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2a FE, ABG - Paul Biluan
2a FE, ABG - Paul Biluan
2a FE, ABG - Paul Biluan
FLUID SPACES
Intracellular Extracellular Intravascular Interstitial Transcellular does not participate in homeostasis HOMEOSTASIS balance and equilibrium of fluids and electrolytes
F/E TRANSPORT
IV SOLUTIONS
Isotonic Solutions PNSS,D5W, PLR Hypotonic Solutions 0.45NaCl, 0.3NaCl Hypertonic Solutions D10W, D5NSS, D5LR
Total 2600ml
HOMEOSTASIS
Kidney filters and regulates f/e retention and excretion Aldosterone causes sodium and water retention and potassium loss ADH causes reabsorption of water
FLUID IMBALANCES
Dehydration occurs when the fluid intake of the body is not sufficient to meet the fluid needs of the body Goal of treatment: restore fluid volume replace electrolytes eliminate the cause of FVD.
ASSESSMENTS INCREASED PR, RR DECREASED BP, LOC, Temp, UO, Peristalsis Increased urinary specific gravity Dry, Poor skin turgor Thirst Decreased body weight Decreased GI motility and bowel sounds.
LABORATORY FINDINGS Increased serum osmolality Increased hematocrit Increased blood urea nitrogen(BUN) level Increased serum sodium level.
INTERVENTIONS Initiate IVF Increase OFI Monitor VS NVS Input/Output Electrolyte levels Weight.
ASSESSMENTS INCREASED BP, PR, RR, UO, Peristalsis Distended neck and hand veins Elevated central venous pressure Moist crackles upon auscultation Headache, LOC Changes, Visual Disturbances Skeletal muscle weakness Paresthesias Pitting edema in dependent
LABORATORY FINDINGS Decreased serum osmolality Decreased hematocrit Decreased BUN level Decreased serum sodium level Decreased urine specific gravity.
INTERVENTIONS Prevent further fluid overload, and restore normal fluid balance Administer diuretics Restrict fluid and sodium intake Monitor VS NVS Input/Output Electrolyte levels Weight
ELECTROYTE IMBALANCES
SODIUM (Na)
135-145 mEq/L
Controls ECF osmotic pressure Necessary for neuromuscular functioning Determines intracellular reactions Maintains acid base balance
HYPONATREMIA
ETIOLOGY tx with diuretics Na restriction GI loss decreased aldosterone third space loss diaphoresis
ASSESSMENT Generalized skeletal muscle weakness Diminished DTR Headache Confusion, LOC Changes Seizures Increased motility and hyperactive bowel sounds Abdominal cramping and diarrhea Decreased urinary specific gravity
MANAGEMENT IVF 0.9 NaCl/IV Replace other electrolytes as needed Salty foods in diet Safety precaution.
HYPERNATREMIA
ETIOLOGY Hyperventilation high Na intake salt tablets rapid saline infusion water deprivation diarrhea
ASSESSMENT Early: spontaneous muscle twitches, irregular muscle contractions Late: skeletal muscle weakness, deep tendon reflexes diminished or absent LOC Changes Increased urinary specific gravity Decreased urinary output Dry skin, dry sticky buccal mucosa
MANAGEMENT Restrict Na in diet Monitor I & O and behavioral changes Increase oral fluids or D5W/IV Diuretics Dialysis
POTASSIUM (K+)
3.5-5.1 mEq/L
Excitability of nerves and muscles ICF osmotic pressure Maintains acid-base balance K deficit:alkalosis K excess: acidosis
HYPOKALEMIA
ETIOLOGY
ASSESSMENT DTR, RR Thready, weak, irregular pulse Anxiety, lethargy, confusion, coma Skeletal muscle weakness, eventual flaccid paralysis N/V, constipation, abdominal distention Decreased urinary specific grav, Increased urinary output
ECG changes: ST depression, shallow, flat or inverted Twaves and prominent U wave.
Banana dried fruits (raisins,prunes) orange raw carrot raw tomato baked potato Melon Watermelon
Potassium supplement Oral: K+ durule tab 1-3 tabs daily KCl IV incorporation slow drip K sparing diuretics
HYPERKALEMIA
ETIOLOGY Excess intake Retention of K Extracellular shift.
