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LTC CHF Case Presentation
LTC CHF Case Presentation
LTC CHF Case Presentation
CARE CASE
STUDY: CHF BY: ALIVIA ONEY
WITH
HYPONATREMIA
RESIDENT OVERVIEW
Resident PMH: MEDICATIONS: PERTINENT LABS: OTHER
Information: • Multiple fractures • Lasix for CHF • Na IMPORTANT
• 92 YO male from MVA • tolvaptan tablet
• 7/12- 123 mmol/L INFORMATION:
• 72 in, 158.8 lbs., • dysphagia • 7/20- 124 mmol/L
1x/week- • Resident is given
• obstructive and • 7/25- 130 mmol/L
21.6 BMI (normal hyponatremia Gatorade for low
reflux uropathy • Glucose- 114 ^
WT) • metoprolol and levels
• hematuria • K- 4.6 (WNL)
• skilled d/t recent lisinopril for HTN • Current diet
• Volume depletion • BUN/Cr- 16/0.81
laminectomy, • docusate sodium (WNL) order- heart
acute CHF • Hypocalcemia
tablet- • Hgb/Hct- 11.8/34.1 healthy
episode • Acute RF (low)
constipation • Fluid restriction-
• CHF dx within • Retention of urine
• famotidine- 1200 mL
time since last • Afib • The increased Na
stomach upset • Very healthy for
admission in 2018, • HTN lab came a couple
currently has • CHF days after advanced age
hyponatremia tolvaptan
administration
What is Congestive
Heart Failure?
◦Left-sided
◦Left ventricle weakens, CO to body is decreased
◦Backflow into the pulmonary vein- high pressure
causes pulmonary congestion/edema
◦Right-sided
◦Right ventricle weakens, CO to lungs diminished
◦Backup of blood in the vena cava and body
circulation causes edema in legs, liver, and
abdominal organs.
◦Systolic vs. diastolic
◦Pumping vs. filling issues- both result in
decreased CO and a backflow of blood
◦Compensatory mechanisms: low CO = low BP =
activation of RAAS to preserve blood pressure
and volume
◦this can cause the hyponatremia
EPIDEMIOLOGY- prevalence:
• Lower in Europe than US and Canada
• ~6.2 million adults in the US, 1.8% population
ETIOLOGY:
• Conditions that weaken the heart
• CAD, inflammation, HTN, cardiomyopathy, MI, irregular heartbeat, DM
EPIDEMIOLOGY,
ETIOLOGY, S/S, CLINICAL MANIFESTATIONS: dependent on the type of CHF (left sided
vs. right sided)
AND TYPICAL • Shortness of breath, esp. when laying down, fatigue, confusion, cough,
TREATMENT weakness
• Abdominal pain, N/V, ascites and edema of lower limbs, WT gain, distended
neck veins, hyponatremia
Labs: Brain natriuretic peptide (BNP), raised during HF; other tests to determine kidney and
liver function
Comorbidities: Kidney/liver damage, lack of perfusion and excess build up of fluid in organs
Malnutrition, lack of appetite from ascites and lack of perfusion compromises absorption