0056XF00339756_980475v

DIAGNOSTIC REPORT
MC-2535
PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

CODE/NAME & ADDRESS : C000021105 ACCESSION NO : 0056XF003397 AGE/SEX : 38 Years Male
FORTIS HOSPITAL - SHALIMAR BAGH - O PATIENT ID : FH.13187537 DRAWN : 06/06/2024 14:53:00
FORTIS HOSPITAL, A-BLOCKSHALIMIAR BAGH,
CLIENT PATIENT ID: UID:13187537 RECEIVED : 06/06/2024 14:52:40
NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
CLINICAL INFORMATION :
UID:13187537 REQNO-7210680
OPD-OPD
BILLNO-100324OPCS128336
Test Report Status Final Results Biological Reference Interval Units
HAEMATOLOGY - CBC
CBC-5, EDTA WHOLE BLOOD
BLOOD COUNTS, EDTA WHOLE BLOOD
HEMOGLOBIN (HB) 13.5 13.0- 17.0 g/dL
METHOD : SLS- HEMOGLOBIN DETECTION METHOD
RED BLOOD CELL (RBC) COUNT 4.63 4.50 - 5.50 mil/µL
METHOD : IMPEDENCE DETECTION METHOD
WHITE BLOOD CELL (WBC) COUNT 4.67 4.0 - 10.0 thou/µL
METHOD : FLOWCYTOMETRY
PLATELET COUNT 150 150 - 410 thou/µL
METHOD : DC / IMPEDENCE DETECTION METHOD
RBC AND PLATELET INDICES

HEMATOCRIT (PCV) 41.0 40.0 - 50.0 %
METHOD : SLS PULSE HEIGHT DETECTION METHOD
MEAN CORPUSCULAR VOLUME (MCV) 88.6 83 - 101 fL
METHOD : CALCULATED PARAMETER
MEAN CORPUSCULAR HEMOGLOBIN (MCH) 29.2 27.0 - 32.0 pg
MEAN CORPUSCULAR HEMOGLOBIN 32.9 31.50 - 34.50 g/dL
CONCENTRATION(MCHC)
RED CELL DISTRIBUTION WIDTH (RDW) 13.2 11.60 - 14.0 %
METHOD : ELECTRONIC IMPEDANCE
MENTZER INDEX 19.1
WBC DIFFERENTIAL COUNT

