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HISTORY

EMIL KREAPLIN 1856-1926


ENDOGENOUS PSYCHOSIS
DEMENTIA PRAECOX 1896

E.Bleuler

Schizophrenia
SPLITTING PERSONALITY

Prof. Shato JEPAN/KOREA 2013


Gangguan intregritas
Stigma BAGI PASIEN/KEL
BRAIN DISEASE

16 %

32
%

9%

43 %

Cambrige university in 5 years Research of SR

NOT COMPLIANCE
SUPPORT!!!

35 %

AMBIVALENCE TO DRUG

LACK OF INSIGHT
MANIA/HYPOMAN HAPPY
ACTUAL SIDE EFFECTS ALLERGY,
EPS, M. SYND
SOCIAL ECONOMIC PROBLEMS + .
4

TATALAKSANA PSYCHOSIS
MANAGEMENT ?
PREVENTION
SPECIFIC BIOLOGICAL
RISK (GENE)
NEURODEVELOPMENT/D
EGENE-RATIVE
PSYCHOLOGICAL
TRAUMA
SPECIFIC RISK
CONDITIONS (POVERTY,
ILLNESS, DRUG
ABUSE)

R
S
P
A
D
G
S

JCI (PATIENT SAFETY )


MULTIMODAL
TREATMENT/REHABILITA
TION
PRODROMAL-ACUTECHRONIC
MEDICATION, T SYMPT,
SE
DENIAL, COMPLIANCE,
RELAPSING
AGITATION-SUICIDEDRUG AB
CBT, FAM TH/, SOCIAL
INTERVENTION

EARLY DETECTION ASSESSMENT


PSYCHOLOGICAL PROTECTOR/BUFFER
PSYCHOEDUCATION. P SOLVING.
RESILIENCE. COPING M
PSYCHOLOGICAL
READINESS/FUNCTIONAL

PREVENTION
GENETIC 80%

PSYCHOLOGICAL

INFECTION/ALLERG
Y/
IMUN SYSTEM
20%
PREMORBIDNEURODEVELOPM
ENT
OBSTETRIC
COMPLICATION

SOCIAL-CULTURE
PARENTING

ELIMINATE
RISK
FACTORS

ENVIRONTMENT
PRE-PERINATAL

MENTAL HEALTH
CARE
SPECIAL
EDUCATION
PSYCHOLOGICAL
BUFFER

DEGENERATIVE P

SYMPTOMS
PSYCHOSIS

RISPERDAL CONSTA 25
-37.5 mg
FIRST EPISODE OF
PSYCHOSIS
MAINTENANCE THERAPY

MEDICATIONS
COGNITIVE BEHAVIORAL THERAPY
PSYCHOLOGICAL SUPPORT
7

SEROTONIN RECEPTOR

10

DOPAMIN-SEROTONIN INTERACTION

11

BRAIN CIRCUITS SYMPTOM

12

GENES CODE PROT


DISORDERS ? 80%
TWIN STUDIES
CHROMOSOMES LOCUS 22q 11,6p22,8p1221, 1q21-22,7q21-22,1q42,13q32-34 , 12q24
GENE
DTNBP1,COMT,NGG1,RGS4,GRM3,DISC1,G72,
DAAO (MULTIPLE GENE)
BRAIN
STRUCTUREVENT,CORTICAL/LIMBIC,SUBCOR
TICAL,GREY/WHITE MATTER
FUNCTIONAL GENOMIC AND PROTEOMIC m
RNA
13

14

DRUG TREATMENT
Typical , atypical anti psychotics , ANTYPICAL +
OTHERS
Acute , relapsing , Schizophrenia
Target symptoms , Monitoring Side effect
Maintenance Treatment
Complaince medication
Functioning
Regression

Risperdal Consta

!!!!!....... 100 %

Optimalization

><
15

TREATMENT PHASE
Prodromal phase Period of Deteriorating
function PSYCHOTROPIC DRUGS (PD)
Acute phase HALUSINASI+WAHAM
PD
Recovery phase PD
Residual Phase Apathy, Lack of
Motivation, Withdrawal, restricted of flat
affect PD
Chronically impaired remain sympt PD
Co Morbidity (Depression/Mania)PD
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KASUS
1. NW . 14 . SMP 1 . TIDAK SEKOLAH 8 BL
BICARA KACAU, FLIGHT OF IDEAS, MANIK
TIDAK MAU MAKAN OBAT, TIDAK MAU
DIRAWAT ++++ Risperdal Consta ..6 X
2. N 17 tahun . MR+ G. Affective . 4 X
3.

P. 62 th SR kronis eksaserbasi akut..

st psikiatri tenang 3 tahun.

17

Risperdal Consta NON


COMPLIANCE
RELAPSING
AGITATION Orbitofrontal
SCHIZOPHRENIA CHRONIC
DRUG ABUSE - DRUG RESISTANT
SUICIDE
18

MULTIMODAL THE BEST

MEDICATION PRIORITY +
PSYCHOLOGICAL INTERVENTION
MORE EFFECTIVE

19

WHAT RISK FACTOR?


WHEN BEST TO
INTERVENE EARLY?
BIOPSIKOSOS
BRAIN !

WHICH MEDICATION?
HOW MUCH, HOW
LONG FOR FIST
EPISODE?
RISPERDAL 2-6 mg!

HOW LONG,
WHAT MAINTENANCE
DOSE ?

RISPERDAL
CONSTA 25-37.5
mg!!

EBM
FOR PSYCHOSIS

HOW BEST FOR


SYNERGIZE
MEDICATION AND
OTHER THERAPIES?
MULTIMODAL T/

WHICH
PSYCHOTHERAPY?
FOR WHOM, HOW
LONG AND WHEN ?
INDIVIDUAL/FAM/SOS !

WHICH DRUG WORKS


BEST, FOR
STAGE/TYPE?
TIPICAL/ATYPICAL
ANTIPSIKOTIK!
20

RISPERDAL CONSTA

THANK YOU
21

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