting the dentin in the 0-no sealant or filling
inner half of the dentin, affecting the pulp 6- Extensive cavitation (diffuse, wide) with
visible carious dentin
• size of the carious lesion - deep and extensive carious lesions responding the
following clinical criteria:
• occlusal lesion – deep and extensive dentin lesions, involving the last 1/4
of the suprapulp dentin, involving more than ½ of the adjacent cusp (s);
• approximal lesion – cavitated dentin lesion:
• in vestibulo-oral direction - reaching a self-cleaning surface;
• in medio-distal direction - reaching and exceeding the middle of an
adjacent cusp;
• in depth, reaching the last ¼ of the dentin.
• cervical lesion - has direct visibility; deep and extensive carious lesion in
the dentin.
Closed asymptomatic pulpitis /carious lesion D4 (Peneva) code 06 (ICDAS II)
– Extensive cavitation (diffuse) with visible carious dentin;
• Periphery of the carious lesion – cavitated from the occlusal, with fractured
and undermined enamel edges with transparent underlying carious dentin;
• color and consistency of the carious dentin – they determinate the rate of
the carious process;
• without pulp exposure;
• X-ray control (intraoral Ro gr of the tooth) – the carious lesion reaches the
innermost ¼ of the dentin.
Differential diagnosis between:
• Dentin caries D3a – in the outer half of the dentin и D3b in the
inner ½ of the dentin (Peneva) – according to the described criteria
determining the size of the lesion;
• Deep dentin caries and closed asymptomatic pulpitis – according
to the described crteria;
• Reversible and irreversible pulpitis - absence or presence of
spontaneous night pain;
• Closed and opened pulpitis – absence or presence of pulp
exposure.
Treatment protocol for carious lesion
D3b (Peneva)/ code 05 ( ICDAS II)
• Contraindications: In immunocompromised
patients, organ transplant patients.
Diagnoses:
• Symptomatic apical periodontitis (Periodontitis symptomatica apicalis) –
occurrence of specific periodontal (in childhood – periapical) pain: constant
severe pain which is the reason for the visit in the dental office, pain during
percussion, pressure and chewing. The mucosa which covers the periapical
region is painful. The tooth feels higher than the other teeth and the contact
with the antagonist is painful.
• Acute apical abscess (Abscessus apicalis acuta) – quick appearance,
spontaneous pain, pain under pressure, rapid development, spontaneous
pain, pressure pain, pus formation and swelling of the tissue around the
affected tooth. Depending on the jaw in which the tooth is located, the
swelling may involve the sublingual area or a palatal abscess may form. The
swelling may affect the cheeks and orbit.
Diagnoses:
• Asymptomatic periapical disease (Periodontitis asymptomatica apicalis) the
symptom "periodontal pain" is not present, without prolonged pain, there is no
pain or slight pain on percussion, palpation and pressure. There is no erythema of
the mucosa in the periapical area. Such apical periodontitis may not be noticed by
the patient and can be found accidentally during examination or on routine x-ray.
The data that the tooth is non-vital, as well as the stage of root development of the
contralateral tooth are important.
• Chronic apical abscess (Abscessus apicalis chronica) - prolonged beginning, mild to
absent discomfort with gradual release of the pressure through a formed sinus
tract. It may be the result of a naturally occurring exit of purulent exudate in an
acute process that leads to chronicity. The fistula is a typical feature of chronic
periapical abscess. It passes through the periapical structure, the bone, perforates
the periosteum and corticalis, and then the soft tissues around the root of the
tooth. It provides continuous drainage of the accumulating exudate in the oral
cavity. In very rare cases, the sinus tract can come out extraorally through the skin.
Necessary requirements:
• Correct diagnosis – guarantee for the treatment success;
• The tooth should be restorable (enough tooth structures to
ensure the stability and the hermeticism of the filling)
• Diagnostic X-ray– gives information for the localisation of the
lesion, the stage of the root development, it is used later for
control;
• Maximum good isolation of the field;
• Use of sterile instruments.
Treatment protocol:
Apexification with calcium hydroxide:
• The procedure requires multiple visits and assistance from the patients
and their parents, as it may take a year or more to achieve the formation
of an apical barrier to allow the root canal obturation.
• Calcium hydroxide helps disinfection of the root canal, as it increases the
dissolution of necrotic tissue when used alone or in combination with
NaOCl. The high pH and solubility of calcium hydroxide preserves its
antimicrobial effect in the root canal for a long time.
• The barrier is formed by cells originating from the adjacent connective
tissue. It is histologically porous and may consist of cementum, dentin,
bone or osteodentin.
Treatment protocol for asymptomatic apical
periodontitis and chronic apical abscess – first visit
• Local anesthesia and isolation with rubber dam;
• Opening an endodontic cavity to provide access to the entire surface of the
tectum pulpae for the respective tooth;
• Complete removal of the infected dentin first from the walls of the cavity and
then from the bottom;
• Rinsing and drying the cavity before opening the pulp;
• A thin sterile fissure burr enters the communication with the pulp and the tectum
of the pulp is removed along its contours until full access to the pulp cavity;
• Mechanical and chemical treatment of the root canal. Multistage and gradual
cleaning is performed with a K-file, under irrigation with antiseptic solutions -
2.5% sodium hypochlorite and EDTA;
• Determination of the working length radiographically. Apexlocators are not
reliable in cases of immature teeth;
Treatment protocol for asymptomatic apical
periodontitis and chronic apical abscess – first visit
• Disinfection of the root canal:
• careful irrigation with 2.5% sodium hypochlorite with a syringe and a needle with a
side opening, in order to dissolve the necrotic pulp tissues;
• The needle should not tighten in the root canal and the working length is shorter to
avoid extrusion of solution through the apical foramen;
• Passive ultrasonic activation of the solution is recommended to make the removal of
necrotic tissues easier.