NEUTROPHILS 50 40 - 80 %
Page 1 Of 10
Dr. Manish Chugh MBBS,MD

(PATHOLOGY)
ATTENDING CONSULTANT
View Details View Report

PERFORMED AT :
CLINICAL LABORATORY
Fortis Hospital,Aa Block,Shalimar Bagh,Hospital Reg.No.882
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
Tel : 011-45302222/4045, Fax : 011-45302211 - CIN - L85110DL1996PLC076704
Email : contactus.sb@fortishealthcare.com
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
LYMPHOCYTES 37 20 - 40 %
MONOCYTES 7 2.0 -10.0 %
EOSINOPHILS 6 1.0 - 6.0 %
ABSOLUTE NEUTROPHIL COUNT 2.34 2.0 - 7.0 thou/µL
ABSOLUTE LYMPHOCYTE COUNT 1.73 1.0 - 3.0 thou/µL
ABSOLUTE MONOCYTE COUNT 0.33 0.20 - 1.0 thou/µL
ABSOLUTE EOSINOPHIL COUNT 0.28 0.02 - 0.50 thou/µL
NEUTROPHIL LYMPHOCYTE RATIO (NLR) 1.3
Interpretation(s)
RBC AND PLATELET INDICES-Mentzer index (MCV/RBC) is an automated cell-counter based calculated screen tool to differentiate cases of Iron deficiency anaemia(>13)
from Beta thalassaemia trait
(<13) in patients with microcytic anaemia. This needs to be interpreted in line with clinical correlation and suspicion. Estimation of HbA2 remains the gold standard for
diagnosing a case of beta thalassaemia trait.
WBC DIFFERENTIAL COUNT-The optimal threshold of 3.3 for NLR showed a prognostic possibility of clinical symptoms to change from mild to severe in COVID positive
patients. When age = 49.5 years old and NLR = 3.3, 46.1% COVID-19 patients with mild disease might become severe. By contrast, when age < 49.5 years old and NLR <
3.3, COVID-19 patients tend to show mild disease.
(Reference to - The diagnostic and predictive role of NLR, d-NLR and PLR in COVID-19 patients A.-P. Yang, et al. International Immunopharmacology 84 (2020) 106504
This ratio element is a calculated parameter and out of NABL scope.
Page 2 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
COAGULATION
PROTHROMBIN TIME, CITRATE PLASMA
PROTHROMBIN TIME (PT) 12.7 11.8 - 15.1 SECONDS
METHOD : CLOTTING TIME BASED
INTERNATIONAL NORMALIZED RATIO (INR) 1.21 <1.40 RATIO
MEAN PROTHROMBIN TIME OF CONTROL 10.7 SECONDS
PLASMA (MNPT)
Interpretation(s)
PROTHROMBIN TIME, CITRATE PLASMA-TEST DESCRIPTION- Prothrombin Time measures the integrity of the extrinsic pathway and the adequacy of critical coagulation
factors involved in it, namely, Factor VII. This test is therefore, used for monitoring oral anticoagulation therapy which lowers the levels of multiple vitamin K dependent
coagulation factors in blood (Factors II, VII, IX and X) including Factor VII. The result of PT is expressed as International Normalized Ratio (INR) to neutralize the influence
of variable sensitivity of the reagents (thromboplastin) used in the assay by different laboratories
TEST INTERPRETATION- INCREASED PT may be due to:
1.Factor deficiencies,2. Drugs (e.g. Coumarin-type drugs for anticoagulant therapy, salicytes),3. Severe liver damage (e.g. poisoning, hepatitis,
cirrhosis),4.Hypofibrinogenemia (Acquired or Inherited),5.Hemorrhagic disease of the newborn.,6 Poor fat absorption (e.g.obstructive
jaundice,fistulas,sprue,steatorrhea,celiac disease,colitis,chronic diarrhoea.)
RECOMMENDATION-This is a very sensitive reagent and therefore it is advisable of follow up I.N.R. value rather than P.T. in seconds.The recommended
I.N.R. is 2 - 3 for patients on oral anticoagulant in all conditions except mechanical valve replacement and prevention of myocardial infarction where the
I.N.R. may be maintained between 2.5-3.5. Please stop anticoagulant therapy if the I.N.R. is > 4.5.
Page 3 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
BIOCHEMISTRY
GLUCOSE RANDOM, PLASMA
RBS(RANDOM BLOOD SUGAR) 88 Non-Diabetic: < 200 mg/dL
Diabetic: > or = 200
"In individuals with symptoms
of hyperglycemia or
hyperglycemic crisis."
METHOD : HEXOKINASE
LIVER & KIDNEY PROFILE

ASPARTATE AMINOTRANSFERASE (AST/SGOT), SERUM
ASPARTATE AMINOTRANSFERASE 26 15 - 37 U/L
(AST/SGOT)
METHOD : UV WITH P5P
ALANINE AMINOTRANSFERASE (ALT/SGPT), SERUM

ALANINE AMINOTRANSFERASE (ALT/SGPT) 29 < 45.0 U/L
METHOD : UV WITH P5P
ALKALINE PHOSPHATASE, SERUM

ALKALINE PHOSPHATASE 78 30 - 120 U/L
METHOD : PNPP - AMP BUFFER
BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM

BILIRUBIN, TOTAL 0.77 0.2 - 1.0 mg/dL
METHOD : JENDRASSIK AND GROFF
Page 4 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
BILIRUBIN, DIRECT 0.22 High 0.0 - 0.2 mg/dL

METHOD : JENDRASSIK AND GROFF
BILIRUBIN, INDIRECT 0.55 0.1 - 1.0 mg/dL
GAMMA GLUTAMYL TRANSFERASE, SERUM

GAMMA GLUTAMYL TRANSFERASE (GGT) 23 15 - 85 U/L
METHOD : IFCC
LACTATE DEHYDROGENASE, SERUM

LACTATE DEHYDROGENASE 151 85 - 227 U/L
METHOD : LACTATE -PYRUVATE UV
ALBUMIN+GLOBULIN+A/G RATIO, SERUM

ALBUMIN 4.6 3.4 - 5.0 g/dL
METHOD : BCP
GLOBULIN 3.0 2.0 - 4.1 g/dL
ALBUMIN/GLOBULIN RATIO 1.5 1.0 - 2.1 RATIO
TOTAL PROTEIN, SERUM

