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  • Pathophysiology Mi

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    Katrina Alyanna Ricafort
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    The document describes the pathophysiology of a myocardial infarction (heart attack). It involves: 1) A change in plaque in the coronary artery which activates platelets, causing platelet aggregation, adhesion, and buildup leading to thrombosis. 2) Thrombosis causes a decrease in blood flow and oxygen delivery to heart tissue, leading to myocardial cell death. 3) Myocardial cell death causes changes in heart function and electrical activity that can produce symptoms like chest pain, palpitations, weakness, and arrhythmias.

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    PATHOPHYSIOLOGY-MI.pptx

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    The document describes the pathophysiology of a myocardial infarction (heart attack). It involves: 1) A change in plaque in the coronary artery which activates platelets, causing platelet aggregation, adhesion, and buildup leading to thrombosis. 2) Thrombosis causes a decrease in blood flow and oxygen delivery to heart tissue, leading to myocardial cell death. 3) Myocardial cell death causes changes in heart function and electrical activity that can produce symptoms like chest pain, palpitations, weakness, and arrhythmias.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as pptx, pdf, or txt
    31 views8 pages

    Pathophysiology Mi

    Uploaded by

    Katrina Alyanna Ricafort
    The document describes the pathophysiology of a myocardial infarction (heart attack). It involves: 1) A change in plaque in the coronary artery which activates platelets, causing platelet aggregation, adhesion, and buildup leading to thrombosis. 2) Thrombosis causes a decrease in blood flow and oxygen delivery to heart tissue, leading to myocardial cell death. 3) Myocardial cell death causes changes in heart function and electrical activity that can produce symptoms like chest pain, palpitations, weakness, and arrhythmias.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as pptx, pdf, or txt
    of 8

    PATHOPHYSIOLOGY

    Change in the condition of


    plaque in the coronary artery

    Activation of platelets

    Platelet Aggregation,
    Adhesion, & Buildup of
    Platelet Mass

    Thrombosis

    Release of
    Thromboplastin

    Platelet release Thromboxane


    A2, Serotonin, Platelet Factors
    3 and 4
    ATHEROSCLEROSIS

    Rest Nausea Sweating


    Pain and/or
    Vomiting

    Claudication Fainting Cool


    Extremities
    Coagulation and
    Vasospasm

    Enlargement of
    Thrombus

    Occlusive
    Thrombus

    Ischemia of PALPITATIONS
    Tissue in Region
    Supplied by Artery

    Coronary Blood
    Supply Less than
    Demand
    MYOCARDIAL CELL
    DEATH (NECROSIS)
    Decrease
    Oxygen

    Increase Carbon Stimulation of


    Dioxide Levels WEAKNESS / Sympathetic NS
    FATIGUE

    Decrease
    Blood pH INCREASE
    IN HEART
    RATE

    DEEP &
    LABORED Increase Basal
    BREATHING Metabolic Rate

    DIAPHORESIS
    Increase Increase Decrease Blood Increase Afterload
    Epinephrine Oxygen Need Supply to GI Tract
    and
    Norepinephrine Vasoconstriction
    Production
    NAUSEA /
    VOMITTING

    HYPERTENSION

    ARRYTHMIAS
    MYOCARDIAL CELL DEATH (NECROSIS)

    Decreased Altered Release of Anaerobic Myocardial


    Contractility Repolarization Lysosomal Glycolysis Irritability
    of the Enzymes
    Decrease Myocardium
    Ventricular Lactic Acid
    Function Production

    Increase Decrease DYSRHYTHMIA


    ANGINA
    Preload Carbon
    Dioxide

    Decrease
    Blood Flow to
    Brain

    LOSS OF
    CONSCIOUSNESS

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