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Pulp Capping: Guided By: Dr. J N Shukla Dr. Rahul Mishra Dr. Madhulika Srivastava
Pulp Capping: Guided By: Dr. J N Shukla Dr. Rahul Mishra Dr. Madhulika Srivastava
Pulp Capping: Guided By: Dr. J N Shukla Dr. Rahul Mishra Dr. Madhulika Srivastava
Submitted by:
Rockey Shrivastava
Xth Batch BDS
Roll - 30
Guided by:
Dr. J N Shukla
Dr. Rahul Mishra
Dr. Madhulika
Srivastava
CONTENTS
1. INTRODUCTION
2. TREATMENT MODALITIES
3. TYPES OF PULP CAPPING
4. INDIRECT PULP CAPPING
5. PATENT DENTIN MEASURING DEVICE
6. DIRECT PULP CAPPING
7. FEATURES OF SUCCESSFUL PULP CAPPING
8. PULP CAPPING MATERIALS
9. CONCLUSION
10.REFERENCES
INTRODUCTION
TREATMENT MODALITIES
PULP TREATMENT
CONSERVATIVE
1. Protective base
Pulpectomy
2. Indirect pulp therapy
filling
3. Direct pulp therapy
4. Pulpotomy
RADICAL
1.
2. Root
PULP CAPPING
INDICATIONS
When pulp inflammation has been judged to
be minimal and complete removal of caries
would cause pulp exposure
CONTRAINDICATIONS
Any signs of
pulpal or
periapical
pathology
Soft leathery
dentin covering
a very large
area of the
cavity, in a non
restorable tooth
PROCEDURE
The tooth is anesthetized and isolated with rubber dam
All the caries except that immediately over the pulp is
removed (use large round bur at low speed)
A zone of AFFECTED demineralized dentin is left behind
Not all undermined enamel is removed
A sedative dressing of either zinc oxide eugenol or
calcium hydroxide is placed
CONTD..
The tooth may then be restored with ZOE or
amalgam
The formation of reparative dentin beneath the
caries (average rate 1.4 microns per day)
The treated tooth is re entered after 6 to 8 weeks
and the remaining caries is excavated
Pulpal protection with adequate base and permanent
restoration
(If the restoration has a good margin and at the
recall visit a layer of secondary dentin is evident ,
reentry is not necessary)
- limit of safe
danger of
through
Internal resorption
Calcifications
Chronic pulp inflammation
Necrosis
Intraradicular involvement
INDICATIONS
CONTRAINDICATIONS
Pain at night
Spontaneous pain
Tooth mobility
Thickening of periodontal membrane
intraradicular radiolucency
Excess bleeding at the exposure site
Purulent or serous exudate
PULP EXPOSURE
CALCIUM HYDROXIDE
TECHNIQUE
Hemostasis
Disinfect cavity
Calcium hydroxide
IRM
resin
modified GIC
dentin bonding
system
Restoration
DENTIN BONDING
SYSTEM
Hemostasis
Disinfect cavity
Bonding system
Adhesive
Restoration
CALCIUM HYDROXIDE
Calcium hydroxide is the
material of choice.
Herman in 1930 1st
introduced Ca(OH)2 for pulp
capping.
Ca(OH)2 causes necrosis of
adjacent pulp tissue and
inflammation of contiguous
tissue.
Dentin bridge formation
occurs at the junction of
necrotic and inflamed tissue
Pure calcium
hydroxide
Dentinogenesis
bactericidal then
bacteriostatic.
Promotes healing
and repair
High pH
stimulates
fibroblasts
May degrade
during acid etching
and tooth flexure
Marginal failure
with amalgam
condensation
Doesnt adhere to
dentin or resin
restoration
A D VA N TA G E S
Neutralization of
acids
Stops internal
resorption
Inexpensive and
easy to use
Particles may
obturate open
tubules
D I S A D VA N TA G E S
ISOBUTYL CYANOACRYLATE
LASER
Andreas Moritz
in 1998
evaluated the
effect of Co2
laser on direct
pulp capping.
Success rate89%
PROPOLIS
CONCLUSION
Y
T
S
N
A UE
Q ?
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S
N
IO
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BIBLIOGRAPHY