TOTAL PROTEIN 7.6 6.4 - 8.2 g/dL
METHOD : BIURET
Page 5 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
BLOOD UREA NITROGEN (BUN), SERUM

BLOOD UREA NITROGEN 11 6 - 20 mg/dL
METHOD : UREASE KINETIC
CREATININE, SERUM
CREATININE 0.94 0.90 - 1.30 mg/dL
METHOD : JAFFE KINETIC METHOD
BUN/CREAT RATIO
BUN/CREAT RATIO 11.70 5.00 - 15.00
ELECTROLYTES (NA/K/CL), SERUM

SODIUM, SERUM 144 136 - 145 mmol/L
METHOD : ISE INDIRECT
POTASSIUM, SERUM 4.04 3.50 - 5.10 mmol/L
CHLORIDE, SERUM 101 98 - 107 mmol/L
URIC ACID, SERUM

URIC ACID 5.5 3.5 - 7.2 mg/dL
METHOD : URICASE UV
Page 6 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
Interpretation(s)
GLUCOSE RANDOM, PLASMA-Abnormal glucose level should be followed up with FBS,PPBS,HbA1c if clinically indicated.
ASPARTATE AMINOTRANSFERASE (AST/SGOT), SERUM-Aminotransferase (AST) is an enzyme found in various parts of the body .AST is found in the liver, heart, skeletal
muscle, kidneys, brain, and red blood cells, and it is commonly measured clinically as a marker for liver health. AST levels increase during chronic viral hepatitis, blockage
of the bile duct, cirrhosis of the liver, liver cancer, kidney failure, hemolytic anemia, pancreatitis, hemochromatosis. AST levels may also increase after a heart attack or
strenuous activity.
ALANINE AMINOTRANSFERASE (ALT/SGPT), SERUM-ALT test measures the amount of this enzyme in the blood.ALT is found mainly in the liver, but also in smaller amounts
in the kidneys,heart,muscles and pancreas.It is commonly measured as a part of a diagnostic evaluation of hepatocellular injury to determine liver health.AST levels
increase during acute hepatitis, sometimes due to a viral infection, ischemia to the liver, chronic hepatitis,obstruction of bile ducts,cirrhosis.
ALKALINE PHOSPHATASE, SERUM-Alkaline phosphatase (ALP) is a protein found in almost all body tissues. Tissues with higher amounts of ALP include the liver, bile ducts,
and bone. Elevated Alkaline Phosphaqtase levels are seen in Biliary obstruction,Osteoblastic bone tumors, osteomalacia, hepatitis, Hyperparathyroidism,Leukemia,
Lymphoma,Paget''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''
disease,Rickets,Sarcoidosis etc. Lower-than-normal ALP levels seen in Hypophosphatasia, Malnutrition, Protein
deficiency,Wilson'''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''
disease .
BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM-Bilirubin is a yellowish pigment found in bile and is a breakdown product of normal heme catabolism. Bilirubin is excreted in
bile and urine, and elevated levels may give yellow discoloration in jaundice.Elevated levels results from increased bilirubin production (eg, hemolysis and ineffective
erythropoiesis), decreased bilirubin excretion (eg, obstruction and hepatitis), and abnormal bilirubin metabolism (eg, hereditary and neonatal jaundice). Conjugated (direct)
bilirubin is elevated more than unconjugated (indirect) bilirubin in Viral hepatitis, Drug reactions, Alcoholic liver disease Conjugated (direct) bilirubin is also elevated more
than unconjugated (indirect) bilirubin when there is some kind of blockage of the bile ducts like in Gallstones getting into the bile ducts, tumors & Scarring of the bile ducts.
Increased unconjugated (indirect) bilirubin may be a result of Hemolytic or pernicious anemia, Transfusion reaction & a common metabolic condition termed Gilbert
syndrome, due to low levels of the enzyme that attaches sugar molecules to bilirubin.
Total Bili- Source: Wallach"s Interpretation of Diagnostic tests, 9th ed

