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    Pathophysiology CKD

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    Reymon Mary Janine
    Genetic and lifestyle factors like excess sodium and glucose intake can predispose to and precipitate chronic kidney disease by prolonging hypertension and diabetes. This causes decreased blood flow and patchy cell death in the kidneys over time, resulting in fibrosis, glomerular destruction, and decreased filtration. As renal function declines further, the kidneys fail to excrete waste or regulate fluids and electrolytes, allowing edema, uremia, and accumulated protein waste products in the blood.

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    Pathophysiology CKD

    Uploaded by

    Reymon Mary Janine
    3K views1 page
    Genetic and lifestyle factors like excess sodium and glucose intake can predispose to and precipitate chronic kidney disease by prolonging hypertension and diabetes. This causes decreased blood flow and patchy cell death in the kidneys over time, resulting in fibrosis, glomerular destruction, and decreased filtration. As renal function declines further, the kidneys fail to excrete waste or regulate fluids and electrolytes, allowing edema, uremia, and accumulated protein waste products in the blood.

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    PATHOPHYSIOLOGY CKD

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    Genetic and lifestyle factors like excess sodium and glucose intake can predispose to and precipitate chronic kidney disease by prolonging hypertension and diabetes. This causes decreased blood flow and patchy cell death in the kidneys over time, resulting in fibrosis, glomerular destruction, and decreased filtration. As renal function declines further, the kidneys fail to excrete waste or regulate fluids and electrolytes, allowing edema, uremia, and accumulated protein waste products in the blood.

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    Download as DOC, PDF, TXT or read online from Scribd
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    Download as doc, pdf, or txt
    3K views1 page

    Pathophysiology CKD

    Uploaded by

    Reymon Mary Janine
    Genetic and lifestyle factors like excess sodium and glucose intake can predispose to and precipitate chronic kidney disease by prolonging hypertension and diabetes. This causes decreased blood flow and patchy cell death in the kidneys over time, resulting in fibrosis, glomerular destruction, and decreased filtration. As renal function declines further, the kidneys fail to excrete waste or regulate fluids and electrolytes, allowing edema, uremia, and accumulated protein waste products in the blood.

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    PATHOPHYSIOLOGY Predisposing Factors Factors - Genetic

    Precipitating - Life style - Excessive intake of sodium and Glucose

    Prolonged hypertension and diabetes Can cause decreased blood flow to kidney and patchy necrosis of the renal parenchyma Overtime, fibrosis occurs Glomeruli are destroyed Decreased Glomeruli filtration rate Kidney fails to excrete nitrogenous waste products

    Impaired maintenance of fluid and electrolytes balance As renal function continues to decline The end products of protein metabolism accumulates in the blood

    edema

    Uremia (excess of urea and nitrogenous waste products in the blood) Serum Creatinine BUN

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