Ameloblastoma

Aameloblastoma
Ameloblastoma
Ameloblastoma
Peripheral Ameloblastoma
ameloblastoma
Ameloblastoma
2. Adenoameloblastoma.
Histological the tumor is characterized by duct like structures
consisting the epithelium compartment of the tumor.
It is encapsulated.
Clinically :
It occurs in young age 2
nd
&3
rd
decade of life.
More common in female.
More common in maxilla.
Usually associated with unerupted tooth
Radio graphically :
Unilocular radiolucent lesion which may resemble.
Treatment :
Since it is encapsulated and not aggressive like ameloplastoma,
conservative surgical excision or enucleation is sufficient.
Adeomatoid
odontogenic
tumor
Adenomatoid odontogenic tumor
Adenomatatoid odontogenic tumor
Adeno-
Ameloblastoma
Adenoameloblastoma
3. Odontoma
a. Compound composite Odon tome .
In which the hard tissues resemble teeth regardless the size and number, and
appear in x-ray as variable numbers of tooth like structure surrounded with
radiolucent line.
b. Complex composite Odon tome.
In which calcified dental structures (enamel, dentin and cement ) may be
scattered without any special pattern.
4. Cementoma .
It is rare solitary slow growing lesion.
It affects the mndibular molar and premolar area.
Radio graphically, it appears as well defined radio-opaque mass surrounded
by radiolucent.
The tumor mass is usually attached to the roots of the teeth.

Per apical cementoma
Complx
odontoma
Complex odontoma
Complex odontoma
Compound
odontoma
Compound odontoma
Compound odontoma
Compound
odontoma
Osteogenic tumors.
1. Osteoma.
a. Enostosis (central osteoma ).
It may be a central area of dense cortical bone.
Radio graphically it is a central circumscribed radio-opaque area.
b. Enostosis (peripherals osteoma ).
Such as torus palatinus, and torus mandibhlaris.
Treatment is by surgical excision.
2. fibro-osteoma or ossifying fibroma.
It is a relatively slow growing tumor that cause early displacement of teeth.
It also cause varying degrees of facial deformity.
Radio graphically, radio-opaque of varying densities
3. Chondroma .
It is a rare tumor of embryonic tissue origin from remnants of immature
cartilage cells.
It is slowly growing and usually appears as a radiolucent area in
radiographs.
It has a great tendency to malignant change.
o s t e o m a
osteoma
Ossifying fiberoma
Peripheral ossifying fibroma
Buccal exostosis
Torus palatinus
Buccal exostosis
Torus mandibularis
4. Myxoma ( fibromyxoma).
It is a tumor of embryonic tissue origin from primitive immature cells, the
one is collapsed by mucoid material.
This tumor is not capsulated.
It is locally invasive, painless slowly growing, producing facial
disfigurement.
It affects the mandible more than the maxilla.
Radio graphically. Honey-comb appearance.
5. Central giant cell lesion ( granuloma or tumor ).
Trauma is suspected to cause the benign lesion.
It affect the symphysis or the angle of the mandible and the maxilla at the
canine fossa.
It produce a painless swelling.
It causes looseness of the teeth and root resorption.
When the cortical plate is perforated by the tumor, bleeding become
frequently from slight trauma to tumor tissues
Radigraphically :
Mulilocular radiolucency with irregular margin.
Treatment :Surgical removal.
Giant cell granuloma
Central giant cell granuloma
Central giant
cell
granuloma
Central giant cell granuloma
Tumor like lesions of the jaws
: Fibrous dysplasia
It shows replacement of normal spongiosa with fibrous tissues, it is slowly
growing.
It tends grow in the 2
nd
decade of life, then stop.
It may produce pronounced facial asymmetry.
It may appear as a radiolucent area in the early stage , then diffuse radio-
opaque mass.
Treatment : conservative surgical contouring.
(Familial Fibrous Dysplasia): Cherubim
It is familial fibrous dysplasia, that becomes noticeable during the 2
nd
and
3
rd
years of life when fullness of the cheeks.
Gradual improvement or cessation of growth occurs after 10years of age.
Polyostotic Fibrous Dysplasia(Albrights
Syndrome
Affects limb,skull,and jaws during childhood, with cutaneous
pigmentation, endocrine disorders, precocious puberty & premature
skeleton maturation.
Hyperparathyroidism ( Brown tumor )
Increase of serum calcium phosphorus, and
serum alkaline phsphatase which is an
important sign of this lesion.
Radigraphically :Ground glass appearance of
the alveolar bone.
Resolution occur only after parathyroidectomy

Ossifying fibroma
Ossifying fibroma
Fibrous dysplasia
Fibrous dysplasia
cherubism
cherubim
cherubim
cherubism
cherubim
Hyperparathyroidism ( brown tumor)
Hyperpara thyroidism
Premalignant lesions
a. Leukoplakia :
It is white patch or plaque occurring on the surface of the mucous
membrane.
The buccal mucosa, commeasures are most frequently involved,
followed by alveolar mucosa, tongue, lip, hard and soft palates, floor of
the mouth and gingiva.
The surface of the lesion is often rough.
The lesion are white, gray or yellowish white.
b. Leukoedema.
It varies from family opalescence of the mucosa to more definite grayish
white cast with a coarsely wrinkled surface in later stages.
It involves most of the buccal mucosa, and the oral surface of the lips.
c. Intraepithelial carcinoma : ( carcinoma in situ ).
It is a precancerous dyskeratotic process.
Keratininzation is common feature.
The disease in the oral mucosa may appear as an erythromatous, velvet
plaque.
Most common located on gingiva and soft palate.

leukoplakia
leukoplakia
leukoplakia
leukoplakia