Presented by:

Venus, Abi
Buenafe, Drexler
• Pregnancy places a normal heart under immense physical strain.
• If the heart is already compromised by an existing anomaly, this
can result in a poor outcome for both fetus and mother.
• The onset of pregnancy marks the beginning of progressive and
profound changes in the physiology of the cardiovascular
system.
• Rheumatic
• Congenital
• Arrhythmia
• Cardiomyopathy
• Most common lesion is Mitral Stenosis
• Occurs when damage to one or more heart valves after one or
more episodes of acute rheumatic fever.
• Patients are at high risk for developing heart failure, sub acute
endocarditis and thromboembolic disease.
• Increase risk for fetal wastage.
• Onset of pedal edema: 40weeks of gestation
• Severe Mitral Stenosis lead to atrial fibrillation, which can lead
to Congestive Heart Failure.
• Fever, joint pain
• Skin rash, lumps under skin
• Jerky movements
• Fatigue, chest pain, swelling(edema), breathlessness
• Chronic inflammation of the heart which may lead to heart
failure or even death.
• Maternal and neonatal death
• Preterm delivery
heart failure in late pregnancy, during pregnancy, and post-
delivery
• Increased risk of complications.
• More frequent antenatal visits.
• More rest.
• Diet is directed to restrict weight gain and prevent anemiaas it
increases cardiac strain.
• Infection should be avoided and properly treated.
• Hospitalization: if signs of decompensation occur, the earliest
evidence is tachycardia exceeding 100 beats per minute and
crepitation at the lung base.
• Digoxin: indicated in atrial fibrillation to slow the ventricular response
and in acute heart failure to increase myocardial contractility.

• Diuretics are used in acute and chronic heart failure with potassium
supplements in prolonged therapy.

• Beta-adrenergic blockers: as propanolol may be indicated for

arrhythmia associated with ischemic heart disease.

• Amynophylline: relieves bronchospasm

• Heparin: indicated in patients with artificial valves or atrial

fibrillation.