Direct Bili - Source: Tietz Text book of Clinical Chemistry & Molecular Diagnostics, 4th ed
GAMMA GLUTAMYL TRANSFERASE, SERUM-Gamma glutamyl transferase (GGT) is an enzyme found in cell membranes of many tissues mainly in the liver, kidney, and
pancreas. It is also found in other tissues including intestine, spleen, heart, brain, and seminal vesicles. The highest concentration is in the kidney, but the liver is
considered the source of normal enzyme activity. Serum gamma-glutamyl transferase (GGT) has been widely used as an index of liver dysfunction. Elevated serum GGT
activity can be found in diseases of the liver, biliary system, and pancreas .Conditions that increase serum GGT are obstructive liver disease, high alcohol consumption, and
use of enzyme-inducing drugs etc.
ALBUMIN+GLOBULIN+A/G RATIO, SERUM-ALBUMIN+GLOBULIN+A/G RATIO, SERUM
Serum total protein,also known as total protein, is a biochemical test for measuring the total amount of protein in serum..Protein in the plasma is made up of albumin and
globuli.
Higher-than-normal levels may be due to: Chronic inflammation or infection, including HIV and hepatitis B or C, Multiple myeloma,
Waldenstrom''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''''s disease
Lower-than-normal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage),Burns ,Glomerulonephritis, Liver disease, Malabsorption,Malnutrition,Nephrotic
syndrome,Protein-losing enteropathy etc.Human serum albumin is the most abundant protein in human blood plasma. It is produced in the liver. Albumin constitutes about
half of the blood serum protein. Low blood albumin levels (hypoalbuminemia) can be caused by:Liver disease like cirrhosis of the liver, nephrotic syndrome, protein-losing
enteropathy,Burns,,hemodilution, increased vascular permeability or decreased lymphatic clearance,malnutrition and wasting etc.
TOTAL PROTEIN, SERUM-is a biochemical test for measuring the total amount of protein in serum.Protein in the plasma is made up of albumin and globulin.
Higher-than-normal levels may be due to: Chronic inflammation or infection, including HIV and hepatitis B or C, Multiple myeloma,Waldenstroms disease.
Lower-than-normal levels may be due to: Agammaglobulinemia, Bleeding (hemorrhage),Burns,Glomerulonephritis, Liver disease, Malabsorption, Malnutrition, Nephrotic
syndrome,Protein-losing enteropathy etc.
BLOOD UREA NITROGEN (BUN), SERUM-Causes of Increased levels include Pre renal (High protein diet, Increased protein catabolism, GI haemorrhage, Cortisol,
Dehydration, CHF Renal), Renal Failure, Post Renal (Malignancy, Nephrolithiasis, Prostatism)
Causes of decreased level include Liver disease, SIADH.
CREATININE, SERUM-Higher than normal level may be due to:
• Blockage in the urinary tract, Kidney problems, such as kidney damage or failure, infection, or reduced blood flow, Loss of body fluid (dehydration), Muscle problems, such
as breakdown of muscle fibers, Problems during pregnancy, such as seizures (eclampsia)), or high blood pressure caused by pregnancy (preeclampsia)
Lower than normal level may be due to:• Myasthenia Gravis, Muscuophy
URIC ACID, SERUM-Causes of Increased levels:-Dietary(High Protein Intake,Prolonged Fasting,Rapid weight loss),Gout,Lesch nyhan syndrome,Type 2 DM,Metabolic
syndrome Causes of decreased levels-Low Zinc intake,OCP,Multiple Sclerosis
Page 7 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
SEROLOGY
HEPATITIS B SURFACE ANTIGEN, SERUM
HEPATITIS B SURFACE ANTIGEN NON REACTIVE NON REACTIVE
METHOD : RAPID IMMUNOCHROMATOGRAPHY
HCV ABS, SERUM

