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Group 7 fetal distress
Group 7 fetal distress
Anaemia
Hypertension
Diabetes
Maternal infections
Asthma
Post term pregnancies
Labour induction
Lying in supine position
Fetal anaemia
Prematurity
CAUSES
In a normal fetus with adequate oxygenation ,glycolysis and citric acid cycle are
predominant.
In case of chronic placental insufficiency and intrauterine hypoxia:
Formation and deposition of lactic acid and pyruvic acid occurs through anaerobic
glycolysis cause myocardial hypoxia, respiratory acidosis and metabolic acidosis.
Fetal tachycardia and bradycardia occur due to initial stimulation and later depression of
the cardiac pacemaker.
Fetus may pass meconium , due to vagal stimulation which enhances increased intestinal
activity and causes opening of the anal sphincter.
Total sudden cessation of oxygenation will affect the pontine region and cause sudden fetal
death.
CLINICAL PRESENTATION
Physical exam
Health history
Cardiotocography –
Ultrasonography –biophysical profile
Amnioscopy
Hormonal studies
Daily fetal movement count
Contraction stress test
NURSING MANAGMENT
`Fetal assessment
FHR baseline ,variability ,the presence of periodic or non-periodic
acceration and decellerations, depth, duration
`maternal assessment
Positioning, infection, uterine activity, blood pressure, medication
Psychosocial elements; anxiety, pain, lack of knowledge
MANAGEMENT CT…
Turn the woman on her side or a modified knee chest position to relieve
pressure on the umbilical cord.
Administer oxygen(6-8 l/min) to improve the maternal-fetal oxygenation
Increase IV infusions to correct dehydration or dilute oxytocin stimulatory
effects
Monitor amnioinfusion, an invasive procedure that involves the instilling
warm fluid into the uterine cavity by infusion pump to increase fluid
volume and relieve pressure on the umbilical cord.
Correct hypertension if present by infusion of crystalloids eg NS,RL
COMPLICATIONS
Brain injury
Cerebral palsy
Still birth
REFERENCES
Midwifery nursing