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Download Color Textbook of Histology 3rd Edition Gartner Test Bank all chapters
Download Color Textbook of Histology 3rd Edition Gartner Test Bank all chapters
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Gartner & Hiatt: Color Textbook of Histology, 3rd Edition
Test Bank
MULTIPLE CHOICE
b. T helper cells
c. T killer cells
d. B memory cells
e. B effector cells
Explanation:
The answer is a. The innate immune system relies on natural killer cells, macrophages, neutrophils, and
complement and Toll-like receptors. T cells and B cells belong to the adaptive immune system.
b. T helper cells
c. neutrophils
d. macrophages
e. complement
Explanation:
The answer is b. The adaptive immune system relies on T cells and B cells. The innate immune system
relies on natural killer cells, macrophages, neutrophils, and complement and Toll-like receptors.
c. it exhibits specificity
e. it has diversity
Explanation:
The answer is d. The innate immune system relies on Toll-like receptors for initiating inflammatory and
immune responses. The adaptive immune system is slower acting, has immunologic memory, exhibits
specificity, and has diversity.
4. A child 6 years of age sees his pediatrician for repeated infections and developing blood in his
diarrhea. The physician orders a complete blood count, and the results show very low platelet count and
a low lymphocyte levels. Because the little boy also has eczema, the pediatrician suspects Wiskott-
Aldrich syndrome. Children with this disease
Explanation:
The answer is d. Children with Wiskott-Aldrich syndrome have low T and B cell counts. They are at very
high risk for infection and they usually do not survive into their teens. Many of those who reach their
teenage years succumb to leukemia or lymphoma or other cancers. Because their T and B cell counts are
very low, these children are prone to respiratory infections and are also easy targets for viral, fungal, and
bacterial infections. Only boys are susceptible to Wiskott-Aldrich syndrome.
a. thymus
b. lymph node
c. spleen
d. pharyngeal tonsil
e. palatine tonsil
Explanation:
The answer is a. The thymus and bone marrow are the two primary lymphoid organs because it is in
those two regions that T cells and B cells, respectively, become immunocompetent. After the
lymphocytes become immunocompetent, they migrate to the secondary (peripheral) lymphoid organs:
the lymph nodes, diffuse lymphoid tissues, tonsils, and spleen.
a. effector cells
b. plasma cells
c. memory cells
d. virgin cells
e. naïve cells
Explanation:
The answer is c. Memory cells undergo cell division after first exposure to an antigen, so that the number
of memory cells in that particular clone is increased. Because of the presence of an expanded population
of memory cells with an increased affinity for the antigen, subsequent exposure to the same antigen
induces a secondary (anamnestic) response that is much faster, more potent, and longer lasting than the
primary response. Effector cells are responsive to the antigenic challenge, and plasma cells manufacture
antibodies. Virgin cells (also known as naïve cells) are B and T cells that have not yet been exposed to
antigens. None of these cells are responsible for clonal expansion.
7. Graves’ disease is an autoimmune disorder in which the person forms antibodies against his or her
b. thyroxine
c. thyroid gland
d. parathyroid gland
e. TSH receptors
Explanation:
The answer is e. Patients with Graves’ disease form antibodies against the TSH receptors of their own
follicular cells. These persons produce excess thyroid hormone and therefore have an enlarged thyroid
gland and exophthalmos (protruding eyeballs). They do not form antibodies against their TSH or against
thyroxine. Their thyroid and parathyroid glands are not perceived to be antigens.
8. The membrane attack system is the body’s innate defense system against microbial attack. Which of
the following components of the complement system plays a key role in this defense?
a. C2
b. C3
c. C4
d. Factor B
e. Factor D
Explanation:
The answer is b. C3 plays a key role in the complement system, because deficiency of this protein leads
to recurring bacterial infection. C3 can directly activate complement by its interaction with bacteria or
even bacterial toxins. C2 and C4 deficiency can result in an autoimmune disease. Factors B and D are
responsible for initiating the alternative pathway.
9. A patient is producing an antibody isotype that is composed of a dimer held together by J protein and
is secreted into his saliva. Which of the following isotypes is being produced by this patient?
a. IgA
b. IgD
c. IgE
d. IgG
e. IgM
Explanation:
The answer is a. IgA, also known as the secretory antibody, is composed of a dimer that is held together
by a J protein. IgA is secreted into the gut lumen, saliva, tears, milk, nasal mucus, and the vagina. IgD is a
surface immunoglobulin that B cells secrete onto their own cell surface. IgE is the reaginic antibody and
is present on the surface of mast cells and basophils. Cross-linking of IgE by antigens on basophils and
mast cells facilitates the release of pharmacologic agents from these cells. IgG crosses the placenta and
is secreted into milk; it protects the fetus and the neonate by providing them with passive immunity. It is
also responsible for antibody-dependent cell-mediated cytotoxicity by activating natural killer cells. IgM
is a pentameric form of the antibody and is the first isotype to be formed during a primary response.
a. TH0
b. TH1
c. TH2
d. T reg
e. CTL
Explanation:
The answer is e. CTLs (cytotoxic lymphocytes; T killer cells) possess CD8 molecules on their surface, and
they can interact with major histocompatibility complex I (MHCI) molecules on virally altered cells. If
their T-cell receptors recognize the epitopes displayed by the MHCI molecule on these virally altered
cells, then the cytotoxic T lymphocyte kills that cell. All T-helper cells and T reg cells possess CD4
molecules on their cell surfaces, and they can recognize MHCII molecules located on antigen-presenting
cells and B cells.
Explanation:
The answer is e. Type VI reticular cells compose thymic corpuscles. Type I and type III reticular cells are
located in the thymic cortex. Type I cells are responsible for the formation of the blood-thymus barrier,
whereas type II reticular cells subdivide the thymic cortex into lymphocyte-filled compartments. Type IV
reticular cells are found in close association with type III reticular cells, and they form the
corticomedullary junction. Type V reticular cells form the cytoreticulum of the medulla.
a. lymph node
b. lymphoid nodule
c. spleen
d. thymus
e. pharyngeal tonsil
Explanation:
The answer is c. The marginal zone is the interface between the white pulp and the red pulp of the
spleen and is the first place in the spleen where antigens are permitted to interact with the parenchyma
of the spleen.
Explanation:
The answer is e. The region delineated by the circle is the white pulp of the spleen, as is evident by the
surrounding red pulp and the dense, irregular collagenous connective tissue capsule that sends
trabeculae into the substance of this organ. Lymph nodes are usually surrounded by fat, and in the
cortex a subcapsular sinus and cortical lymph nodules would be evident. The medulla of the lymph
nodes has no lymphoid nodules, and medullary cords and medullary sinuses would be present. The
thymic cortex and the thymic medulla have no lymphoid nodules.
Explanation:
The answer is b. This photomicrograph represents the medulla of a lymph node, as is evident by the
presence of the medullary cords, medullary sinuses, and trabecula and the absence of the cortical
lymphoid nodules. The cortex of the thymus has dense accumulations of thymocytes, and the medulla of
the thymus is dominated by Hassall corpuscles (thymic corpuscles). The white pulp of the spleen has
lymphoid nodules and periarterial lymphatic sheaths.