HEPATITIS C ABS NON REACTIVE NON REACTIVE
METHOD : RAPID IMMUNOCHROMATOGRAPHY
Interpretation(s)
HEPATITIS B SURFACE ANTIGEN, SERUM-Hepatitis B is caused by infection with HBV, a enveloped DNA agent that is classified as hepadnavirus.This test detects the
presence of viral surface antigen (HbsAg) in serum sample and is indicative of an active HBV infection, either acute or chronic.
Test Utility:
HbsAg is the first serologic marker appearing in the serum 6-16 weeks following hepatitis B viral infection. In typical HBV infection, HBsAg will be detected 2-4 weeks before
the liver enzyme levels (ALT) become abnormal and 3-5 weeks before patient develops jaundice.In acute cases HbsAg usually disappears 1-2 months after the onset of
symptoms.Persistence of HbsAg for more than 6 months indicates development of either a chronic carrier state or chronic liver disease.The presence of HbsAg is frequently
associated with infectivity. HbsAg when accompanied by Hepatitis Be antigen and/or hepatitis B viral DNA almost always indicates infectivity.
Limitations:
- For diagnostic purposes, results should be used in conjunction with patient history and other hepatitis markers for diagnosis of acute or chronic infection. If the antibody
results are inconsistent with clinical evidence, additional testing is suggested to confirm the result.
- HBsAg detection will only indicate the presence of surface antigens in the serum and should not be used as the sole criteria for diagnosis, staging or monitoring of HBV
infection This test may be negative during ""window period"" i.e. after disappearance of anti-HBs.
- The current assay being a highly sensitive test , may yield a small percentage of false positive reports. Hence all HbsAg positive specimens should be confirmed with an
assay based upon Neutralisation of Human anti Hepatitis B Surface antibody.
HCV ABS, SERUM-Hepatitis C Virus (HCV) is a blood borne flavivirus. It is one of the most important causes of post-blood transfusion as well as community acquired non-A
non-B hepatitis and chronic liver failure. Although the majority of infected individuals may be asymptomatic, HCV infection may develop into chronic hepatitis, cirrhosis
and/or increased risk of hepatocellular carcinoma.
Notes & Limitations:

- HCV antibody is typically not detected until approximately 14 weeks after infection (or 5 weeks after appearance of the first biochemical marker of illness) and is almost
always detectable by the late convalescent stage of infection. - A negative result may also be observed due to loss of HCV antigen, years following resolution of
infection. Infants born to hepatitis C infected mothers may have delayed seroconversion to anti-HCV. Hence a negative result should be evaluated cautiously with respect to
clinical findings. It is to be noted that absence of HCV antibodies after 14 weeks of exposure is strong evidence against HCV infection.
- Presence of HCV antibodies does not imply an active Hepatitis C infection but is indicative of both past and/or recent infection .It has been reported that as many as 90%
of individuals receiving intravenous commercial immunoglobulin test falsely positive for HCV antibody. Also,patients with autoimmune liver disease may show a false positive
HCV antibody result. Hence it is advisable to confirm a positive antibody result with a supplemental test. A positive result when followed by a positive supplemental test (i.e.
HCV-RNA-PCR) suggests active hepatitis C infection.
Page 8 Of 10
DR. Monika Gupta MBBS,MD

(MICROBIOLOGY)
SR. CONSULTANT
MICROBIOLOGIST
PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
CLINICAL PATH - URINALYSIS

LIVER & KIDNEY PROFILE
PHYSICAL EXAMINATION, URINE
COLOR YELLOW
APPEARANCE CLEAR
Comments
URINE ANALYSIS : MICROSCOPIC EXAMINATION OF URINE IS DONE FROM CENTRIFUGED URINARY SEDIMENT.
URINE PROTIEN RECHECKED BY S.S.A MANUAL METHOD.
CHEMICAL EXAMINATION, URINE
PH 6.0 4.7 - 7.5
SPECIFIC GRAVITY >=1.030 1.003 - 1.035
PROTEIN DETECTED (TRACE) NOT DETECTED
GLUCOSE NOT DETECTED NOT DETECTED
KETONES NOT DETECTED NOT DETECTED
BLOOD NOT DETECTED NOT DETECTED
BILIRUBIN NOT DETECTED NOT DETECTED
UROBILINOGEN NORMAL NORMAL
NITRITE NOT DETECTED NOT DETECTED
METHOD : DIAZONIUM ION, BLANKED (ROCHE)
LEUKOCYTE ESTERASE NEGATIVE
MICROSCOPIC EXAMINATION, URINE

