This document discusses hydrocephalus, which is an abnormal enlargement of the ventricles due to excessive accumulation of cerebrospinal fluid. It affects 1-1.5% of the population and can be congenital or acquired. Symptoms in neonates and infants include head enlargement and bulging fontanelles, while in children symptoms include headaches and vomiting. Treatment goals are to relieve hydrocephalus and its complications. Surgical treatment via shunt placement is usually needed to drain cerebrospinal fluid from the ventricles to the peritoneum or right atrium. Shunt infection is a serious complication.
This document discusses hydrocephalus, which is an abnormal enlargement of the ventricles due to excessive accumulation of cerebrospinal fluid. It affects 1-1.5% of the population and can be congenital or acquired. Symptoms in neonates and infants include head enlargement and bulging fontanelles, while in children symptoms include headaches and vomiting. Treatment goals are to relieve hydrocephalus and its complications. Surgical treatment via shunt placement is usually needed to drain cerebrospinal fluid from the ventricles to the peritoneum or right atrium. Shunt infection is a serious complication.
This document discusses hydrocephalus, which is an abnormal enlargement of the ventricles due to excessive accumulation of cerebrospinal fluid. It affects 1-1.5% of the population and can be congenital or acquired. Symptoms in neonates and infants include head enlargement and bulging fontanelles, while in children symptoms include headaches and vomiting. Treatment goals are to relieve hydrocephalus and its complications. Surgical treatment via shunt placement is usually needed to drain cerebrospinal fluid from the ventricles to the peritoneum or right atrium. Shunt infection is a serious complication.
Abnormal enlargement of the ventricels due to an excessive accumultion of CSF resulting from a disturbance of its flow, absorption or secretion. Prevalence : 1-1,5% Type: Patophysiology Communicating Noncommunicating Present Congenital : 0,9-1,8/1000 birth Acquired Neonatus - Infancy Progresive head enlargement Cranium enlarges ata rate > facial growth Bulging fontanelle Enlargement of scalp veins (vena ectasis) Macewen’s sign: cracked pot sound Sunset phenomenom Splitting cranial sutures Transilumination Children: headache, vomiting, diplopia, ataxia, visual loss, behavioral changes Raise ICP, Cushing’s triad: bradycardia, hypertension, decreased RR Symptom Imaging: USG Head CT Scan MRI Plain X-Ray Brain atropy Hydranencephaly Developmental anomaly: agenesis corpus callosum Goals: Relieve hydrocephaly
Treat complications
Manage problem resulting from effects of
disorder on psychomotor development USUALLY SURGICAL! Medical: Diuretics: Furosemida : 1 mg/kg/day TID Acetazolamide : 25 mg/kg/days TID Temporary: Eksternal ventricular drain Persistent : Shunt Endoscopic Third Ventriculostomy (ETV) Therapy of choice! Direct removal of source of obstruction (neoplasm, cyst, or hematoma) Most require shunt procedure to drain CSF from ventricles to extracranial area; usually peritoneum(VP shunt), or right atrium (VA shunt) for absorption. Type: VP (ventriculoperitoneal) shunt VA (ventriculoatrial) shunt VPl (ventriculopleural) shunt LP (Lumboperitoneal) shunt Shunt infection is most serious complication! Period of greatest risk is 1 to 2 months following placement. Staph and strep most common organisms Mechanical difficulties kinking, plugging, migration of tubing. Malfunction is most often by mechanical obstruction! Look for signs of increased ICP; fever, inflammation and abdominal pain. Slit ventricel Subdural hematoma Underlying disease Cerebral mantle 24