Obstructive shock is characterized by reduced cardiac output and oxygen delivery due to extracardiac processes that impair blood flow rather than problems with the heart itself. Common causes include tension pneumothorax, cardiac tamponade from fluid around the heart, and pulmonary embolism from blood clots in the lungs. Tension pneumothorax requires immediate needle decompression and chest tube placement. Cardiac tamponade may require bedside ultrasound and pericardiocentesis. Massive pulmonary embolism is treated with small fluid volumes, vasopressors, and potentially thrombolysis if it causes profound hypotension.
Obstructive shock is characterized by reduced cardiac output and oxygen delivery due to extracardiac processes that impair blood flow rather than problems with the heart itself. Common causes include tension pneumothorax, cardiac tamponade from fluid around the heart, and pulmonary embolism from blood clots in the lungs. Tension pneumothorax requires immediate needle decompression and chest tube placement. Cardiac tamponade may require bedside ultrasound and pericardiocentesis. Massive pulmonary embolism is treated with small fluid volumes, vasopressors, and potentially thrombolysis if it causes profound hypotension.
Obstructive shock is characterized by reduced cardiac output and oxygen delivery due to extracardiac processes that impair blood flow rather than problems with the heart itself. Common causes include tension pneumothorax, cardiac tamponade from fluid around the heart, and pulmonary embolism from blood clots in the lungs. Tension pneumothorax requires immediate needle decompression and chest tube placement. Cardiac tamponade may require bedside ultrasound and pericardiocentesis. Massive pulmonary embolism is treated with small fluid volumes, vasopressors, and potentially thrombolysis if it causes profound hypotension.
Obstructive shock is characterized by reduced cardiac output and oxygen delivery due to extracardiac processes that impair blood flow rather than problems with the heart itself. Common causes include tension pneumothorax, cardiac tamponade from fluid around the heart, and pulmonary embolism from blood clots in the lungs. Tension pneumothorax requires immediate needle decompression and chest tube placement. Cardiac tamponade may require bedside ultrasound and pericardiocentesis. Massive pulmonary embolism is treated with small fluid volumes, vasopressors, and potentially thrombolysis if it causes profound hypotension.
• Obstructive shock is also characterized by a reduction in oxygen
delivery related to reduced CO, but in this case the etiology of the reduced CO is an extracardiac processes impairing blood flow. Processes that can impede venous return to the heart and reduce CO include tension pneumothorax (PTX), cardiac tamponade, and restrictive pericarditis. Similarly processes that obstruct cardiac outflow, such as pulmonary embolism (right heart) or aortic dissection (left heart), are included in this shock type category.
Harrisons Principles of Internal Medicine
• Tension pneumothorax is a clinical not radiographic diag-nosis characterized by unilateral decreased breath sounds, unilateral chest hyperresonance, and tracheal deviation in the setting of respiratory distress and shock. Treatment is immediate needle decompression followed by chest tube thoracostomy placement. • Pericardial tamponade likewise should be considered early in the evaluation of undifferentiated shock. Patients with blunt or penetrating chest trauma can rapidly de-compensate with minimal bleeding into the pericardium, while those with uremia and cancer usually develop an effusion over time. Symptoms include hypotension, elevated right side pressures (JVD) pulsus paradoxus (a fall in systolic blood pressure in inspiration), and Kussmaul’s sign (increased jugular venous pressure on inspiration). Bedside U/S is extremely sensitive in detecting pericardial fluid and can be instrumental in guiding pericardiocentesis, although in the patient in extremis, blind pericardiocentesis might be lifesaving CURRENT Lange Diagnosis & Treatment Emergency Medicine • Massive PE presents as chest pain, syncope, tachypnea, and hypotension with signs of acute right ventricular overload with JVD and ECG changes. Fluid administration might worsen right ventricular failure and should be given only cautiously. Blood pressure should be augmented with an appropriate vasopressor such as norepinephrine 0.5–1 μg/min titrated to response. Immediate surgical embolectomy is sometimes effective but not usually feasible. Shock complicating PE is an indication for thrombolytics if no other contraindication exists.
CURRENT Lange Diagnosis & Treatment Emergency
Medicine Treatment Obstructive Shock • Cardiac Tamponade Administer 1-2 L of normal saline followed by emergent bedside pericardiocentesis. Perform an emergency department thoracotomy in patients with penetrating thoracic trauma who fail to respond. • Pulmonary Embolism Administer small boluses of normal saline (250-500 mL) followed by vasopressor support in unstable patients. Fibrinolysis is the treatment of choice for massive PE presenting with profound hypotension (MAP <60), severe refractive hypoxemia (Sp02 <90 despite supplemental 02), or cardiac arrest. • Tension Pneumothorax Administer 1-2 L of normal saline while performing emergent needle thoracostomy followed by chest tube placement. Lange Clinical Emergency Medicine