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Gastrointenstinal
Gastrointenstinal
GASTROINTENSTIN
AL
Anatomy of
Upper GI Tract
Anatomy of Upper GI
Tract
Cavitas Oris
Human Physiology - From Cells to Systems 7th ed - L. Sherwood (Cengage, 2010) BBS
Swallowing Physiology
Human Physiology - From Cells to Systems 7th ed - L. Sherwood (Cengage, 2010) BBS
Human Physiology - From Cells to Systems 7th ed - L. Sherwood (Cengage, 2010) BBS
Human Physiology - From Cells to Systems 7th ed - L. Sherwood (Cengage, 2010) BBS
Disfagia
defect
Anatomy Defect
Neurology Defect
Diagnose
Aloananesis
Physical Examination
Videofluoroskopi
Radiology
Therapy
Feeding Tube
Structure Anatomy function Therapy
Nonnutritive Sucking Stimulation
Surgery
Pharmacology
GERD
Risk Factor
Long LES
Anti Colinergik consumption
Hormone
Hiatus hernia
Heartburn sensation
Disfagia
Regugitation
NCCP
Astma
Laryngitis
Diagnose
Endoscopic
Esophagographic with barium
pH Monitoring
Berstein test
Manometry
Sintigraphy
Proton pump inhibitor Acid
supression test
Treatment
No Smoking and
alcoholic
Reduce lipid
Antasid
consumption
Reduce body mass
Antagonis H2 Receptor
Do not wear tight shirt
Prokinetik
Avoid acid secretion food Sukralfat
Avoid weaken LES drugs PPI
Non Pharmacology
Pharmacology
Antasid
Antagonis H2 Receptor
Prokinetic
Metoclopramide
Antagonis dopamine receptor
Effect: Drowzinnes, confusion, tremor
Dosage 3x10mg
Domperidone
Effect below metoclopramide
Dosage: 3x10-20mg/day
Cisapride
Antagonis receptor 5 HT4
Dosage: 3x10 mg
PPI
Complication
Striktur Esophagus
Esophagus Barret
Achalasia
Pathognomonic
Inflammation in the myenteric plexus
Achalasia
Achalasia
Progressive dysphagia
Inability to completely
Dysphagia (most
common)
Regurgitation
Chest pain
Heartburn
Weight loss
Signs & Symptoms
Epidemiology (Frequency)
United States
The incidence of achalasia is approximately 1
International
Chagas disease may cause a similar disorder.
Sex
The male-to-female ratio of achalasia is 1:1.
Age
Achalasia typically occurs in adults aged 25-60
Medical Care
Goal: relieves symptoms by eliminating the outflow resistance
caused by the hypertensive and nonrelaxing LES
Mouth Ulcer
Primary Acute Herpetic Gingivostomatitis (HSV-1, 2)
Mouth Ulcer
Herpes Zoster (Reactivation of VZV)
Mouth Ulcer
Aphthous Ulcers