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  • Burn Pathophysiology

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    Charisse Taylan
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    The body responds to burns in several stages. Initially, tissue perfusion increases in the outermost zone to sustain the least damaged tissue. The middle zone experiences decreased perfusion, leading to inflammation and injury. The inner zone suffers necrosis due to the point of maximum damage. Systemically, burns cause massive edema, suppressed cardiac function, respiratory issues like ARDS, immunosuppression increasing infection risk, metabolic changes like increased BMR, and GI changes like paralytic ileus. Monitoring for shock is important due to hemodynamic and fluid/electrolyte imbalances.

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    The body responds to burns in several stages. Initially, tissue perfusion increases in the outermost zone to sustain the least damaged tissue. The middle zone experiences decreased perfusion, leading to inflammation and injury. The inner zone suffers necrosis due to the point of maximum damage. Systemically, burns cause massive edema, suppressed cardiac function, respiratory issues like ARDS, immunosuppression increasing infection risk, metabolic changes like increased BMR, and GI changes like paralytic ileus. Monitoring for shock is important due to hemodynamic and fluid/electrolyte imbalances.

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    25 views3 pages

    Burn Pathophysiology

    Uploaded by

    Charisse Taylan
    The body responds to burns in several stages. Initially, tissue perfusion increases in the outermost zone to sustain the least damaged tissue. The middle zone experiences decreased perfusion, leading to inflammation and injury. The inner zone suffers necrosis due to the point of maximum damage. Systemically, burns cause massive edema, suppressed cardiac function, respiratory issues like ARDS, immunosuppression increasing infection risk, metabolic changes like increased BMR, and GI changes like paralytic ileus. Monitoring for shock is important due to hemodynamic and fluid/electrolyte imbalances.

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    of 3

    BODY’S

    RESPONSE TO A BURN
    • Outermost
    • Tissue perfusion increased
    • Sustains the least damage • Point of maximum damage
    • Inner zone of injury where cellular
    death occurs

    • Decreased tissue perfusion


    • Middle area
    • Inflammation and tissue injury
    Cardiovascular Changes
    Fluid and Electrolyte
    Release of the inflammatory CYTOKINE
    Severe burn NECROSIS FACTOR

    Massive systemic edema Suppressed myocardial infarction

    BP d/t cardiac output

    Hemodynamic/ Systemic changes Monitor for SHOCK

    Hyponatremia & Hyperkalemia HYPOKALEMIA


    Respiratory changes
    Increased Hematocrit Due to inflammatory mediators

    bronchoconstriction
    oliguria Reabsorb fluid diuresis
    ARDS

    Immunological Changes Metabolic changes


    BMR increases up to 3 to times to its
    Depressed immune system
    original rate
    Immunosuppression

    Increased risk for infection and sepsis


    Gastro-Intestinal Changes
    Decreased blood flow to the GIT
    Causing paralytic ileus, GI dysfunction
    and Curling Ulcer

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