RED BLOOD CELLS NOT DETECTED NOT DETECTED /HPF
PUS CELL (WBC’S) 2-3 0-5 /HPF
Page 9 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India
DIAGNOSTIC REPORT
MC-2535

NEW DELHI 110088
ABHA NO : REPORTED : 06/06/2024 16:50:05
11-45302222
UID:13187537 REQNO-7210680
OPD-OPD
METHOD : MICROSCOPIC EXAMINATION

EPITHELIAL CELLS 1-2 0-5 /HPF
CASTS NOT DETECTED
CRYSTALS NOT DETECTED
BACTERIA NOT DETECTED NOT DETECTED
YEAST NOT DETECTED NOT DETECTED
**End Of Report**
Please visit www.agilusdiagnostics.com for related Test Information for this accession
CONDITIONS OF LABORATORY TESTING & REPORTING

1. It is presumed that the test sample belongs to the patient 5. AGILUS Diagnostics confirms that all tests have been
named or identified in the test requisition form. performed or assayed with highest quality standards, clinical
2. All tests are performed and reported as per the safety & technical integrity.
turnaround time stated in the AGILUS Directory of Services. 6. Laboratory results should not be interpreted in isolation;
3. Result delays could occur due to unforeseen it must be correlated with clinical information and be
circumstances such as non-availability of kits / equipment interpreted by registered medical practitioners only to
breakdown / natural calamities / technical downtime or any determine final diagnosis.
other unforeseen event. 7. Test results may vary based on time of collection,
4. A requested test might not be performed if: physiological condition of the patient, current medication or
i. Specimen received is insufficient or inappropriate nutritional and dietary changes. Please consult your doctor
ii. Specimen quality is unsatisfactory or call us for any clarification.
iii. Incorrect specimen type 8. Test results cannot be used for Medico legal purposes.
iv. Discrepancy between identification on specimen 9. In case of queries please call customer care
container label and test requisition form (91115 91115) within 48 hours of the report.
Agilus Diagnostics Ltd
Fortis Hospital, Sector 62, Phase VIII,
Mohali 160062
Page 10 Of 10

(PATHOLOGY)

PERFORMED AT :
CLINICAL LABORATORY
New Delhi, 110088 ULR No.56000001859098-0056
New Delhi, India

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DIAGNOSTIC REPORT

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

RBC AND PLATELET INDICES

WBC DIFFERENTIAL COUNT

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

LIVER & KIDNEY PROFILE

ALANINE AMINOTRANSFERASE (ALT/SGPT), SERUM

ALKALINE PHOSPHATASE, SERUM

BILIRUBIN (TOTAL, DIRECT, INDIRECT), SERUM

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

BILIRUBIN, DIRECT 0.22 High 0.0 - 0.2 mg/dL

GAMMA GLUTAMYL TRANSFERASE, SERUM

LACTATE DEHYDROGENASE, SERUM

ALBUMIN+GLOBULIN+A/G RATIO, SERUM

TOTAL PROTEIN, SERUM

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

BLOOD UREA NITROGEN (BUN), SERUM

ELECTROLYTES (NA/K/CL), SERUM

URIC ACID, SERUM

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

Total Bili- Source: Wallach"s Interpretation of Diagnostic tests, 9th ed

Dr. Manish Chugh MBBS,MD

View Details View Report

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

HCV ABS, SERUM

Notes & Limitations:

DR. Monika Gupta MBBS,MD

PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

CLINICAL PATH - URINALYSIS

MICROSCOPIC EXAMINATION, URINE

Dr. Manish Chugh MBBS,MD

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PATIENT NAME : AJAY KUMAR YADAV REF. DOCTOR :

Test Report Status Final Results Biological Reference Interval Units

METHOD : MICROSCOPIC EXAMINATION

CONDITIONS OF LABORATORY TESTING & REPORTING

Dr. Manish Chugh MBBS,MD

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