ASSESSMENT Early: Muscle twitches, cramps, paresthesia Late: profound weakness, ascending flaccid paralysis in the arms and legs BP, PR, RR Increased motility, hyperactive bowel sounds, Diarrhea ECG changes: Tall peak waves, flat P waves, widened QRS complexes and prolonged PR intervals
MANAGEMENT Avoid K-rich foods Diuretic 10% glucose with regular insulin/IV Ca Gluconate Dialysis.
CALCIUM (K+)
4.5 5.8 mEq/L or 8.6 -10 mg/dL 2 types of Ca Ionized Plasma protein bound Free Ionized Ca is needed for Blood coagulation Muscle contraction Nerve function Bone and teeth formation Vit D and PTH increases GI Ca absorption
HYPOCALCEMIA
ETIOLOGY Decreased ionized Ca Inadequate intake Excess loss Decreased bone and GI tract absorption.
ASSESSMENT Irritable skeletal muscles: twitches, cramps, tetany, seizures Irritability, Paresthesias, numbness Positive Trosseaus and Chvosteks sign Hyperactive DTR Decreased heart rate, Hypotension Increased gastric motility, Hyperactive bowel sounds Abdominal cramping, diarrhea ECG changes: Prolonged ST interval, prolonged QT interval
MANAGEMENT High Ca diet Ca gluconate, oral Ca salts Phosphate binder (AL-OH) Monitor breathing (laryngeal stridor) Seizure precaution
HYPERCALCEMIA
ETIOLOGY Loss from bones immobilization Excess intake Mobilization from bones steroid.
ASSESSMENT Increased heart rate in the early phase Bradycardia in late phases Increased blood pressure Bounding, full peripheral pulses Profound muscle weakness DTR, RR, Peristalsis Disorientation, lethargy or coma Decreased motility and hypoactive bowel sounds Increased urinary ooutput leading to dehydration Formation of renal calculi ECG changes: Shortened ST
MANAGEMENT Increase fluid intake (3-4 L/day), prevent urolithiasis Acid-ash fruit juices (prune, cranberry), ascorbic acid NSS/IV and diuretic Mithramycin reduces serum Ca level Protect from injury to avoid fracture
HYPOMAGNESEMIA
1.5 2.5 mEq/L ETIOLOGY Decreased intake Impaired GI absorption Excessive excretion
ASSESSMENT Tachycardia, hypertension Shallow respirations DTR, Twitches, paresthesias Positive Trousseaus and Chvosteks signs Tetany, seizures Decreased motility, decreased bowel sounds Irritability, Confusion ECG changes: Tall T waves, depressed ST segments
MANAGEMENT Diet supplements: fruits, green leafy vegetable, whole grain cereals, meats, nuts, seafoods Mg salts oral/parenteral Promote safety, prevention of injury Monitor for laryngeal stridor.
HYPERMAGNESEMIA
ETIOLOGY Renal failure Diabetic ketoacidosis Frequent use of magnesium-containing antacids, cathartics
ASSESSMENT Bradycardia, dysrhythmias, Hypotension Respiratory insufficiency Diminished or absent deep tendon reflexes Skeletal muscle weakness Drowsiness and lethargy that progresses to coma
HYPOPHOSPHATEMI A
2.5 4.5 mg/dL ETIOLOGY Hyperparathyroidism Hypercalcemia
HYPERPHOSPHATEMI A
ETIOLOGY Hypoparathyroidism Hypocalcemia ASSESSMENT Signs of hypocalcemia
ACID-BASE IMBALANCES
Ph 7.35-7.45 PCO2 35 45 mmHg HCO3 22-26 mEq/L PaO2 80-100 mmHg SaO2 95-100%
1.
2.
Check pH To determine acidity/alkalinity Determine if Respiratory or Metabolic Use ROME technique Respi CO2 Meta HCO3 Check for compensation FC if pH normalizes UC if no change in the other indicator PC if the other indicator fluctuates
Exercise
Ph 7.34 PCO2 49 HCO3 26
Exercise
Ph 7.50 PCO2 35 HCO3 30
Exercise
Ph 7.36 PCO2 34 HCO3 15
Exercise
Ph 7.38 PCO2 49 HCO3 22
Exercise
Ph 7.48 PCO2 25 HCO3 28