KAPITA SELEKTA

Bagian/SMF Ilmu Kesehatan Kulit dan Kelamin

Fakultas Kedokteran Universitas Syiah Kuala
RSUD Dr Zainoel Abidin Banda Aceh 2023

September 2023
ANATOMI KULIT
EFLORESENSI
IMUNOLOGIC
SKIN DISEASE
CUTANEUS LUPUS ERYTHEMATOSUS

SLE DLE
SCLERODERMA

salt-and-pepper salt-and-pepper Raynaud phenomenon

hyperpigmentation hypopigmentation in early disease
VIRAL
INFECTION
VERRUCA VULGARIS hyperkeratosis

Virus : Human Papillomavirus (HPV)

Marks : hyperkeratosis, papillomatosis,
acanthosis, koylocytes.

acanthosis

koylocytes
CONDYLOMA ACCUMINATA
Virus : HPV-6 / HPV-11
MOLLUSCUM CONTAGIOSUM
VARICELLA
Virus : Varicella virus (herpes virus family)

Clinical Features
•Fever •Backache Typical cases of varicella in a 4-year-old children.
Note the presence of lesions in all stages of
•Chills •Sore throat and dry cough development and erosions at sites of excoriation

•Malaise (in some people)

•Headache •Rash crusted lession
•Anorexia •Pruritus

pustules
HERPES ZOSTER
Virus : Varicella Zoster Virus
MORBILI (MEASLES)
Measles virus of the Paramyxoviridae family

Clinical Features
•Fever (as high as 40.5°C )

PRODROME
•Malaise
•Conjunctivitis
•Coryza
•Cough (brassy or barking)
•Koplik spots (pathognomonic enanthem)

EXANTHEM
Nonpruritic, erythematous macules and papules
progressing in a cranial-to-caudal direction
Begins on the forehead and behind the ears and
spreads to involve the neck, trunk, and extremities
The rash usually peaks within 3 days
Desquamation and brownish dyspigmentation in
fair patients can occur as the rash resolves
RUBELLA erythematous macules

Virus : Rubella virus (an enveloped positive-

stranded RNA virus in the Togaviridae)

Clinical Features
Pruritic pink to red macules and papules (on the
face, neck, trunk, and extremities)
Lesions on the trunk may coalesce
Lesions on the extremities often remain discrete
The rash usually begins to disappear in 2 to 3 days,
unlike rubeola, which can be more persistent.
Desquamation may follow the resolution of the
rash

erythematous papule
HERPES SIMPLEX
Virus :
•Herpes Simplex Virus Type 1 (HSV1)
•Herpes Simplex Virus Type 2 (HSV2) erosions or crusts
lesions
HAND, MOUTH, AND FOOT DISEASE
lesions are oval, linear,
or crescentic, and run
parallel to the skin
lines on the finger

erythematous papules
BACTERIAL
INFECTION
IMPETIGO BULLOSA IMPETIGO KRUSTOSA

Etiologic agent : S. aureus Etiologic agent : S. aureus / GAS

ECTHYMA
Etiologic agent : S. aureus

classically evolves from

untreated impetigo occluded by grayish-yellow
footwear and clothing and crust and pus
extends more deeply, penetrating
the epidermis and producing
“punched-out” ulcers
occurs most commonly on the
lower extremities of children,
neglected elderly patients, and
individuals with diabetes
SUPERFICIAL FOLLICULITIS
= follicular or Bockhart impetigo

A small, fragile, dome-shaped multiple pustules

pustule occurs at the infundibulum
of a hair follicle
PYOGENIC PARONYCHIA
Etiologic agent : S. aureus
Others :
Candida, Pseudomonas, Streptococcus,
dermatophytes

abscess formation

red and tender soft tissue

 FURUNCLE & CARBUNCLE
A furuncle usually from a preceding, more superficial
folliculitis and often evolves into an abscess
The carbuncle involved area is red and indurated, and
multiple pustules soon appear on the surface, draining
externally around multiple hair follicles

multiple furuncles

nodular lesion covered

by purulent crust

small pustules
CELLULITIS
Etiologic agent :
Group A β-hemolytic streptococci (Streptococcus
pyogenes) and staphylococci (particularly S. aureus)
are the most commonly identified pathogens
ERYHTRASMA
Etiologic agent :
Corynebacterium minutissimum