• Irrigation with saline solution;
• Drying the canal with sterile paper points;
• Placing antiseptic dressing in the root canal in 1, 2 or more visits, in
combination with irrigation according to the clinical situation (exudation,
gangrenous odor from the canal).
Treatment protocol for asymptomatic apical
periodontitis and chronic apical abscess
SECOND VISIT
• Irrigation of the canal and application of non-hardening calcium
hydroxide (in length shorter than the working length) with a cannula, and
pressing with a sterile cotton pellet;
• Temporary filling which ensures hermeticism for a longer period of time
(GIC, IRM).
NEXT VISIT: 2-3 WEEKS LATER;
• Disinfection of the root canal;
• Application of new portion of calcium hydroxide paste in the root canal,
apically from the enamel-cement junction;
• Long-term sealing of endodontic access.
Control visits:
• The tooth is observed clinically and radiographically at 3-
month intervals: during these 3 months it may be necessary
to refill the tooth with calcium hydroxide, because the paste
is absorbed very quickly!
• the filling of calcium hydroxide paste;
• presence of an initial barrier;
• thickening or possible elongation of the root walls;
• condition of the periapical bone and periodontal space.
NB!
• If the radiograph shows that calcium hydroxide has been resorbed from
the canal, the procedure is repeated. If the canal remains filled and
partial progress is seen in the formation of the apical septum, the tooth is
left for another three months.
• The procedure is repeated every three months until the final formation of
a hard apical septum, until thickening or elongation, formation of
periodontal space and formation of lamina dura of the alveolar bone.
• The barrier can be formed coronally from the apex of the root and in this
case the working length is reduced. This septum should not be perforated
to fill the canal to the apex, as the tissue that forms it is healthy.
• After the formation of the apical barrier, the canal is filled with gutta-
percha and a sealer.
Apexification with МТА:
Advantages:
• Shorter treatment time – apexification in one visit;
• Prevents microleakage;
• Possibility to harden in a humid environment;
• Reduced costs and clinical time.
Clinical protocol:
• First visit – the same as for the apexification with calcium hydroxide.
• Second visit – after 2 weeks.
• Isolation with rubber dam, irrigation;
• Drying the root canal with paper points;
• Application of collagen matrix apically (optional);
• Placement and compaction (using ultrasound) of MTA in the apical part of
the root (4-5 mm) - the placement of MTA in the apical part is more
complicated than the use of calcium hydroxide. The material is placed with
the help of special endodontic pluggers and is compacted by indirect
ultrasonic activation. A moist cotton pellet or paper point is placed on the
MTA, providing moisture for it to harden, and the tooth is sealed with a
temporary filling.
Third visit:
• After few days;
• The hardness of the cement is checked with an endodontic instrument;
• If the material is not hardened - a new one must be applicated;
• After hardening - filling of the root canal and restoration;
• The short setting time (~ 10 min) of some new generations of calcium
silicate cements may shorten the treatment period, allowing the
placement of an apical plug, root canal filling and restoration of the
tooth in one visit.
Treatment protocol for
symptomatic apical periodontitis:
First visit:
• After diagnosis:
• If the tooth has a filling, it should be removed;
• If the tooth is not treated, an endodontic cavity is opened and the carious
tissue is removed;
• Removing the roof of the pulp cavity;
• Serous or purulent exudate, and sometimes bloody exudate comes out;
• Cleaning the necrotic tissue of the cavum;
• Irrigation with sodium hypochlorite;
• If possible, the tooth is sealed hermetically.
The next visit
• Is on the following day. Again the same cleaning and disinfection
are done. These procedures are applied until the complete
disappearance of clinical symptoms.
• Sealing the tooth - after the protocol for disinfection of the root
canal, the tooth is left with an applicated drug in the root canal -
Ledermix, triple antibiotic paste or Indextol. It is sealed with
temporary filling;
The next visit
• In case of successful sealing without complaints, one of the
apexification techniques is applied.
• After achieving the apexification, a routine root filling and
restoration of the tooth with permanent filling is done.
Treatment protocol for acute
apical abscess:
Clinical protocol
• Drainage of the tooth - opening the pulp cavity, cleaning and creating a way to drain
the exudate from the root canal and periapical area. The tooth is left open;
• In the submucosal phase - incision for drainage of purulent exudate from the soft
tissues, leaving drainage;
• Influence of the general condition:
• a broad-spectrum antibiotic is required to affect the anaerobic microflora;
• Sultamicillin – suspension 250 mg/5ml - dose for children is per kilogram of body
weight for 24 hours - 100-150 mg in three doses;
• The best for periapical abscess is Amoxicillin/Clavulanic acid – 25-50-80 mg/ kg/24 h in three doses
+ Metronidazole - 20-30 mg/ kg/weight in 3 doses for 24 hours;
• Clindomycin - suitable for Gr(-), Gr(+) cocci and anaerobic microflora - 15–45 mg per kg/weight for
24 hours in 3-4 doses with 200 ml of water;
• Lincomycin - 30-60 mg/kg/weight for 24 hours in 3-4 doses.
• Cefador (cephalosporin II generation) - 20-30 mg per kg/weight for 24 hours in 3 doses before
meals; Antipyretics for normalizing the temperature;
• Inclusion of probiotics, vitamins and hydration with liquid intake;
Clinical protocol
• Irrigation of the root canal and monitoring of the general condition is
done every day until the symptoms disappear. This usually happens after
the fifth or seventh day.
• Irrigation and disinfection of the root canal;
• Placing antibiotic paste in the root canal;
• Apexification technique according to the described protocol.