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Calcaneal Fractures: by Philip Parr
Calcaneal Fractures: by Philip Parr
By Philip Parr
INTRODUCTION
Calcaneal fractures were first described by Malgaigne in
1843, but were not consistently diagnosed until the
development of plain radiography in the late 1890’s.10
The industrial revolution led to the development of taller
buildings, and the automobile, so that falls from heights
and MVA’s became increasingly more common, and
remain the most common cause of calcaneal fractures.10
INTRODUCTION
Calcaneal fractures account for 2% of all fractures.
Displaced intraarticular fractures represent 60-75% of all
calcaneal fractures.
10% of patients with calcaneal fractures have associated
spine fractures, and 26% have other extremity injuries.
90% of calcaneal fractures occur in young men in their
working prime.
HISTORICAL TREATMENT OF
CALCANEAL FRACTURES
• As early as 1908, Cotton and Wilson suggested that
ORIF of a calcaneal fracture was contraindicated.1
• McLaughlin likened attempts of operative fixation as
“nailing custard pie to a wall”.2
• Cotton and Wilson recommended closed treatment with
use of a medially placed sandbag, a laterally placed felt
pad, and a hammer to reduce the lateral wall and
“reimpact” the fracture.
• This treatment was abandoned in the 1920’s.
HISTORICAL TREATMENT OF
CALCANEAL FRACTURES
• Bohler in 1931 recommended operative treatment.
• However, operative treatment was rarely done due to
technical problems associated with it.
– Anesthesia not always effective
– Radiology not well-developed
– Abx did not exist
– Sound understanding of internal fixation was lacking
HISTORICAL TREATMENT OF
CALCANEAL FRACTURES
Throughout the 1940’s and 1950’s treatment varied
between ORIF attempts and subtalar joint arthrodesis.
In the 1960’s and 1970’s, as the result of an article by
Lindsay and Dewar showing operative intervention was
unnecessary, calcaneal fractures were mostly treated
non-operatively.
HISTORICAL TREATMENT OF
CALCANEAL FRACTURES
In the last 30 years, better anesthesia, Abx, the AO
principles, CT, and fluoroscopy, have allowed surgeons
to obtain good outcomes with operative intervention in
most fractures3.
Even with improvement, the treatment still remains
challenging and with many complications.4
To operate or not to operate???
RADIOGRAPHIC ANATOMY
Bohler’s Angle- Formed by line from highest
point of anterior process to highest point of
posterior facet and the line running along the
superior portion of the calcaneal tuberosity.
RADIOGRAPHIC ANATOMY
Gissanes angle: Formed by a line that runs
along the lateral border of the posterior
facet, and a line extending along the beak of
the calcaneus.
Radiographic Anatomy
Compression Trabeculae
Traction Trabeculae
Medial Talocalcaneal
Ligament
QUICK CLASSIFICATION REFRESHER:
Rowe 1a: Plantar Rowe 1b: ST secondary to
Tuberosity inversion
Rowe 1c: ant Rowe IIa: Beak fx
Rowe IIIb
Rowe Va Rowe Vb
SANDERS CLASSIFICATION
Based on Posterior Facet
After coronal CT,
Sanders typically used to
classify.
A Non-displaced fracture,
regardless of the amount
of fracture lines is a
Sanders Type I
MECHANISM OF INJURY OF
CALCANEAL FRACTURES
MECHANISM OF INJURY OF
CALCANEAL FRACTURES
High-energy
Force through subtalar
joint driving talus lateral
process into everted
calcaneus to create
fracture patterns
described by Essex-
Lopresti.5
MECHANISM OF INJURY OF
CALCANEAL FRACTURES
The “axe” of the lateral
process of talus is driven
into lateral wall of
calcaneus.
The force extends
posteriomedially into the
ST and medial wall.
This produces a fracture
that runs superior lateral
to inferior medial.5
MECHANISM OF INJURY OF
CALCANEAL FRACTURES
The lateral process of the talus is impacted at the crucial
angle of Gissane, which divides the lateral wall and the
body of the calcaneus9.
Residual force is then dissipated medially into the
sustentaculum tali which may be sheared off.
If the momentum stops here then part or all of the fissure
described is what we see.
If the momentum continues however…
MECHANISM OF INJURY OF
CALCANEAL FRACTURES
A secondary fracture line is then resulted from increased
force9:
Tongue-type fracture:
Secondary fracture line runs
straight back to the posterior
border of the tuberosity, from
the crucial angle of Gissane.
MECHANISM OF INJURY OF
CALCANEAL FRACTURES
The final stage9:
The front end of the tongue is driven down, but the tuberosity
is forced upwards by the ground. It separates from the body
as the primary fracture line opens up.
OPERATIVE VS NON-OPERATIVE CARE
Parmar et al, in a 1993 study of 56 patients who had been
randomized by DOB to either operative or non-operative
care, demonstrated that there was…
NO DIFFERENCE between the groups at one year of
follow-up.
OPERATIVE VS NON-OPERATIVE CARE
In another 1993 study by O’Farell et al, twelve patients
were assigned, without randomization, to operative care
and twelve were assigned to non-operative care.6 After
fifteen months of follow-up, the patients who had been
managed operatively had returned to work sooner and
walked better than those who had been managed…
NON-OPERATIVELY
OPERATIVE VS NON-OPERATIVE CARE
In a meta-analysis published in 2000, Randle et al stated
that “there is a trend for surgically treated patients to
have better outcomes; however, the strength of evidence
for recommending operative treatment is weak.”7
OPERATIVE TREATMENT WITH *
OPERATIVE COMPARED WITH NON-
OPERATIVE TREATMENT OF DISPLACED
INTRA-ARTICULAR CALCANEAL
FRACTURES8