Corynebacterium minutissimum produces

coproporphyrin III and a coral red fluorescence
that can be seen on Wood’s lamp evaluation.

reddish-brown
patches in axilla

yellowish
hyperkeratosis
ERYSIPELAS
CUTANEUS TUBERCULOSIS
Etiologic agent :
Mycobacterium tuberculosis
LEPROSY
Etiologic agent :
Mycobacterium leprae
SYPHILIS
Causative agent :
Treponema pallidum subspecies pallidum SECONDARY

PRIMARY chancre
FUNGAL
INFECTION
PTYRIASIS VERSICOLOR
Causative agent : hypopigmented patch
Malassezia furfur and related fungi.

hyperpigmented patch
TINEA FACIALIS, CORPORIS, CRURIS
Causative agent :
T. rubrum, T. mentagrophytes, or M. canis

Typical annular rings

TINEA FACIALIS TINEA CORPORIS TINEA CRURIS

TINEA CAPITIS NON-INFLAMATORY TYPE
(GRAY-PATCH)

TYPES CAUSATIVE AGENT

A large, round, hyperkeratotic

plaque of alopecia
TINEA CAPITIS INFLAMATORY TYPE

BLACK DOT TYPE

black dot
TINEA BARBAE KERION TYPE
SUPERFICIAL TYPE

Sharply demarcated red edematous nodule

Scattered follicular papules, studded with multiple yellowish weeping
pustules, and small nodules pustules. Note hairs have been lost from this
nodule
TINEA MANUS

Causative agent :
Trichophyton rubrum
Trichophyton interdigitale
Epidermophyton occosum

TINEA MANUS & TINEA PEDIS

TINEA PEDIS
TINEA PEDIS INTERDIGITAL TYPE

TYPES CAUSATIVE AGENT

opaque white scales and erosions

TINEA PEDIS
VESICOBULLOUS
MOCASSIN TYPE TYPE

patchy erythema and scalling distribution ruptured bullae, erosions,

and erythema
CANDIDIASIS
INGROWN NAIL
INSECT BITES/INFESTATION
PEDICULOSIS CAPITIS & PUBIS
SCABIES
Causative agent : Sarcoptes scabeii
CUTANEUS LARVA MIGRANS

creeping eruption
FILARIASIS
DERMATITIS EKSIM
DERMATITIS KONTAK IRITAN
DERMATITIS KONTAK ALERGIKA
DERMATITIS ATOPIK
LESI ERITRO-SQUAMOSA
SEBORRHOIC DERMATITIS
PSORIASIS VULGARIS
PITYRIASIS ROSEA
KELAINAN KELENJAR
SEBASEA DAN EKRIN
ACNE VULGARIS

MILD MODERATE SEVERE

ROSASEA
MILIARIA CUTANEUS XEROSIS
HYPERHIDROSIS
KELAINAN PIGMENTASI
VITILIGO
ALLERGIC SKIN DISEASE
URTICARIA
URTICARIA

DERMOGRAPHISM
 INFLAMATORY
NON-INFECTION
LICHEN PLANUS
Lichen planus is an idiopathic T
cell–mediated process without a
clear autoantigen.
The lesions are well-marginated,
flat-topped, red-violet polygonal
papules.
The distribution is symmetrical and
grouped lesions affect the flexural
aspects of the arms and legs.

Flat-topped, polygonal, sharply defined papules

of violaceous color, grouped and confluent.
ANNULAR GRANULOMA
LOCALIZED TYPE
 GENERALIZED TYPE
ANNULAR GRANULOMA
PERFORATING TYPE
HAIR
ANDROGENIC ALOPECIA
ANDROGENIC ALOPECIA
ALOPECIA AREATA
TRAUMA
BURNS
BURNS
 REFERENCES
Kang S, Amagai M. 2019.Fitzpatrick’s Dermatology 9th Edition Volume 1.
Mc Grow Hill Education
Lipsker, Dan. Clinical Examination and Differential Diagnosis of Skin
Lesions. Springer
Goodheart, Herbert P. Goodheart’s Photoguide to Common Pediatric
and Adult Skin Disorders 4th Edition. Wolters Kluwer
James, William D. Andrews’ Disease of The Skin Clinical Dermatology.
Saunders
THANK YOU